H. Kato

发表

Inhibition of NF‐κB by MG132 through ER stress‐mediated induction of LAP and LIP

S. Nakajima, H. Kato, Shuhei Takahashi, 2011, FEBS letters.

mTORC1 serves ER stress-triggered apoptosis via selective activation of the IRE1–JNK pathway

R. Katoh, S. Nakajima, H. Kato, 2011, Cell Death and Differentiation.

Pre-emptive Quality Control Protects the ER from Protein Overload via the Proximity of ERAD Components and SRP.

Takashi Sato, H. Kai, T. Natsume, 2015, Cell reports.

Pleiotropic Potential of Dehydroxymethylepoxyquinomicin for NF-κB Suppression via Reactive Oxygen Species and Unfolded Protein Response

K. Umezawa, S. Nakajima, H. Kato, 2013, The Journal of Immunology.

and Unfolded Protein Response B Suppression via Reactive Oxygen Species k Dehydroxymethylepoxyquinomicin for NF-Pleiotropic Potential of

K. Umezawa, S. Nakajima, H. Kato, 2013 .

of Akt and mTOR-Dependent Dephosphorylation β Branch of the UPR : Involvement of C / EBP B through the ATF 6 κ Activation of NF-Selective Abrogation of BiP / GRP 78 Blunts

H. Kato, J. Paton, A. Paton, 2011 .

Selective Abrogation of BiP/GRP78 Blunts Activation of NF-κB through the ATF6 Branch of the UPR: Involvement of C/EBPβ and mTOR-Dependent Dephosphorylation of Akt

S. Nakajima, H. Kato, M. Kitamura, 2011, Molecular and Cellular Biology.

ER-resident sensor PERK is essential for mitochondrial thermogenesis in brown adipose tissue

Katsuaki Sato, S. Torii, H. Ichijo, 2020, Life Science Alliance.

Dual Regulation of Cadmium-Induced Apoptosis by mTORC1 through Selective Induction of IRE1 Branches in Unfolded Protein Response

R. Katoh, H. Kato, M. Kitamura, 2013, PloS one.

Cardiovascular , Pulmonary , and Renal Pathology Unfolded Protein Response Causes a Phenotypic Shift of Inflamed Glomerular Cells toward Redifferentiation through Dual Blockade of Akt and Smad Signaling Pathways

R. Katoh, S. Nakajima, H. Kato, 2012 .

Selective, potent blockade of the IRE1 and ATF6 pathways by 4‐phenylbutyric acid analogues

S. Nakajima, H. Kato, M. Kitamura, 2013, British journal of pharmacology.

Induction of CCAAT/enhancer-binding protein-homologous protein by cigarette smoke through the superoxide anion-triggered PERK-eIF2α pathway.

S. Nakajima, H. Kato, Shuhei Takahashi, 2011, Toxicology.

Cordycepin as a sensitizer to tumour necrosis factor (TNF)‐α‐induced apoptosis through eukaryotic translation initiation factor 2α (eIF2α)‐ and mammalian target of rapamycin complex 1 (mTORC1)‐mediated inhibition of nuclear factor (NF)‐κB

S. Nakajima, H. Kato, Y. Chi, 2012, Clinical and experimental immunology.

Aberrant, differential and bidirectional regulation of the unfolded protein response towards cell survival by 3′-deoxyadenosine

R. Katoh, S. Nakajima, H. Kato, 2011, Cell Death and Differentiation.

Blockade of Smad signaling by 3′-deoxyadenosine: a mechanism for its anti-fibrotic potential

R. Katoh, S. Nakajima, H. Kato, 2013, Laboratory Investigation.

Monitoring Lipid Droplet Dynamics in Living Cells by Using Fluorescent Probes.

H. Kato, H. Nishitoh, M. Ishiyama, 2019, Biochemistry.

Blockade of adipocyte differentiation by cordycepin

S. Nakajima, H. Kato, Shuhei Takahashi, 2012, British journal of pharmacology.

Anti-inflammatory subtilase cytotoxin up-regulates A20 through the unfolded protein response.

S. Nakajima, H. Kato, Shuhei Takahashi, 2010, Biochemical and biophysical research communications.

Stress Responses from the Endoplasmic Reticulum in Cancer

H. Kato, H. Nishitoh, 2015, Front. Oncol..

Induction of CCAAT/enhancer-binding protein-homologous protein by cigarette smoke through the superoxide anion-triggered PERK-eIF2α pathway.

S. Nakajima, H. Kato, Shuhei Takahashi, 2011, Toxicology.