J. Hof

发表

Dissociation of obesity and insulin resistance in transgenic mice with skeletal muscle expression of uncoupling protein 1.

S. Klaus, S. Keipert, Y. Katterle, 2008, Physiological genomics.

Aneuploidy in children with relapsed B‐cell precursor acute lymphoblastic leukaemia: clinical importance of detecting a hypodiploid origin of relapse

Michael P. Schroeder, M. P. Schroeder, O. Haas, 2019, British journal of haematology.

Ras pathway mutations are prevalent in relapsed childhood acute lymphoblastic leukemia and confer sensitivity to MEK inhibition.

C. Eckert, J. Allan, C. Harrison, 2013, Blood.

Identification of a genetically defined ultra-high-risk group in relapsed pediatric T-lymphoblastic leukemia

V. Beneš, T. Rausch, J. Korbel, 2017, Blood Cancer Journal.

The activating STAT5B N642H mutation is a common abnormality in pediatric T-cell acute lymphoblastic leukemia and confers a higher risk of relapse

T. Rausch, J. Korbel, A. Stütz, 2014, Haematologica.

Prognostic value of genetic alterations in children with first bone marrow relapse of childhood B-cell precursor acute lymphoblastic leukemia

Julia C. Engelmann, C. Lottaz, C. Eckert, 2013, Leukemia.

Mutations and deletions of the TP53 gene predict nonresponse to treatment and poor outcome in first relapse of childhood acute lymphoblastic leukemia.

C. Eckert, G. Henze, K. Seeger, 2011, Journal of clinical oncology : official journal of the American Society of Clinical Oncology.

Activating mutations in the NT5C2 nucleotidase gene drive chemotherapy resistance in relapsed ALL

A. Ferrando, R. Rabadán, T. Palomero, 2013, Nature Medicine.

Prevalence and prognostic significance of chromosome 21 amplifications in children with relapsed acute lymphoblastic leukemia: the ALL-REZ BFM study group

S. Mundlos, C. Eckert, G. Henze, 2010 .

Pediatric T-cell lymphoblastic leukemia evolves into relapse by clonal selection, acquisition of mutations and promoter hypomethylation

V. Beneš, T. Rausch, J. Korbel, 2015, Haematologica.

NOTCH1 mutation, TP53 alteration and myeloid antigen expression predict outcome heterogeneity in children with first relapse of T-cell acute lymphoblastic leukemia

M. Muckenthaler, C. Eckert, G. Henze, 2017, Haematologica.

High VLA-4 expression is associated with adverse outcome and distinct gene expression changes in childhood B-cell precursor acute lymphoblastic leukemia at first relapse

C. Eckert, G. Henze, K. Seeger, 2011, Haematologica.

Synergistic Activity of Bortezomib and HDACi in Preclinical Models of B-cell Precursor Acute Lymphoblastic Leukemia via Modulation of p53, PI3K/AKT, and NF-κB

C. Eckert, I. Fichtner, G. Henze, 2013, Clinical Cancer Research.

Cell Cycle Analysis: B. Control and Analysis of the Mitotic Cycle in Cultured Plant Root Meristems11Research carried out at Brookhaven National Laboratory under the auspices of the U. S. Atomic Energy Commission.

J. Hof, 1973 .

of p 53 , PI 3 K / AKT , and NF-of B-cell Precursor Acute Lymphoblastic Leukemia via Modulation Synergistic Activity of Bortezomib and HDACi in Preclinical Models

C. Eckert, I. Fichtner, K. Seeger, 2013 .

Cancer Therapy : Preclinical Synergistic Activity of Bortezomib and HDACi in Preclinical Models of B-cell Precursor Acute Lymphoblastic Leukemia via Modulation of p 53 , PI 3 K / AKT , and NFk B

C. Eckert, I. Fichtner, K. Seeger, 2013 .

Subclonal NT5C2 mutations are associated with poor outcomes after relapse of pediatric acute lymphoblastic leukemia.

A. Ferrando, Hossein Khiabanian, C. Eckert, 2020, Blood.

The activating STAT5B N642H mutation is a common abnormality in pediatric T-cell acute lymphoblastic leukemia and confers a higher risk of relapse

T. Rausch, J. Korbel, A. Stütz, 2014, Haematologica.

Synergistic Activity of Bortezomib and HDACi in Preclinical Models of B-cell Precursor Acute Lymphoblastic Leukemia via Modulation of p53, PI3K/AKT, and NF-κB

C. Eckert, I. Fichtner, G. Henze, 2013, Clinical Cancer Research.