X. Jiang

发表

Decreased cholesteryl ester transfer protein (CETP) mRNA and protein and increased high density lipoprotein following lipopolysaccharide administration in human CETP transgenic mice.

A. Tall, J. Breslow, A. Walsh, 1995, The Journal of clinical investigation.

Decreased early atherosclerotic lesions in hypertriglyceridemic mice expressing cholesteryl ester transfer protein transgene.

E. Rubin, A. Tall, J. Breslow, 1995, The Journal of clinical investigation.

Genetic cholesteryl ester transfer protein deficiency caused by two prevalent mutations as a major determinant of increased levels of high density lipoprotein cholesterol.

H. Mabuchi, X. Jiang, Akihiro Inazu, 1994, The Journal of clinical investigation.

Cholesteryl Ester Transfer Protein and Phospholipid Transfer Protein Have Nonoverlapping Functions in Vivo *

A. Tall, X. Jiang, M. Lin, 2000, The Journal of Biological Chemistry.

ApoA-II maintains HDL levels in part by inhibition of hepatic lipase. Studies In apoA-II and hepatic lipase double knockout mice.

A. Tall, J. Breslow, X. Jiang, 1999, Journal of lipid research.

Targeted mutation of plasma phospholipid transfer protein gene markedly reduces high-density lipoprotein levels.

A. Tall, X. Jiang, M. Lin, 1999, The Journal of clinical investigation.

1999 George Lyman Duff memorial lecture: lipid transfer proteins, HDL metabolism, and atherogenesis.

A. Tall, Y. Luo, X. Jiang, 2000, Arteriosclerosis, thrombosis, and vascular biology.

Remodeling of HDL by CETP in vivo and by CETP and hepatic lipase in vitro results in enhanced uptake of HDL CE by cells expressing scavenger receptor B-I.

A. Tall, X. Jiang, H. Oliveira, 1999, Journal of lipid research.

A missense mutation in the cholesteryl ester transfer protein gene with possible dominant effects on plasma high density lipoproteins.

S. Yamashita, P. Kussie, X. Jiang, 1993, The Journal of clinical investigation.

Increased high density lipoprotein (HDL), defective hepatic catabolism of ApoA-I and ApoA-II, and decreased ApoA-I mRNA in ob/ob mice. Possible role of leptin in stimulation of HDL turnover.

A. Tall, X. Jiang, D. Silver, 1999, The Journal of biological chemistry.

Increased prebeta-high density lipoprotein, apolipoprotein AI, and phospholipid in mice expressing the human phospholipid transfer protein and human apolipoprotein AI transgenes.

A. Tall, J. Breslow, A. Walsh, 1996, The Journal of clinical investigation.

Dietary cholesterol increases transcription of the human cholesteryl ester transfer protein gene in transgenic mice. Dependence on natural flanking sequences.

A. Tall, J. Breslow, A. Walsh, 1992, The Journal of clinical investigation.

Probucol enhances selective uptake of HDL-associated cholesteryl esters in vitro by a scavenger receptor B-I-dependent mechanism.

A. Tall, X. Jiang, R. Ramakrishnan, 1999, Arteriosclerosis, thrombosis, and vascular biology.

Reduced high density lipoprotein cholesterol in human cholesteryl ester transfer protein transgenic mice.

A. Tall, J. Breslow, A. Walsh, 1991, The Journal of biological chemistry.

Mammalian adipose tissue and muscle are major sources of lipid transfer protein mRNA.

A. Tall, I. Goldberg, P. Moulin, 1991, The Journal of biological chemistry.

The CCAAT/enhancer-binding protein trans-activates the human cholesteryl ester transfer protein gene promoter.

A. Tall, X. Jiang, L. Agellon, 1992, The Journal of biological chemistry.

Phospholipid transfer protein gene knock-out mice have low high density lipoprotein levels, due to hypercatabolism, and accumulate apoA-IV-rich lamellar lipoproteins.

A. Tall, X. Jiang, M. Lin, 2000, Journal of lipid research.

Profound induction of hepatic cholesteryl ester transfer protein transgene expression in apolipoprotein E and low density lipoprotein receptor gene knockout mice. A novel mechanism signals changes in plasma cholesterol levels.

A. Tall, J. Breslow, A. Walsh, 1996, The Journal of clinical investigation.

Regulation of Murine Plasma Phospholipid Transfer Protein Activity and mRNA Levels by Lipopolysaccharide and High Cholesterol Diet (*)

X. Jiang, Xian-cheng Jiang, C. Bruce, 1995, The Journal of Biological Chemistry.

Structure-function relationships of human cholesteryl ester transfer protein: analysis using monoclonal antibodies.

E. Rassart, A. Tall, P. Kussie, 1996, Journal of lipid research.

Receptors and Lipid Transfer Proteins in HDL Metabolism

T. Arai, A. Tall, X. Jiang, 2000, Annals of the New York Academy of Sciences.

Down-regulation of mRNA for the low density lipoprotein receptor in transgenic mice containing the gene for human cholesteryl ester transfer protein. Mechanism to explain accumulation of lipoprotein B particles.

A. Tall, J. Breslow, A. Walsh, 1993, The Journal of biological chemistry.

IFN-gamma potentiates atherosclerosis in ApoE knock-out mice.

A. Tall, C. Schindler, S. Gupta, 1997, The Journal of clinical investigation.

ApoA-I knockout mice: characterization of HDL metabolism in homozygotes and identification of a post-RNA mechanism of apoA-I up-regulation in heterozygotes.

A. Tall, S. Eisenberg, J. Breslow, 1997, Journal of lipid research.

L-arginine prevents xanthoma development and inhibits atherosclerosis in LDL receptor knockout mice.

R. Michler, P. Cannon, X. Jiang, 1997, Circulation.

Increased sphingomyelin content of plasma lipoproteins in apolipoprotein E knockout mice reflects combined production and catabolic defects and enhances reactivity with mammalian sphingomyelinase.

A. Tall, H. Pownall, X. Jiang, 1998, The Journal of clinical investigation.

Point mutagenesis of positively charged amino acids of cholesteryl ester transfer protein: conserved residues within the lipid transfer/lipopolysaccharide binding protein gene family essential for function.

M. Boguski, A. Tall, X. Jiang, 1995, Biochemistry.