C. Galli

发表

Action site and cellular effects of cytotoxin VacA produced byHelicobacter pylori

M. Molinari, M. Molinari, B. Aricó, 2008, Folia Microbiologica.

Evidence for H(+)-induced insertion of influenza hemagglutinin HA2 N-terminal segment into viral membrane.

G. Semenza, C. Galli, J. Brunner, 1994, The Journal of biological chemistry.

The Use of Calnexin and Calreticulin by Cellular and Viral Glycoproteins*

Maurizio Molinari, M. Molinari, M. Pieren, 2005, Journal of Biological Chemistry.

N‐glycan processing selects ERAD‐resistant misfolded proteins for ER‐to‐lysosome‐associated degradation

M. Molinari, C. Galli, T. Soldà, 2021, The EMBO journal.

Segregation and rapid turnover of EDEM1 by an autophagy-like mechanism modulates standard ERAD and folding activities.

M. Molinari, S. Olivari, C. Galli, 2008, Biochemical and biophysical research communications.

Sequential assistance of molecular chaperones and transient formation of covalent complexes during protein degradation from the ER

M. Molinari, M. Pieren, C. Galli, 2002, The Journal of cell biology.

RETRACTED: Role of the SEL1L:LC3-I Complex as an ERAD Tuning Receptor in the Mammalian ER

Riccardo Bernasconi, M. Molinari, C. Galli, 2012, Molecular Cell.

Substrate-specific requirements for UGT1-dependent release from calnexin.

R. Kaufman, M. Molinari, C. Galli, 2007, Molecular cell.

The acid activation of Helicobacter pylori toxin VacA: structural and membrane binding studies.

J. Ruysschaert, M. Molinari, N. Norais, 1998, Biochemical and biophysical research communications.

Selective Inhibition of Ii-dependent Antigen Presentation by Helicobacter pylori Toxin VacA

R. Rappuoli, A. Lanzavecchia, M. Molinari, 1998, The Journal of experimental medicine.

Vacuoles Induced by Helicobacter pylori Toxin Contain Both Late Endosomal and Lysosomal Markers*

R. Rappuoli, M. Molinari, J. Luzio, 1997, The Journal of Biological Chemistry.

Stringent requirement for HRD1, SEL1L, and OS-9/XTP3-B for disposal of ERAD-LS substrates

Riccardo Bernasconi, M. Molinari, T. Nakajima, 2010, The Journal of cell biology.

Role of EDEM in the Release of Misfolded Glycoproteins from the Calnexin Cycle

Maurizio Molinari, P. Lucca, M. Molinari, 2003, Science.

Cyclosporine A-Sensitive, Cyclophilin B-Dependent Endoplasmic Reticulum-Associated Degradation

J. Luban, Riccardo Bernasconi, M. Molinari, 2010, PloS one.

-site speciﬁc intrabodies to decrease and prevent generation of Alzheimer’s A (cid:1) peptide

M. Molinari, C. Galli, P. Paganetti, 2005 .

RETRACTED: Autoadaptive ER-Associated Degradation Defines a Preemptive Unfolded Protein Response Pathway

Riccardo Bernasconi, M. Molinari, C. Galli, 2013, Molecular Cell.

SEC62 and TMX4 control asymmetric autophagy of the nuclear envelope upon LINC complex disassembly

M. Molinari, C. Galli, T. Soldà, 2022, bioRxiv.

Trypanosoma cruzi: the Tc-85 surface glycoprotein shed by trypomastigotes bears a modified glycosylphosphatidylinositol anchor.

C. Galli, W. Colli, M. J. Alves, 1996, Experimental parasitology.

Synthesis of 5-Acyl-3-(ethoxycarbonyl)-2-isoxazolines 2-Oxides by a Tandem Conjugate Addition-Ring Closure of Ethyl Nitroacetate with .alpha.-Bromo Enones

C. Galli, P. Righi, G. Rosini, 1995 .

Contrasting functions of calreticulin and calnexin in glycoprotein folding and ER quality control.

Marek Michalak, Ari Helenius, Peter Cresswell, 2004, Molecular cell.

A Novel Stress-induced EDEM Variant Regulating Endoplasmic Reticulum-associated Glycoprotein Degradation*

Maurizio Molinari, M. Molinari, L. Ruddock, 2005, Journal of Biological Chemistry.

Malectin Participates in a Backup Glycoprotein Quality Control Pathway in the Mammalian ER

Riccardo Bernasconi, M. Molinari, C. Galli, 2011, PloS one.

Chronic Delivery of Antibody Fragments Using Immunoisolated Cell Implants as a Passive Vaccination Tool

R. Gruetter, C. Sandi, M. Cordero, 2011, PloS one.

Translocon component Sec62 acts in endoplasmic reticulum turnover during stress recovery

M. Peter, L. Varani, M. Quadroni, 2016, Nature Cell Biology.

EDEM Contributes to Maintenance of Protein Folding Efficiency and Secretory Capacity*

Maurizio Molinari, M. Molinari, R. Vago, 2004, Journal of Biological Chemistry.

Endoplasmic Reticulum and Lysosomal Quality Control of Four Nonsense Mutants of Iduronate 2-Sulfatase Linked to Hunter's Syndrome.

M. Baumgartner, A. Cavalli, M. Molinari, 2020, DNA and cell biology.

ER‐to‐lysosome‐associated degradation of proteasome‐resistant ATZ polymers occurs via receptor‐mediated vesicular transport

M. Molinari, C. Galli, P. Paganetti, 2018, The EMBO journal.

β-site specific intrabodies to decrease and prevent generation of Alzheimer's Aβ peptide

Maurizio Molinari, M. Molinari, C. Galli, 2005, The Journal of cell biology.

Corrigendum: Translocon component Sec62 acts in endoplasmic reticulum turnover during stress recovery

M. Peter, L. Varani, M. Quadroni, 2016, Nature Cell Biology.

H+-induced Membrane Insertion of Influenza Virus Hemagglutinin Involves the HA2 Amino-terminal Fusion Peptide but Not the Coiled Coil Region*

T. Vorherr, C. Galli, R. Glück, 1996, The Journal of Biological Chemistry.

Persistent glycoprotein misfolding activates the glucosidase II/UGT1-driven calnexin cycle to delay aggregation and loss of folding competence.

R. Kaufman, M. Molinari, C. Galli, 2005, Molecular cell.

TMX4-driven LINC complex disassembly and asymmetric autophagy of the nuclear envelope upon acute ER stress

M. Molinari, C. Galli, A. Raimondi, 2023, Nature communications.

Retraction notice to: role of the SEL1L:LC3-I complex as an ERAD tuning receptor in the mammalian ER.

Fulvio Reggiori, Maurizio Molinari, Riccardo Bernasconi, 2014, Molecular cell.

Stringent requirement for HRD1, SEL1L, and OS-9/XTP3-B for disposal of ERAD-LS substrates

Riccardo Bernasconi, M. Molinari, T. Nakajima, 2010, The Journal of cell biology.

ER-to-lysosome-associated degradation acts as failsafe mechanism upon ERAD dysfunction

M. Molinari, C. Galli, Elisa Fasana, 2024, EMBO reports.