J. Divito

Genome-wide association analysis reveals putative Alzheimer's disease susceptibility loci in addition to APOE.

Christoph Lange, Xin Wang, Deborah Blacker, 2008, American journal of human genetics.

LOSS-OF-FUNCTION OF ATXN1 INCREASES A β LEVELS BY POTENTIATING β SECRETASE PROCESSING OF APP

R. Tanzi, Jesse A. Stevenson, J. Divito, 2009 .

Assessment of Alzheimer’s disease case–control associations using family-based methods

D. Blacker, R. Tanzi, C. Lange, 2009, neurogenetics.

Amyloid-β production via cleavage of amyloid-β protein precursor is modulated by cell density.

R. Tanzi, Jesse A. Stevenson, J. Divito, 2010, Journal of Alzheimer's disease : JAD.

Loss of Function of ATXN1 Increases Amyloid β-Protein Levels by Potentiating β-Secretase Processing of β-Amyloid Precursor Protein*

R. Tanzi, Jesse A. Stevenson, J. Divito, 2010, The Journal of Biological Chemistry.

GAB2 as an Alzheimer disease susceptibility gene: follow-up of genomewide association results.

Deborah Blacker, Basavaraj Hooli, Lars Bertram, 2009, Archives of neurology.