D. Carlson

发表

IκB overexpression in cardiomyocytes prevents NF-κB translocation and provides cardioprotection in trauma

D. Carlson, D. White, D. Maass, 2003 .

Protective role of heat stress in burn trauma*

D. Carlson, D. White, J. Horton, 2004, Critical care medicine.

Tumor necrosis factor-α-induced caspase activation mediates endotoxin-related cardiac dysfunction*

D. Carlson, D. White, B. Giroir, 2005, Critical care medicine.

BURN PLASMA MEDIATES CARDIAC MYOCYTE APOPTOSIS VIA ENDOTOXIN: 96

E. Lightfoot, D. Carlson, D. Maass, 2001 .

Beclin-1-Dependent Autophagy Protects the Heart During Sepsis

J. Minei, B. Rothermel, Joseph A. Hill, 2018, Circulation.

Damage-Associated Molecular Patterns and the Systemic Immune Consequences of Severe Thermal Injury

D. Tang, R. Kang, D. Carlson, 2020, The Journal of Immunology.

Burn plasma mediates cardiac myocyte apoptosis via endotoxin.

E. Lightfoot, D. Carlson, D. Maass, 2001, American journal of physiology. Heart and circulatory physiology.

Molecular and pharmacological approaches to inhibiting nitric oxide after burn trauma.

D. Carlson, D. Maass, B. Giroir, 2003, American journal of physiology. Heart and circulatory physiology.

function after major burn injury dyshomeostasis and improves cardiac contractile Caspase inhibition reduces cardiac myocyte

D. Carlson, D. Maass, P. Sikes, 2015 .

Caspase inhibition reduces cardiac myocyte dyshomeostasis and improves cardiac contractile function after major burn injury.

D. Carlson, D. Maass, J. Horton, 2007, Journal of applied physiology.

ALTERATIONS IN THE CARDIAC INFLAMMATORY RESPONSE TO BURN TRAUMA IN MICE LACKING A FUNCTIONAL TOLL-LIKE RECEPTOR 4 GENE

D. Carlson, D. Maass, J. Horton, 2008, Shock.

Macrophage migration inhibitory factor mediates late cardiac dysfunction after burn injury.

J. DiMaio, D. Carlson, D. White, 2005, American journal of physiology. Heart and circulatory physiology.

MACROPHAGE MIGRATION INHIBITORY FACTOR (MIF) MEDIATES LATE CARDIAC DYSFUNCTION FOLLOWING THERMAL INJURY: 320

D. Carlson, B. Giroir, J. Horton, 2004 .

Antioxidant vitamin therapy alters sepsis-related apoptotic myocardial activity and inflammatory responses.

D. Carlson, D. White, D. Maass, 2006, American journal of physiology. Heart and circulatory physiology.

Interleukin-1 Receptor-associated Kinase 2 Is Critical for Lipopolysaccharide-mediated Post-transcriptional Control*

G. Stark, D. Carlson, James A. Thomas, 2009, Journal of Biological Chemistry.

Macrophage migration inhibitory factor is a cardiac-derived myocardial depressant factor.

J. DiMaio, D. Carlson, D. White, 2003, American journal of physiology. Heart and circulatory physiology.

Tau Is Elevated in Pediatric Patients on Extracorporeal Membrane Oxygenation

A. Everett, D. Carlson, J. Gatson, 2020, ASAIO journal.

The role of reactive oxygen species in the heart after electrical stimulation.

D. Carlson, 2008, Critical care medicine.

antioxidant therapy defense after burn injury: the beneficial effects of Cardiac mitochondrial damage and loss of ROS

R. Tompkins, D. Carlson, D. Maass, 2015 .

The cGAS-STING pathway connects mitochondrial damage to inflammation in burn-induced acute lung injury in rat.

D. Tang, R. Kang, D. Carlson, 2021, Burns : journal of the International Society for Burn Injuries.

Muscle Homeostasis Is Disrupted in Burned Adults.

Holly B. Weis, H. Phelan, D. Carlson, 2019, Journal of burn care & research : official publication of the American Burn Association.

An Assessment of Research Priorities to Dampen the Pendulum Swing of Burn Resuscitation.

L. Cancio, C. Wade, K. Chung, 2020, Journal of burn care & research : official publication of the American Burn Association.

Association between COVID-19 and angiotensin-converting enzyme inhibitors with the spotlight on zinc: an opinion

D. Carlson, B. Khiatah, A. Amos, 2021, Annals of medicine.

expression in the heart Burn trauma alters calcium transporter protein

J. Minei, J. Jalife, P. Ward, 2015 .

Burn trauma alters calcium transporter protein expression in the heart.

D. Carlson, D. Maass, J. Horton, 2004, Journal of applied physiology.

Mitochondrial ROS Induces Cardiac Inflammation via a Pathway through mtDNA Damage in a Pneumonia-Related Sepsis Model

J. Minei, D. Carlson, S. Wolf, 2015, PloS one.

Severe Burn-Induced Inflammation and Remodeling of Achilles Tendon in a Rat Model

D. Carlson, S. Wolf, R. Huebinger, 2017, Shock.

Serum Level of Musclin Is Elevated Following Severe Burn.

D. Carlson, S. Wolf, A. Clark, 2019, Journal of burn care & research : official publication of the American Burn Association.

Targeting bacterial adherence inhibits multidrug-resistant Pseudomonas aeruginosa infection following burn injury

K. Orth, A. Krachler, D. Carlson, 2016, Scientific Reports.

Cardiac Molecular Signaling After Burn Trauma

D. Carlson, J. Horton, 2006, Journal of burn care & research : official publication of the American Burn Association.

Cardiac gene expression and function in a mouse model of community-acquired methicillin-resistant Staphylococcus aureus sepsis: Role of inflammatory caspases 1 and 11

D. Carlson, Lei Zhang, M. Peterson, 2019, European Journal of Inflammation.

Burn Serum Stimulates Myoblast Cell Death Associated with IL-6-Induced Mitochondrial Fragmentation

D. Carlson, S. Wolf, R. Huebinger, 2017, Shock.

Estrogen-provided cardiac protection following burn trauma is mediated through a reduction in mitochondria-derived DAMPs.

J. Minei, D. Carlson, D. Maass, 2014, American journal of physiology. Heart and circulatory physiology.

Malondialdehyde-acetaldehyde haptenated protein binds macrophage scavenger receptor(s) and induces lysosomal damage.

L. Klassen, D. Carlson, G. Thiele, 2004, International immunopharmacology.

Na-NMR study 23 P- and 31 burn trauma: a Time course of myocardial sodium accumulation after

J. Minei, D. Carlson, D. White, 2015 .

Bacterial adhesion inhibitor prevents infection in a rodent surgical incision model

J. Minei, K. Orth, A. Krachler, 2020, Virulence.

I kappa B overexpression in cardiomyocytes prevents NF-kappa B translocation and provides cardioprotection in trauma.

D. Carlson, D. White, D. Maass, 2003, American journal of physiology. Heart and circulatory physiology.

Adoption of rescue colloid during burn resuscitation decreases fluid administered and restores end-organ perfusion.

D. Carlson, B. Arnoldo, J. Kubasiak, 2021, Burns : journal of the International Society for Burn Injuries.