J. Etich

发表

Neutral sphingomyelinase (SMPD3) deficiency disrupts the Golgi secretory pathway and causes growth inhibition

W. Stoffel, J. Heilig, Ina Hammels, 2016, Cell Death and Disease.

miR-322 stabilizes MEK1 expression to inhibit RAF/MEK/ERK pathway activation in cartilage

A. Niehoff, J. Heilig, Björn Bluhm, 2017, Development.

Signaling pathways affected by mutations causing osteogenesis imperfecta.

J. Etich, F. Zaucke, O. Semler, 2020, Cellular signalling.

Osteogenesis imperfecta—pathophysiology and therapeutic options

J. Etich, F. Zaucke, Lennart Lessmeier, 2020, Molecular and Cellular Pediatrics.

MiR-26a modulates extracellular matrix homeostasis in cartilage.

Björn Bluhm, J. Etich, B. Brachvogel, 2015, Matrix biology : journal of the International Society for Matrix Biology.

Sorting of growth plate chondrocytes allows the isolation and characterization of cells of a defined differentiation status

S. Vogel, K. Mark, J. Etich, 2010, Journal of bone and mineral research : the official journal of the American Society for Bone and Mineral Research.

Depletion of Collagen IX Alpha1 Impairs Myeloid Cell Function

A. Niehoff, Björn Bluhm, P. Lang, 2018, Stem cells.

Deficiency of annexins A5 and A6 induces complex changes in the transcriptome of growth plate cartilage but does not inhibit the induction of mineralization

U. Schlötzer-Schrehardt, A. Niehoff, S. Moss, 2010, Journal of bone and mineral research : the official journal of the American Society for Bone and Mineral Research.

Interaction between KDELR2 and HSP47 as a Key Determinant in Osteogenesis Imperfecta Caused by Bi-allelic Variants in KDELR2.

G. Pals, Q. Waisfisz, H. Meijers-Heijboer, 2020, American journal of human genetics.

Gene Expression Profiling of the Extracellular Matrix Signature in Macrophages of Different Activation Status: Relevance for Skin Wound Healing

J. Etich, F. Zaucke, B. Brachvogel, 2019, International journal of molecular sciences.

Identification of a myofibroblast-specific expression signature in skin wounds.

J. Etich, H. Abken, B. Brachvogel, 2017, Matrix biology : journal of the International Society for Matrix Biology.

Identification of a reference gene for the quantification of mRNA and miRNA expression during skin wound healing

J. Etich, B. Brachvogel, Lena Pitzler, 2017, Connective tissue research.

miR‐126‐3p Promotes Matrix‐Dependent Perivascular Cell Attachment, Migration and Intercellular Interaction

M. Paulsson, J. Etich, B. Brachvogel, 2016, Stem cells.

Identification of Novel Binding Partners (Annexins) for the Cell Death Signal Phosphatidylserine and Definition of Their Recognition Motif*

M. Paulsson, J. Etich, D. Mielenz, 2010, The Journal of Biological Chemistry.

Isolated Anxa5+/Sca-1+ perivascular cells from mouse meningeal vasculature retain their perivascular phenotype in vitro and in vivo.

P. Farlie, J. Etich, B. Brachvogel, 2007, Experimental cell research.

Respiratory chain inactivation links cartilage-mediated growth retardation to mitochondrial diseases

R. Wiesner, A. Niehoff, M. Plomann, 2019, The Journal of cell biology.

PECAM1+/Sca1+/CD38+ Vascular Cells Transform into Myofibroblast-Like Cells in Skin Wound Repair

M. Paulsson, J. Etich, H. Abken, 2013, PloS one.

Post-weaning epiphysiolysis causes distal femur dysplasia and foreshortened hindlimbs in fetuin-A-deficient mice

P. Boor, I. Costa, J. Etich, 2017, PloS one.

Identification of Novel Binding Partners (Annexins) for the Cell Death Signal Phosphatidylserine and Definition of Their Recognition Motif*

M. Paulsson, J. Etich, D. Mielenz, 2010, Journal of Biological Chemistry.