Jocelyn M. Bischof

Chronic diazoxide treatment decreases fat mass and improves endurance capacity in an obese mouse model of Prader-Willi syndrome.

Jocelyn M. Bischof, R. Wevrick, 2018, Molecular genetics and metabolism.

Magel2‐null mice are hyper‐responsive to setmelanotide, a melanocortin 4 receptor agonist

W. Colmers, Jocelyn M. Bischof, R. Wevrick, 2016, British journal of pharmacology.

Regionally reduced brain volume, altered serotonin neurochemistry, and abnormal behavior in mice null for the circadian rhythm output gene Magel2

R. Henkelman, Jocelyn M. Bischof, R. Wevrick, 2009, American journal of medical genetics. Part B, Neuropsychiatric genetics : the official publication of the International Society of Psychiatric Genetics.

Muscle dysfunction caused by loss of Magel2 in a mouse model of Prader-Willi and Schaaf-Yang syndromes.

J. Greer, F. Berry, Jocelyn M. Bischof, 2016, Human molecular genetics.

Inactivation of the mouse Magel2 gene results in growth abnormalities similar to Prader-Willi syndrome.

C. Stewart, Jocelyn M. Bischof, R. Wevrick, 2007, Human molecular genetics.

Inactivation of the mouse Magel 2 gene results in growth abnormalities similar to Prader-Willi syndrome

C. Stewart, Jocelyn M. Bischof, R. Wevrick, 2007 .

Stochastic Loss of Silencing of the Imprinted Ndn/NDN Allele, in a Mouse Model and Humans with Prader-Willi Syndrome, Has Functional Consequences

F. Michel, D. Swaab, F. Watrin, 2013, PLoS genetics.

A necdin/MAGE-like gene in the chromosome 15 autism susceptibility region: expression, imprinting, and mapping of the human and mouse orthologues

Jocelyn M. Bischof, R. Wevrick, Thea K. Chibuk, 2001, BMC Genetics.

A MAGEL2-deubiquitinase complex modulates the ubiquitination of circadian rhythm protein CRY1

Jocelyn M. Bischof, R. Wevrick, K. V. Carias, 2020, PloS one.

A MAGE/NDN‐like gene in zebrafish

M. Ekker, Jocelyn M. Bischof, R. Wevrick, 2003, Developmental dynamics : an official publication of the American Association of Anatomists.

Genome-wide analysis of gene transcription in the hypothalamus.

Jocelyn M. Bischof, R. Wevrick, 2005, Physiological genomics.

Regionally reduced brain volume, altered serotonin neurochemistry, and abnormal behavior in mice null for the circadian rhythm output gene Magel2

R. Henkelman, Jocelyn M. Bischof, R. Wevrick, 2009, American journal of medical genetics. Part B, Neuropsychiatric genetics : the official publication of the International Society of Psychiatric Genetics.