M. Saganich

发表

Contributions of Kv3 Channels to Neuronal Excitability

A. Erisir, B. Rudy, D. Lau, 1999, Annals of the New York Academy of Sciences.

Differential Expression of Genes Encoding Subthreshold-Operating Voltage-Gated K+ Channels in Brain

B. Rudy, M. Saganich, É. Machado, 2001, The Journal of Neuroscience.

Cloning of Components of a Novel Subthreshold-Activating K+ Channel with a Unique Pattern of Expression in the Cerebral Cortex

B. Rudy, M. Saganich, M. Nadal, 1999, The Journal of Neuroscience.

DPP10 Modulates Kv4-mediated A-type Potassium Channels*

B. Rudy, Edward Zagha, M. Saganich, 2005, Journal of Biological Chemistry.

Molecular Diversity of K+ Channels

B. Rudy, D. Lau, A. Chow, 1999, Annals of the New York Academy of Sciences.

Deficits in Synaptic Transmission and Learning in Amyloid Precursor Protein (APP) Transgenic Mice Require C-Terminal Cleavage of APP

S. Heinemann, D. Bredesen, M. Saganich, 2006, The Journal of Neuroscience.

Kv3.1-Kv3.2 channels underlie a high-voltage-activating component of the delayed rectifier K+ current in projecting neurons from the globus pallidus.

B. Rudy, A. Hernández-Cruz, H. Moreno, 1999, Journal of neurophysiology.

B-Ephrin Reverse Signaling Is Required for NMDA-Independent Long-Term Potentiation of Mossy Fibers in the Hippocampus

S. Heinemann, M. Saganich, M. Henkemeyer, 2006, The Journal of Neuroscience.

B-EphrinReverseSignalingIsRequiredfor NMDA-IndependentLong-TermPotentiationof MossyFibersintheHippocampus

M. Saganich, J. N. Armstrong, Michael J. Saganich, 2006 .