D. Maass

发表

IκB overexpression in cardiomyocytes prevents NF-κB translocation and provides cardioprotection in trauma

D. Carlson, D. White, D. Maass, 2003 .

MOLECULAR OR PHARMACOLOGIC INHIBITION OF THE CD14 SIGNALING PATHWAY PROTECTS AGAINST BURN-RELATED MYOCARDIAL INFLAMMATION AND DYSFUNCTION

D. White, D. Maass, J. Horton, 2008, Shock.

Specific inhibition of mitochondrial oxidative stress suppresses inflammation and improves cardiac function in a rat pneumonia-related sepsis model.

J. Minei, F. Nwariaku, H. Sadek, 2012, American journal of physiology. Heart and circulatory physiology.

in a rat pneumonia-related sepsis model suppresses inflammation and improves cardiac function Specific inhibition of mitochondrial oxidative stress

J. Minei, F. Nwariaku, H. Sadek, 2012 .

Cardiac effects of burn injury complicated by aspiration pneumonia-induced sepsis.

D. Maass, J. Horton, James A. Thomas, 2003, American journal of physiology. Heart and circulatory physiology.

Simultaneous quantification of four native estrogen hormones at trace levels in human cerebrospinal fluid using liquid chromatography-tandem mass spectrometry.

J. Simpkins, D. Maass, K. Schug, 2011, Journal of pharmaceutical and biomedical analysis.

Regulation of VEGF-induced endothelial cell migration by mitochondrial reactive oxygen species.

G. Dulan, F. Nwariaku, P. Shaul, 2011, American journal of physiology. Cell physiology.

BURN PLASMA MEDIATES CARDIAC MYOCYTE APOPTOSIS VIA ENDOTOXIN: 96

E. Lightfoot, D. Carlson, D. Maass, 2001 .

Role of cytosolic vs. mitochondrial Ca2+ accumulation in burn injury-related myocardial inflammation and function.

D. Maass, J. Horton, Jean White, 2005, American journal of physiology. Heart and circulatory physiology.

Role of p38 mitogen-activated protein kinase in cardiac myocyte secretion of the inflammatory cytokine TNF-alpha.

D. White, D. Maass, J. Horton, 2001, American journal of physiology. Heart and circulatory physiology.

Activation of stress-responsive pathways by the sympathetic nervous system in burn trauma.

D. Maass, J. Horton, C. Ballard-Croft, 2001 .

Cardiac mitochondrial damage and inflammation responses in sepsis.

D. Maass, J. Horton, Q. Zang, 2007, Surgical infections.

Cardiomyocyte intracellular calcium and cardiac dysfunction after burn trauma

D. White, D. Maass, J. Horton, 2002, Critical care medicine.

Burn plasma mediates cardiac myocyte apoptosis via endotoxin.

E. Lightfoot, D. Carlson, D. Maass, 2001, American journal of physiology. Heart and circulatory physiology.

RHO-ASSOCIATED KINASE MODULATES MYOCARDIAL INFLAMMATORY CYTOKINE RESPONSES

D. Maass, J. Horton, C. Ballard-Croft, 2005, Shock.

IL-1beta and IL-6 act synergistically with TNF-alpha to alter cardiac contractile function after burn trauma.

D. Maass, J. Horton, Jean White, 2002, Shock.

Role of interleukin-6 in cardiac inflammation and dysfunction after burn complicated by sepsis.

R. Bassel-Duby, D. Maass, J. Horton, 2007, American journal of physiology. Heart and circulatory physiology.

Overexpression of cardiac I-κBα prevents endotoxin-induced myocardial dysfunction

Zhijian J. Chen, E. Spencer, D. White, 2001 .

Molecular and pharmacological approaches to inhibiting nitric oxide after burn trauma.

D. Carlson, D. Maass, B. Giroir, 2003, American journal of physiology. Heart and circulatory physiology.

The effects of early excision and grafting on myocardial inflammation and function after burn injury.

D. White, D. Maass, J. Horton, 2006, The Journal of trauma.

IL-1 AND IL-6 ACT SYNERGISTICALLY WITH TNF- TO ALTER CARDIAC CONTRACTILE FUNCTION AFTER BURN TRAUMA

D. Maass, W. Horton, Jean White, 2002 .

Increased Cardiomyocyte Intracellular Calcium during Endotoxin-Induced Cardiac Dysfunction in Guinea Pigs

D. White, D. Maass, B. Giroir, 2000, Pediatric Research.

Aspiration pneumonia-induced sepsis increases cardiac dysfunction after burn trauma.

D. White, D. Maass, B. Giroir, 1998, The Journal of surgical research.

Cardiac mitochondrial damage and loss of ROS defense after burn injury: the beneficial effects of antioxidant therapy.

D. Maass, J. Horton, Q. Zang, 2007, Journal of applied physiology.

Sodium/hydrogen exchange activity in sepsis and in sepsis complicated by previous injury: 31P and 23Na NMR study*

D. Maass, J. Horton, P. Zhao, 2005, Critical care medicine.

Role of p38 mitogen-activated protein kinase in cardiac myocyte secretion of the inﬂammatory cytokine TNF- a

D. White, D. Maass, J. Horton, 2001 .

function after major burn injury dyshomeostasis and improves cardiac contractile Caspase inhibition reduces cardiac myocyte

D. Carlson, D. Maass, P. Sikes, 2015 .

Caspase inhibition reduces cardiac myocyte dyshomeostasis and improves cardiac contractile function after major burn injury.

D. Carlson, D. Maass, J. Horton, 2007, Journal of applied physiology.

Effect of aspiration pneumonia-induced sepsis on post-burn cardiac inflammation and function in mice.

D. Maass, J. Horton, Jean White, 2006, Surgical infections.

Nitric oxide modulation of TNF-alpha-induced cardiac contractile dysfunction is concentration dependent.

D. Maass, J. Horton, Jean White, 2000, American journal of physiology. Heart and circulatory physiology.

Hypertonic saline-dextran suppresses burn-related cytokine secretion by cardiomyocytes.

D. Maass, J. Horton, Jean White, 2001, American journal of physiology. Heart and circulatory physiology.

ALTERATIONS IN THE CARDIAC INFLAMMATORY RESPONSE TO BURN TRAUMA IN MICE LACKING A FUNCTIONAL TOLL-LIKE RECEPTOR 4 GENE

D. Carlson, D. Maass, J. Horton, 2008, Shock.

Antioxidant vitamin therapy alters sepsis-related apoptotic myocardial activity and inflammatory responses.

D. Carlson, D. White, D. Maass, 2006, American journal of physiology. Heart and circulatory physiology.

Antioxidant vitamin therapy alters burn trauma-mediated cardiac NF-kappaB activation and cardiomyocyte cytokine secretion.

D. White, D. Maass, B. Giroir, 2001, The Journal of trauma.

Nitric oxide donors alter cardiomyocyte cytokine secretion and cardiac function*

D. Maass, J. Horton, Jean White, 2005, Critical care medicine.

by mitochondrial reactive oxygen species Regulation of VEGF-induced endothelial cell migration

G. Dulan, F. Nwariaku, P. Shaul, 2016 .

migration by mitochondrial reactive oxygen species Regulation of VEGF-induced endothelial cell

M. Ushio-Fukai, P. Shaul, D. Maass, 2011 .

Selective decontamination of the digestive tract attenuated the myocardial inflammation and dysfunction that occur with burn injury.

D. White, D. Maass, J. Horton, 2004, American journal of physiology. Heart and circulatory physiology.

Myocardial inflammatory responses to sepsis complicated by previous burn injury.

D. Maass, J. Horton, Jean White, 2003, Surgical infections.

Reducing susceptibility to bacteremia after experimental burn injury: a role for selective decontamination of the digestive tract.

J. Minei, D. Maass, J. Horton, 2007, Journal of applied physiology.

Burn injury decreases myocardial Na-K-ATPase activity: role of PKC inhibition.

D. White, D. Maass, J. Horton, 2007, American journal of physiology. Regulatory, integrative and comparative physiology.

antioxidant therapy defense after burn injury: the beneficial effects of Cardiac mitochondrial damage and loss of ROS

R. Tompkins, D. Carlson, D. Maass, 2015 .

Burn serum causes a CD14-dependent mitochondrial damage in primary cardiomyocytes.

F. Nwariaku, A. Idris, D. Maass, 2010, American journal of physiology. Heart and circulatory physiology.

Effects of burn injury on myocardial signaling and cytokine secretion: Possible role of PKC.

D. White, D. Maass, J. Horton, 2007, American journal of physiology. Regulatory, integrative and comparative physiology.

EFFECTS OF BURN SERUM ON MYOCARDIAL INFLAMMATION AND FUNCTION

D. White, D. Maass, J. Horton, 2004, Shock.

Development of an acute burn model in adult mice for studies of cardiac function and cardiomyocyte cellular function.

D. Maass, B. Giroir, J. Horton, 2001, Shock.

Cardiac myocyte accumulation of calcium in burn injury: cause or effect of myocardial contractile dysfunction.

D. White, D. Maass, J. Horton, 2005, The Journal of burn care & rehabilitation.

Time course of myocardial sodium accumulation after burn trauma: a (31)P- and (23)Na-NMR study.

D. Maass, J. Horton, P. Zhao, 2001, Journal of applied physiology.

Calcium antagonists improve cardiac mechanical performance after thermal trauma.

D. White, D. Maass, B. Giroir, 1999, The Journal of surgical research.

cardiac function in a rat pneumonia-related sepsis model Specific inhibition of mitochondrial oxidative stress suppresses inflammation and improves

J. Minei, F. Nwariaku, M. Murphy, 2015 .

expression in the heart Burn trauma alters calcium transporter protein

J. Minei, J. Jalife, P. Ward, 2015 .

Burn trauma alters calcium transporter protein expression in the heart.

D. Carlson, D. Maass, J. Horton, 2004, Journal of applied physiology.

Sepsis and burn complicated by sepsis alter cardiac transporter expression.

D. Maass, J. Horton, C. Ballard-Croft, 2007, Burns : journal of the International Society for Burn Injuries.

Estrogen treatment following severe burn injury reduces brain inflammation and apoptotic signaling

J. Simpkins, J. Minei, A. Idris, 2009, Journal of Neuroinflammation.

Arginine in burn injury improves cardiac performance and prevents bacterial translocation.

D. Maass, J. Horton, B. Sanders, 1998, Journal of applied physiology.

17β-Estradiol reappropriates mass lost to the hypermetabolic state in thermally injured rats.

J. Minei, D. Maass, S. Wolf, 2013, The Journal of surgical research.

Age-dependent differences of interleukin-6 activity in cardiac function after burn complicated by sepsis.

D. Maass, J. Horton, James A. Thomas, 2010, Burns : journal of the International Society for Burn Injuries.

Hypertonic saline dextran after burn injury decreases inflammatory cytokine responses to subsequent pneumonia-related sepsis.

D. White, D. Maass, J. Horton, 2006, American journal of physiology. Heart and circulatory physiology.

MURINE IN VIVO MYOCARDIAL CONTRACTILE DYSFUNCTION AFTER BURN INJURY IS EXACERBATED BY PNEUMONIA SEPSIS

D. Maass, J. Horton, W. Johnston, 2005, Shock.

The Time Course of Cardiac NF-&kgr;B Activation and TNF-&agr; Secretion by Cardiac Myocytes After Burn Injury: Contribution to Burn-Related Cardiac Contractile Dysfunction

D. Maass, J. Horton, Jureta W. Horton, 2002, Shock.

Bactericidal/permeability increasing protein attenuates the myocardial inflammation/dysfunction that occurs with burn complicated by subsequent infection.

J. Minei, D. White, D. Maass, 2007, Journal of applied physiology.

D. White, D. Maass, J. Horton, 2004, American journal of physiology. Heart and circulatory physiology.

Activation of Stress-Responsive Pathways by the Sympathetic Nervous System in Burn Trauma

D. Maass, C. Ballard-Croft, C. Ballard‐Croft, 2001, Shock.

Estrogen-provided cardiac protection following burn trauma is mediated through a reduction in mitochondria-derived DAMPs.

J. Minei, D. Carlson, D. Maass, 2014, American journal of physiology. Heart and circulatory physiology.

Protein kinase C inhibition improves ventricular function after thermal trauma.

D. Maass, J. Horton, J. White, 1998, Journal of Trauma.

Major burn trauma in rats promotes cardiac and gastrointestinal apoptosis.

E. Lightfoot, P. Lipsky, D. White, 1999, Shock.

Deficiency in Heat Shock Factor 1 (HSF-1) Expression Exacerbates Sepsis-induced Inflammation and Cardiac Dysfunction.

J. Minei, D. White, D. Maass, 2014, SOJ surgery.

Sepsis-Induced Cardiac Mitochondrial Dysfunction Involves Altered Mitochondrial-Localization of Tyrosine Kinase Src and Tyrosine Phosphatase SHP2

J. Minei, D. Maass, S. Wolf, 2012, PloS one.

ECHOCARDIOGRAPHY ASSESSMENT OF MYOCARDIAL FUNCTION AFTER BURN INJURY

D. Maass, J. Horton, M. Garry, 2006, Shock.

Na-NMR study 23 P- and 31 burn trauma: a Time course of myocardial sodium accumulation after

J. Minei, D. Carlson, D. White, 2015 .

Investigation of the temperature stability of premarin intravenous using liquid chromatography-mass spectrometry.

J. Simpkins, D. Maass, K. Schug, 2011, Journal of pharmaceutical and biomedical analysis.

INCREASING PERCENT BURN IS CORRELATED WITH INCREASING INFLAMMATION IN AN ADULT RODENT MODEL

D. White, D. Maass, J. Horton, 2008, Shock.

Age-dependent differences of interleukin-6 activity in cardiac function after burn complicated by sepsis.

D. Maass, J. Horton, Jiexia Quan, 2010, Burns.

I kappa B overexpression in cardiomyocytes prevents NF-kappa B translocation and provides cardioprotection in trauma.

D. Carlson, D. White, D. Maass, 2003, American journal of physiology. Heart and circulatory physiology.

Deficiency in Heat Shock Factor 1 (HSF-1) Expression Exacerbates Sepsis-induced Inflammation and Cardiac Dysfunction.

J. Minei, D. White, D. Maass, 2014, SOJ surgery.

ECHOCARDIOGRAPHY TO ASSESS POSTBURN MYOCARDIAL FUNCTION: 122

D. Maass, Jureta W. Horton, Rao Naseem, 2004 .

Age-Dependent Differences of Interleukin-6 Activity in Cardiac Function after Burn Complicated by Sepsis 1

D. Maass, J. Horton, Jiexia Quan, 2010 .