Y. Fitz

发表

Risk and the efficacy of antiinflammatory agents: retrospective and confirmatory studies of sepsis.

Charles Natanson, C. Natanson, S. Banks, 2002, American journal of respiratory and critical care medicine.

Ethyl pyruvate decreased early nuclear factor-&kgr;B levels but worsened survival in lipopolysaccharide-challenged mice*

H. Mani, Xuemei Li, X. Cui, 2008, Critical care medicine.

Hydrocortisone decreases lethality and inflammatory cytokine and nitric oxide production in rats challenged with B. anthracis cell wall peptidoglycan

J. Shiloach, X. Cui, P. Eichacker, 2020, Intensive Care Medicine Experimental.

Lethality during continuous anthrax lethal toxin infusion is associated with circulatory shock but not inflammatory cytokine or nitric oxide release in rats.

S. Banks, S. Leppla, M. Moayeri, 2004, American journal of physiology. Regulatory, integrative and comparative physiology.

Sublethal doses of Bacillus anthracis lethal toxin inhibit inflammation with lipopolysaccharide and Escherichia coli challenge but have opposite effects on survival.

S. Leppla, M. Moayeri, M. Haley, 2006, The Journal of infectious diseases.

Late treatment with a protective antigen-directed monoclonal antibody improves hemodynamic function and survival in a lethal toxin-infused rat model of anthrax sepsis.

S. Banks, S. Leppla, G. Choi, 2005, The Journal of infectious diseases.

Delayed treatment with W1-mAb, a chimpanzee-derived monoclonal antibody against protective antigen, reduces mortality from challenges with anthrax edema or lethal toxin in rats and with anthrax spores in mice

R. Purcell, S. Leppla, Syd Johnson, 2011, Critical care medicine.

Norepinephrine increases blood pressure but not survival with anthrax lethal toxin in rats*

S. Leppla, M. Moayeri, M. Haley, 2009, Critical care medicine.

Fluid support worsens outcome and negates the benefit of protective antigen-directed monoclonal antibody in a lethal toxin-infused rat Bacillus anthracis shock model*

S. Leppla, M. Moayeri, X. Cui, 2007, Critical care medicine.

Bacillus anthracis cell wall peptidoglycan but not lethal or edema toxins produces changes consistent with disseminated intravascular coagulation in a rat model.

J. Shiloach, H. Mani, E. Vinogradov, 2013, The Journal of infectious diseases.

Bacillus anthracis cell wall produces injurious inflammation but paradoxically decreases the lethality of anthrax lethal toxin in a rat model

J. Shiloach, H. Mani, X. Cui, 2009, Intensive Care Medicine.

DTPA Fe(III) decreases cytokines and hypotension but worsens survival with Escherichia coli sepsis in rats

S. Banks, M. Haley, X. Cui, 2006, Intensive Care Medicine.

Anthrax immune globulin improves hemodynamics and survival during B. anthracis toxin-induced shock in canines receiving titrated fluid and vasopressor support

S. Leppla, M. Moayeri, X. Cui, 2017, Intensive Care Medicine Experimental.

Risk of death does not alter the efficacy of hydrocortisone therapy in a mouse E. coli pneumonia model

D. Annane, C. Natanson, S. Banks, 2008, Intensive Care Medicine.

Severity of sepsis alters the effects of superoxide anion inhibition in a rat sepsis model.

C. Natanson, S. Banks, R. Danner, 2004, Journal of applied physiology.

Purified cell wall from the probiotic bacterium Lactobacillus gasseri activates systemic inflammation and, at higher doses, produces lethality in a rat model

J. Shiloach, X. Cui, P. Eichacker, 2014, Critical Care.

Bacillus anthracis edema and lethal toxin have different hemodynamic effects but function together to worsen shock and outcome in a rat model.

S. Leppla, M. Moayeri, X. Cui, 2007, The Journal of infectious diseases.

Effective dosing of lipid A analogue E5564 in rats depends on the timing of treatment and the route of Escherichia coli infection.

C. Natanson, S. Banks, R. Danner, 2006, The Journal of infectious diseases.

Nitric oxide production contributes to Bacillus anthracis edema toxin-associated arterial hypotension and lethality: ex vivo and in vivo studies in the rat.

S. Leppla, M. Moayeri, X. Cui, 2016, American journal of physiology. Heart and circulatory physiology.

Parthenolide has limited effects on nuclear factor-κβ increases and worsens survival in lipopolysaccharide-challenged C57BL/6J mice

X. Cui, P. Eichacker, Y. Fitz, 2006 .

Bacillus anthracis lethal toxin, but not edema toxin, increases pulmonary artery pressure and permeability in isolated perfused rat lungs.

S. Leppla, M. Moayeri, X. Cui, 2019, American journal of physiology. Heart and circulatory physiology.

Vascular Biology and Microcirculation Bacillus anthracis lethal toxin, but not edema toxin, increases pulmonary artery pressure and permeability in isolated perfused rat lungs

S. Leppla, M. Moayeri, X. Cui, 2019 .

Catheterization of the Carotid Artery and Jugular Vein to Perform Hemodynamic Measures, Infusions and Blood Sampling in a Conscious Rat Model

X. Cui, Y. Fitz, Yan Li, 2015, Journal of visualized experiments : JoVE.

but not severity of infection in a rat sepsis model improves or worsens survival dependent on the route Neutrophil inhibition with L-selectin-directed MAb

C. Natanson, S. Banks, R. Danner, 2015 .

Neutrophil inhibition with L-selectin-directed MAb improves or worsens survival dependent on the route but not severity of infection in a rat sepsis model.

C. Natanson, S. Banks, R. Danner, 2005, Journal of applied physiology.

Bacillus anthracis edema but not lethal toxin challenge in rats is associated with depressed myocardial function in hearts isolated and tested in a Langendorff system.

X. Cui, P. Eichacker, Y. Fitz, 2015, American journal of physiology. Heart and circulatory physiology.

Controlled trials of rG-CSF and CD11b-directed MAb during hyperoxia and E. coli pneumonia in rats.

C. Natanson, S. Banks, R. Danner, 1996, Journal of applied physiology.

B. anthracis edema toxin increases cAMP levels and inhibits phenylephrine-stimulated contraction in a rat aortic ring model.

X. Cui, P. Eichacker, Y. Fitz, 2013, American journal of physiology. Heart and circulatory physiology.

The effect of heparin administration in animal models of sepsis: A prospective study in Escherichia coli-challenged mice and a systematic review and metaregression analysis of published studies*

R. Danner, H. Mani, X. Cui, 2011, Critical care medicine.

ICAM-1 and CD11b inhibition worsen outcome in rats with E. coli pneumonia.

C. Natanson, S. Banks, R. Danner, 1999, Journal of applied physiology.

Anthrax edema toxin has cAMP-mediated stimulatory effects and high-dose lethal toxin has depressant effects in an isolated perfused rat heart model.

S. Leppla, M. Moayeri, X. Cui, 2011, American journal of physiology. Heart and circulatory physiology.

Cardiopulmonary effects of inhaled nitric oxide in normal dogs and during E. coli pneumonia and sepsis.

C. Natanson, S. Banks, R. Danner, 1998, Journal of applied physiology.

Serial measures of total body oxygen consumption in an awake canine model of septic shock.

C. Natanson, S. Banks, R. Danner, 1996, American journal of respiratory and critical care medicine.

SB203580, a p38 inhibitor, improved cardiac function but worsened lung injury and survival during Escherichia coli pneumonia in mice.

R. Danner, H. Mani, G. Ferreyra, 2010, The Journal of trauma.

TGF-beta1 increases microbial clearance but worsens lung injury during Escherichia coli pneumonia in rats.

C. Natanson, S. Banks, A. Roberts, 2003, Cytokine.

Bacillus anthracis Edema Toxin Increases Fractional Free Water and Sodium Reabsorption in an Isolated Perfused Rat Kidney Model

P. Torabi-Parizi, X. Cui, P. Eichacker, 2017, Infection and Immunity.

Raxibacumab augments hemodynamic support and improves outcomes during shock with B. anthracis edema toxin alone or together with lethal toxin in canines

S. Leppla, M. Moayeri, X. Cui, 2015, Intensive Care Medicine Experimental.

rG-CSF reduces endotoxemia and improves survival during E. coli pneumonia.

C. Natanson, S. Banks, R. Danner, 1997, Journal of applied physiology.

Granulocyte colony-stimulating factor has differing effects comparing intravascular versus extravascular models of sepsis.

C. Natanson, S. Banks, R. Danner, 2004, The Journal of trauma.

The individual survival benefits of tumor necrosis factor soluble receptor and fluid administration are not additive in a rat sepsis model

S. Kern, S. Banks, X. Cui, 2011, Intensive Care Medicine.

Acute G-CSF therapy is not protective during lethal E. coli sepsis.

C. Natanson, S. Banks, R. Danner, 2001, American journal of physiology. Regulatory, integrative and comparative physiology.

Release to Blood in Gram-Negative Bacterial Infection in MCP-1 Intrapulmonary G-CSF Rescues Neutrophil Recruitment to the Lung and Neutrophil

P. Munson, S. Batra, C. Natanson, 2015 .

The effect of heparin administration in animal models of sepsis: A prospective study in Escherichia coli-challenged mice and a systematic review and metaregression analysis of published studies*

R. Danner, H. Mani, X. Cui, 2011, Critical care medicine.

B. anthracis edema toxin increases cAMP levels and inhibits phenylephrine-stimulated contraction in a rat aortic ring model.

X. Cui, P. Eichacker, Y. Fitz, 2013, American journal of physiology. Heart and circulatory physiology.

Shock and lethality with anthrax edema toxin in rats are associated with reduced arterial responsiveness to phenylephrine and are reversed with adefovir.

S. Leppla, M. Moayeri, X. Cui, 2017, American journal of physiology. Heart and circulatory physiology.