S. G. Clark

发表

The C. elegans gene vab-8 guides posteriorly directed axon outgrowth and cell migration.

Cori Bargmann, A. V. Maricq, B. Wightman, 1996, Development.

IRE1 couples endoplasmic reticulum load to secretory capacity by processing the XBP-1 mRNA

Stevan R. Hubbard, S. Hubbard, F. Urano, 2002, Nature.

Genes involved in two Caenorhabditis elegans cell-signaling pathways.

H. Horvitz, S. G. Clark, M. Stern, 1992, Cold Spring Harbor symposia on quantitative biology.

corrigendum: IRE1 couples endoplasmic reticulum load to secretory capacity by processing the XBP-1 mRNA

S. Hubbard, F. Urano, D. Ron, 2002, Nature.

Biochemical characterization of polypeptides encoded by mutated human Ha-ras1 genes

A. Levinson, S. G. Clark, J. Hayflick, 1986, Molecular and cellular biology.

A survival pathway for Caenorhabditis elegans with a blocked unfolded protein response

M. Kiraly, F. Urano, D. Ron, 2002, The Journal of cell biology.

MIG-10/Lamellipodin and AGE-1/PI3K Promote Axon Guidance and Outgrowth in Response to Slit and Netrin

Cori Bargmann, M. Tessier-Lavigne, S. G. Clark, 2006, Current Biology.

δEF1/ZFH-1 Zn-finger-homeodomain protein. zag-1 mutations cause misexpression of neuron-specific genes, block formation of stereotypic axon branches, perturb neuronal migrations, and induce various axon-guidance,

S. G. Clark, Catherine Chiu, 2003 .

The oncogenic activation of human p21ras by a novel mechanism.

A. Levinson, S. G. Clark, M. Walter, 1986, Science.

Expression of normal and activated human Ha-ras cDNAs in Saccharomyces cerevisiae

A. Levinson, S. G. Clark, J. Mcgrath, 1985, Molecular and cellular biology.

The Caenorhabditis elegans locus lin-15, a negative regulator of a tyrosine kinase signaling pathway, encodes two different proteins.

H. Horvitz, S. G. Clark, X. Lu, 1994, Genetics.

A mammalian host-vector system that regulates expression and amplification of transfected genes by temperature induction.

R. Tjian, D. Rio, S. G. Clark, 1985, Science.

C. elegans cell-signalling gene sem-5 encodes a protein with SH2 and SH3 domains

H. Horvitz, S. G. Clark, Scott G. Clark, 1992, Nature.

Arsenite-inducible RNA-associated protein (AIRAP) protects cells from arsenite toxicity

D. Ron, Jinyu Lu, S. G. Clark, 2001, Cell stress & chaperones.

Caenorhabditis elegans ras gene let-60 acts as a switch in the pathway of vulval induction

H. Horvitz, S. G. Clark, H. Robert Horvitz, 1990, Nature.

PAG-3, a Zn-finger transcription factor, determines neuroblast fate in C. elegans.

H. Horvitz, S. Cameron, S. G. Clark, 2002, Development.

C. elegans AP-2 and retromer control Wnt signaling by regulating mig-14/Wntless.

E. Jorgensen, G. Garriga, S. G. Clark, 2008, Developmental cell.

Wnt signaling establishes anteroposterior neuronal polarity and requires retromer in C. elegans

S. G. Clark, B. Prasad, Brinda C. Prasad, 2006, Development.

Talin loss-of-function uncovers roles in cell contractility and migration in C. elegans

J. Schwarzbauer, E. Cram, S. G. Clark, 2003, Journal of Cell Science.

Genetic Analysis of the Myotubularin Family of Phosphatases in Caenorhabditis elegans*

E. Skolnik, H. Fares, S. G. Clark, 2003, Journal of Biological Chemistry.

Compartment-specific perturbation of protein handling activates genes encoding mitochondrial chaperones

F. Urano, D. Ron, S. G. Clark, 2004, Journal of Cell Science.

Multiple Wnts and frizzled receptors regulate anteriorly directed cell and growth cone migrations in Caenorhabditis elegans.

Cori Bargmann, G. Garriga, S. G. Clark, 2006, Developmental cell.

A dynamin GTPase mutation causes a rapid and reversible temperature-inducible locomotion defect in C. elegans.

Cori Bargmann, A. M. van der Bliek, E. Meyerowitz, 1997, Proceedings of the National Academy of Sciences of the United States of America.

Axons Degenerate in the Absence of Mitochondria in C. elegans

H. Horvitz, E. Jorgensen, R. Weimer, 2014, Current Biology.

The Arf GAP CNT-2 Regulates the Apoptotic Fate in C. elegans Asymmetric Neuroblast Divisions

R. Vale, G. Garriga, S. G. Clark, 2011, Current Biology.

Gain-of-function mutations in the Caenorhabditis elegans lin-1 ETS gene identify a C-terminal regulatory domain phosphorylated by ERK MAP kinase.

H. Horvitz, D. Jacobs, K. Kornfeld, 1998, Genetics.

Control of cell fates in the central body region of C. elegans by the homeobox gene lin-39

Andrew D. Chisholm, H. Horvitz, A. Chisholm, 1993, Cell.

C. elegans ZAG-1, a Zn-finger-homeodomain protein, regulates axonal development and neuronal differentiation

S. G. Clark, Catherine Chiu, 2003, Development.

The conserved transmembrane RING finger protein PLR-1 downregulates Wnt signaling by reducing Frizzled, Ror and Ryk cell-surface levels in C. elegans

S. G. Clark, R. Robinson, Laura L Moffat, 2014, Development.

The conserved transmembrane RING finger protein PLR-1 downregulates Wnt signaling by reducing Frizzled, Ror and Ryk cell-surface levels in C. elegans

S. G. Clark, R. Robinson, Laura L Moffat, 2014, Development.

A survival pathway for Caenorhabditis elegans with a blocked unfolded protein response

M. Kiraly, F. Urano, D. Ron, 2002, The Journal of cell biology.