D. White

发表

IκB overexpression in cardiomyocytes prevents NF-κB translocation and provides cardioprotection in trauma

D. Carlson, D. White, D. Maass, 2003 .

Postburn cardiac contractile function and biochemical markers of postburn cardiac injury.

D. White, J. Horton, J. Keffer, 1995, Journal of the American College of Surgeons.

MOLECULAR OR PHARMACOLOGIC INHIBITION OF THE CD14 SIGNALING PATHWAY PROTECTS AGAINST BURN-RELATED MYOCARDIAL INFLAMMATION AND DYSFUNCTION

D. White, D. Maass, J. Horton, 2008, Shock.

Protective role of heat stress in burn trauma*

D. Carlson, D. White, J. Horton, 2004, Critical care medicine.

D. White, J. Horton, L. Mahony, 1990, The American journal of physiology.

IRAK1 deletion disrupts cardiac Toll/IL-1 signaling and protects against contractile dysfunction.

D. Kusewitt, D. White, B. Giroir, 2003, American journal of physiology. Heart and circulatory physiology.

Tumor necrosis factor-α-induced caspase activation mediates endotoxin-related cardiac dysfunction*

D. Carlson, D. White, B. Giroir, 2005, Critical care medicine.

Role of p38 mitogen-activated protein kinase in cardiac myocyte secretion of the inflammatory cytokine TNF-alpha.

D. White, D. Maass, J. Horton, 2001, American journal of physiology. Heart and circulatory physiology.

Cardiomyocyte intracellular calcium and cardiac dysfunction after burn trauma

D. White, D. Maass, J. Horton, 2002, Critical care medicine.

Overexpression of cardiac I-κBα prevents endotoxin-induced myocardial dysfunction

Zhijian J. Chen, E. Spencer, D. White, 2001 .

The effects of early excision and grafting on myocardial inflammation and function after burn injury.

D. White, D. Maass, J. Horton, 2006, The Journal of trauma.

Increased Cardiomyocyte Intracellular Calcium during Endotoxin-Induced Cardiac Dysfunction in Guinea Pigs

D. White, D. Maass, B. Giroir, 2000, Pediatric Research.

Inhibition of tumor necrosis factor prevents myocardial dysfunction during burn shock.

D. White, B. Giroir, J. Horton, 1994, The American journal of physiology.

Bone marrow-derived cells contribute to contractile dysfunction in endotoxic shock.

J. Garcia, D. White, J. Horton, 2005, American journal of physiology. Heart and circulatory physiology.

Aspiration pneumonia-induced sepsis increases cardiac dysfunction after burn trauma.

D. White, D. Maass, B. Giroir, 1998, The Journal of surgical research.

Role of p38 mitogen-activated protein kinase in cardiac myocyte secretion of the inﬂammatory cytokine TNF- a

D. White, D. Maass, J. Horton, 2001 .

Macrophage migration inhibitory factor mediates late cardiac dysfunction after burn injury.

J. DiMaio, D. Carlson, D. White, 2005, American journal of physiology. Heart and circulatory physiology.

TLR4 inactivation and rBPI(21) block burn-induced myocardial contractile dysfunction.

D. White, J. Horton, M. Tsen, 2002, American journal of physiology. Heart and circulatory physiology.

Erratum: IRAK1 deletion disrupts cardiac Toll/IL-1 signaling and protects against contractile dysfunction (American Journal of Physiology Heart and Circulatory Physiology (August 2003) 285 (H597-H606))

A. James, D. Kusewitt, D. White, 2004 .

Antioxidant vitamin therapy alters sepsis-related apoptotic myocardial activity and inflammatory responses.

D. Carlson, D. White, D. Maass, 2006, American journal of physiology. Heart and circulatory physiology.

Antioxidant vitamin therapy alters burn trauma-mediated cardiac NF-kappaB activation and cardiomyocyte cytokine secretion.

D. White, D. Maass, B. Giroir, 2001, The Journal of trauma.

Selective decontamination of the digestive tract attenuated the myocardial inflammation and dysfunction that occur with burn injury.

D. White, D. Maass, J. Horton, 2004, American journal of physiology. Heart and circulatory physiology.

IRAK contributes to burn-triggered myocardial contractile dysfunction.

D. White, J. Horton, M. Tsen, 2002, American journal of physiology. Heart and circulatory physiology.

Macrophage migration inhibitory factor is a cardiac-derived myocardial depressant factor.

J. DiMaio, D. Carlson, D. White, 2003, American journal of physiology. Heart and circulatory physiology.

Lipid peroxidation contributes to cardiac deficits after ischemia and reperfusion of the small bowel.

D. White, J. Horton, 1993, The American journal of physiology.

Burn injury decreases myocardial Na-K-ATPase activity: role of PKC inhibition.

D. White, D. Maass, J. Horton, 2007, American journal of physiology. Regulatory, integrative and comparative physiology.

Effects of burn injury on myocardial signaling and cytokine secretion: Possible role of PKC.

D. White, D. Maass, J. Horton, 2007, American journal of physiology. Regulatory, integrative and comparative physiology.

EFFECTS OF BURN SERUM ON MYOCARDIAL INFLAMMATION AND FUNCTION

D. White, D. Maass, J. Horton, 2004, Shock.

Cardiac responses to burn injury in young and adult guinea pigs.

D. White, J. Horton, 1999, Shock.

Cardiac myocyte accumulation of calcium in burn injury: cause or effect of myocardial contractile dysfunction.

D. White, D. Maass, J. Horton, 2005, The Journal of burn care & rehabilitation.

Calcium antagonists improve cardiac mechanical performance after thermal trauma.

D. White, D. Maass, B. Giroir, 1999, The Journal of surgical research.

Femoral Neuropathy Following Spontaneous Retroperitoneal Hemorrhage After Cardiac Surgery: A Case Report.

D. White, F. Lytle, 2017, A & A case reports.

Hypertonic saline dextran after burn injury decreases inflammatory cytokine responses to subsequent pneumonia-related sepsis.

D. White, D. Maass, J. Horton, 2006, American journal of physiology. Heart and circulatory physiology.

Free radical scavengers prevent intestinal ischemia-reperfusion-mediated cardiac dysfunction.

D. White, J. Horton, 1993, The Journal of surgical research.

Burn injury decreased splanchnic PGI2 release is restored by treatment with lazaroid.

D. White, J. Horton, S. Myers, 1993, Prostaglandins.

Hypertonic saline dextran resuscitation of thermal injury.

C. Baxter, D. White, J. Horton, 1990, Annals of surgery.

Bactericidal/permeability increasing protein attenuates the myocardial inflammation/dysfunction that occurs with burn complicated by subsequent infection.

J. Minei, D. White, D. Maass, 2007, Journal of applied physiology.

D. White, D. Maass, J. Horton, 2004, American journal of physiology. Heart and circulatory physiology.

Cardiac contractile and calcium transport function after burn injury in adult and aged guinea pigs.

D. White, J. Horton, L. Mahony, 1993, The Journal of surgical research.

Burn induced cardiac dysfunction is reduced by pentoxifylline.

D. White, J. Horton, W. Vaughan, 1993, Surgery, gynecology & obstetrics.

Delayed hypertonic saline dextran administration after burn injury.

J. Hunt, D. White, J. Horton, 1995, The Journal of trauma.

Diminished cardiac contractile response to burn injury in aged guinea pigs.

D. White, J. Horton, 1993, The Journal of trauma.

U-74,500A inhibition of burn-induced cardiac dysfunction.

D. White, J. Horton, 1992, The Journal of surgical research.

Major burn trauma in rats promotes cardiac and gastrointestinal apoptosis.

E. Lightfoot, P. Lipsky, D. White, 1999, Shock.

Differences in cardiac responses to resuscitation from burn shock.

C. Baxter, D. White, J. Horton, 1989, Surgery, gynecology & obstetrics.

Deficiency in Heat Shock Factor 1 (HSF-1) Expression Exacerbates Sepsis-induced Inflammation and Cardiac Dysfunction.

J. Minei, D. White, D. Maass, 2014, SOJ surgery.

Targeted disruption of ICAM-1, P-selectin genes improves cardiac function and survival in TNF-alpha transgenic mice.

N. Bowles, D. White, B. Giroir, 2001, American journal of physiology. Heart and circulatory physiology.

Na-NMR study 23 P- and 31 burn trauma: a Time course of myocardial sodium accumulation after

J. Minei, D. Carlson, D. White, 2015 .

Cardiac contractile and sarcoplasmic reticulum function after acute ethanol consumption.

D. White, J. Horton, 1996, The Journal of surgical research.

Cardiac contractile injury after intestinal ischemia-reperfusion.

D. White, J. Horton, 1991, The American journal of physiology.

INCREASING PERCENT BURN IS CORRELATED WITH INCREASING INFLAMMATION IN AN ADULT RODENT MODEL

D. White, D. Maass, J. Horton, 2008, Shock.

Role of xanthine oxidase and leukocytes in postburn cardiac dysfunction.

D. White, J. Horton, Jureta W. Horton, 1995, Journal of the American College of Surgeons.

The effects of aging on the cardiac contractile response to unresuscitated thermal injury.

C. Baxter, D. White, J. Horton, 1988, The Journal of burn care & rehabilitation.

I kappa B overexpression in cardiomyocytes prevents NF-kappa B translocation and provides cardioprotection in trauma.

D. Carlson, D. White, D. Maass, 2003, American journal of physiology. Heart and circulatory physiology.

Monoclonal antibody to intercellular adhesion molecule-1 reduces cardiac contractile dysfunction after burn injury in rabbits.

P. Lipsky, D. White, J. Horton, 1996, The Journal of surgical research.

Deficiency in Heat Shock Factor 1 (HSF-1) Expression Exacerbates Sepsis-induced Inflammation and Cardiac Dysfunction.

J. Minei, D. White, D. Maass, 2014, SOJ surgery.