M. Chvanov

发表

Menadione-induced Reactive Oxygen Species Generation via Redox Cycling Promotes Apoptosis of Murine Pancreatic Acinar Cells*

Stuart Gillies, Mohammed Jaffar, R. Sutton, 2006, Journal of Biological Chemistry.

Aqueous extraction from dachengqi formula granules reduces the severity of mouse acute pancreatitis via inhibition of pancreatic pro-inflammatory signalling pathways.

J. Windsor, Wei Huang, Q. Nunes, 2020, Journal of ethnopharmacology.

Ethyl pyruvate and analogs as potential treatments for acute pancreatitis: A review of in vitro and in vivo studies.

J. Windsor, Wei Huang, R. Sutton, 2019, Pancreatology : official journal of the International Association of Pancreatology (IAP) ... [et al.].

Inhibitors of ORAI1 Prevent Cytosolic Calcium-Associated Injury of Human Pancreatic Acinar Cells and Acute Pancreatitis in 3 Mouse Models

J. Roos, K. Stauderman, Wei Huang, 2015, Gastroenterology.

Direct activation of cytosolic Ca2+ signaling and enzyme secretion by cholecystokinin in human pancreatic acinar cells.

J. Neoptolemos, R. Mukherjee, R. Sutton, 2008, Gastroenterology.

The role of Ca2+ signalling in the physiology and pathophysiology of exocrine pancreas

D. Criddle, M. Chvanov, A. Tepikin, 2020 .

The role of Ca2+ influx in endocytic vacuole formation in pancreatic acinar cells

K. Mikoshiba, R. Sutton, I. Prior, 2014, The Biochemical journal.

The role of Ca 2 + influx in endocytic vacuole formation in pancreatic acinar cells

K. Mikoshiba, R. Sutton, I. Prior, 2014 .

Pharmacologically directed cell disposal: labeling damaged cells for phagocytosis as a strategy against acute pancreatitis.

M. Chvanov, A. Tepikin, O. Petersen, 2010, Molecular interventions.

Mechanisms of Pancreatic Injury Induced by Basic Amino Acids Differ Between L-Arginine, L-Ornithine, and L-Histidine

Wei Huang, P. Szatmary, R. Mukherjee, 2019, Front. Physiol..

Effects of the Mitochondria-Targeted Antioxidant Mitoquinone in Murine Acute Pancreatitis

Wei Huang, P. Szatmary, R. Mukherjee, 2015, Mediators of inflammation.

Free radicals and the pancreatic acinar cells: role in physiology and pathology

M. Chvanov, A. Tepikin, O. Petersen, 2005, Philosophical Transactions of the Royal Society B: Biological Sciences.

Mechanism of mitochondrial permeability transition pore induction and damage in the pancreas: inhibition prevents acute pancreatitis by protecting production of ATP

Wei Huang, R. Mukherjee, R. Sutton, 2015, Gut.

Novel lipophilic probe for detecting near-membrane reactive oxygen species responses and its application for studies of pancreatic acinar cells: effects of pyocyanin and L-ornithine.

Wei Huang, R. Sutton, D. Criddle, 2015, Antioxidants & redox signaling.

Protective Effects of Necrostatin-1 in Acute Pancreatitis: Partial Involvement of Receptor Interacting Protein Kinase 1

J. Bertin, Wei Huang, R. Mukherjee, 2021, Cells.

Expression of human cationic trypsinogen (PRSS1) in murine acinar cells promotes pancreatitis and apoptotic cell death

Wei Huang, J. Neoptolemos, R. Mukherjee, 2014, Cell Death and Disease.

Fatty acid ethyl ester synthase inhibition ameliorates ethanol-induced Ca2+-dependent mitochondrial dysfunction and acute pancreatitis

W. Greenhalf, Wei Huang, R. Mukherjee, 2013, Gut.

Autophagy, Acute Pancreatitis and the Metamorphoses of a Trypsinogen-Activating Organelle

D. Criddle, M. Chvanov, A. Tepikin, 2022, Cells.

LAP-like non-canonical autophagy and evolution of endocytic vacuoles in pancreatic acinar cells

R. Sutton, I. Prior, M. Awais, 2019, Autophagy.

Calcium‐dependent release of NO from intracellular S‐nitrosothiols

M. Chvanov, O. Gerasimenko, A. Tepikin, 2006, The EMBO journal.

Knockout of the Mitochondrial Calcium Uniporter Strongly Suppresses Stimulus-Metabolism Coupling in Pancreatic Acinar Cells but Does not Reduce Severity of Experimental Acute Pancreatitis

R. Sutton, M. Awais, D. Criddle, 2020, Cells.

Systemic histone release disrupts plasmalemma and contributes to necrosis in acute pancreatitis.

Wei Huang, P. Szatmary, R. Sutton, 2017, Pancreatology : official journal of the International Association of Pancreatology (IAP) ... [et al.].

TRO40303 Ameliorates Alcohol-Induced Pancreatitis Through Reduction of Fatty Acid Ethyl Ester–Induced Mitochondrial Injury and Necrotic Cell Death

Wei Huang, R. Sutton, M. Awais, 2017, Pancreas.

Modulation of calcium signalling by mitochondria.

M. Chvanov, S. Barrow, A. Tepikin, 2009, Biochimica et biophysica acta.

Oxidative stress alters mitochondrial bioenergetics and modifies pancreatic cell death independently of cyclophilin D, resulting in an apoptosis-to-necrosis shift

R. Sutton, M. Awais, D. Criddle, 2018, The Journal of Biological Chemistry.

Caspase-8-mediated apoptosis induced by oxidative stress is independent of the intrinsic pathway and dependent on cathepsins.

R. Sutton, A. Watson, D. Criddle, 2007, American journal of physiology. Gastrointestinal and liver physiology.

Intracellular rupture, exocytosis and actin interaction of endocytic vacuoles in pancreatic acinar cells: initiating events in acute pancreatitis

R. Sutton, M. Awais, D. Criddle, 2018, The Journal of physiology.

Caffeine protects against experimental acute pancreatitis by inhibition of inositol 1,4,5-trisphosphate receptor-mediated Ca2+ release

Wei Huang, P. Szatmary, R. Mukherjee, 2015, Gut.

Role of phosphoinositides in STIM1 dynamics and store-operated calcium entry

M. Chvanov, A. Tepikin, O. Petersen, 2009, The Biochemical journal.

ATP depletion induces translocation of STIM1 to puncta and formation of STIM1–ORAI1 clusters: translocation and re-translocation of STIM1 does not require ATP

G. Lur, P. Rudland, R. Barraclough, 2008, Pflügers Archiv - European Journal of Physiology.

Direct activation of cytosolic Ca2+ signaling and enzyme secretion by cholecystokinin in human pancreatic acinar cells.

J. Neoptolemos, R. Mukherjee, R. Sutton, 2008, Gastroenterology.

Deletion of the WD40 domain of ATG16L1 exacerbates acute pancreatitis, abolishes LAP-like non-canonical autophagy and slows trypsin degradation.

D. Criddle, M. Chvanov, A. Tepikin, 2024, Autophagy.