A. Visintin

发表

Ligand-induced conformational changes allosterically activate Toll-like receptor 9

B. Monks, D. Golenbock, E. Latz, 2007, Nature Immunology.

LPS-TLR4 Signaling to IRF-3/7 and NF-κB Involves the Toll Adapters TRAM and TRIF

Daniel R. Caffrey, B. Monks, D. Golenbock, 2003, The Journal of experimental medicine.

TLR9 signals after translocating from the ER to CpG DNA in the lysosome

B. Monks, D. Golenbock, E. Latz, 2004, Nature Immunology.

CD38 triggers cytotoxic responses in activated human natural killer cells.

F. Malavasi, A. Visintin, F. Pericle, 1999, Blood.

The Apoptotic Response to Pneumolysin Is Toll-Like Receptor 4 Dependent and Protects against Pneumococcal Disease

D. Golenbock, M. Wessels, R. Malley, 2005, Infection and Immunity.

Targeted therapy of solid malignancies via HLA class II antigens: a new biotherapeutic approach?

M. Maio, A. Visintin, E. Fonsatti, 2003, Oncogene.

Cells CD38 Triggers Cytotoxic Responses in Activated Human Natural Killer

F. Malavasi, A. Visintin, F. Pericle, 2013 .

IMMUNOBIOLOGY CD 38 Triggers Cytotoxic Responses in Activated Human Natural Killer Cells

F. Malavasi, A. Visintin, F. Pericle, 1999 .

CD38 triggers cytotoxic responses in activated human natural killer cells.

F. Malavasi, A. Visintin, F. Pericle, 1999 .

CD38 triggers cytotoxic responses in activated human natural killer cells.

F. Malavasi, A. Visintin, F. Pericle, 1999, Blood.

Immunoadhesines anti-pathogene

A. Visintin, Douglas T. Goldenbock, 2005 .

Trypsin-Sensitive Modulation of Intestinal Epithelial MD-2 as Mechanism of Lipopolysaccharide Tolerance1

G. Gerken, D. Golenbock, D. Podolsky, 2006, The Journal of Immunology.

Lipopolysaccharide Tolerance Epithelial MD2 as Mechanism of Trypsin-Sensitive Modulation of Intestinal

G. Gerken, D. Golenbock, D. Podolsky, 2006 .

Phagocytosis and intracellular killing of MD-2 opsonized gram-negative bacteria depend on TLR4 signaling.

S. Ram, D. Golenbock, A. Visintin, 2008, Blood.

bacteria depend on TLR4 signaling Phagocytosis and intracellular killing of MD-2 opsonized Gram-negative

S. Ram, A. Visintin, E. Lien, 2013 .

MD‐2 expression is not required for cell surface targeting of Toll‐like receptor 4 (TLR4)

B. Monks, D. Golenbock, A. Visintin, 2006, Journal of leukocyte biology.

Meningococcal Porin PorB Binds to TLR2 and Requires TLR1 for Signaling1

D. Golenbock, J. Gunawardana, A. Visintin, 2006, The Journal of Immunology.

Localization of TLR2 and MyD88 to Chlamydia trachomatis Inclusions

A. Visintin, A. Quayle, R. Ingalls, 2006, Journal of Biological Chemistry.

Pharmacological Inhibition of Endotoxin Responses Is Achieved by Targeting the TLR4 Coreceptor, MD-21

B. Monks, D. Golenbock, E. Latz, 2005, The Journal of Immunology.

Overexpression of protectin (CD59) down‐modulates the susceptibility of human melanoma cells to homologous complement

M. Colombo, M. Maio, G. Romano, 2000, Journal of cellular physiology.

Melanoma cells constitutively release an anchor-positive soluble form of protectin (sCD59) that retains functional activities in homologous complement-mediated cytotoxicity.

M. Maio, A. Visintin, F. Colizzi, 1997, The Journal of clinical investigation.

IL-10 Enhances MD-2 and CD14 Expression in Monocytes and the Proteins Are Increased and Correlated in HIV-Infected Patients1

D. Golenbock, S. Frøland, P. Aukrust, 2009, The Journal of Immunology.

Responsesfor Toll-Like Receptor 1 in Anti-Bacterial Evidence for an Accessory Protein Function

E. Kiss-Toth, D. Wyllie, A. Visintin, 2000 .

Monocytes and Dendritic Cells Regulation of Toll-Like Receptors in Human

S. Dower, D. Wyllie, A. Visintin, 2000 .

Toll‐like receptor 1 inhibits Toll‐like receptor 4 signaling in endothelial cells

A. Visintin, A. Mazzoni, D. Segal, 2002, European journal of immunology.

Prolonged upregulation of the expression of HLA class I antigens and costimulatory molecules on melanoma cells treated with 5-aza-2'-deoxycytidine (5-AZA-CdR).

A. Cattelan, M. Maio, A. Visintin, 1999, Journal of immunotherapy.

Endothelium through Distinct TLRs Proinflammatory Activation in the Bacterial Fimbriae Stimulate

D. Golenbock, A. Visintin, C. Genco, 2008 .

Allergenicity resulting from functional mimicry of a Toll-like receptor complex protein

R. Hegde, C. Karp, M. Wills-Karp, 2008, Nature.

D. Golenbock, S. Frøland, P. Aukrust, 2008 .

Structure, distribution, and functional role of protectin (CD59) in complement-susceptibility and in immunotherapy of human malignancies (Review).

M. Maio, A. Visintin, L. Sigalotti, 1998, International journal of oncology.

Histamine regulates cytokine production in maturing dendritic cells, resulting in altered T cell polarization.

H. Young, A. Visintin, A. Mazzoni, 2001, The Journal of clinical investigation.

Vitiligo following melanoma in Vietnamese pot-bellied pigs: gross, ultrastructural and immunological studies

M. Maio, A. Visintin, F. Roperto, 1998 .

Myeloid Suppressor Lines Inhibit T Cell Responses by an NO-Dependent Mechanism1

A. Visintin, V. Bronte, A. Mazzoni, 2002, The Journal of Immunology.

Lipopolysaccharide Rapidly Traffics to and from the Golgi Apparatus with the Toll-like Receptor 4-MD-2-CD14 Complex in a Process That Is Distinct from the Initiation of Signal Transduction* 210

Douglas T. Golenbock, Egil Lien, Terje Espevik, 2002, The Journal of Biological Chemistry.

Lysines 128 and 132 Enable Lipopolysaccharide Binding to MD-2, Leading to Toll-like Receptor-4 Aggregation and Signal Transduction*

B. Monks, D. Golenbock, E. Latz, 2003, Journal of Biological Chemistry.

The role of disulfide bonds in the assembly and function of MD-2

D. Davies, A. Visintin, A. Mazzoni, 2003, Proceedings of the National Academy of Sciences of the United States of America.

Epithelial MD-2 as Mechanism of Trypsin-Sensitive Modulation of Intestinal

Douglas T. Golenbock, M. Rünzi, Guido Gerken, 2013 .

Secreted MD-2 is a large polymeric protein that efficiently confers lipopolysaccharide sensitivity to Toll-like receptor 4

A. Visintin, A. Mazzoni, D. Segal, 2001, Proceedings of the National Academy of Sciences of the United States of America.

Regulation of Toll-Like Receptors in Human Monocytes and Dendritic Cells1

S. Dower, D. Wyllie, A. Visintin, 2001, The Journal of Immunology.

Malaria hemozoin is immunologically inert but radically enhances innate responses by presenting malaria DNA to Toll-like receptor 9

B. Monks, D. Golenbock, E. Latz, 2007, Proceedings of the National Academy of Sciences.

Negative regulation of Toll-like receptor 4 signaling by the Toll-like receptor homolog RP105

A. Tarakhovsky, Y. Belkaid, D. Golenbock, 2005, Nature Immunology.

Evidence for an Accessory Protein Function for Toll-Like Receptor 1 in Anti-Bacterial Responses1

E. Kiss-Toth, D. Wyllie, A. Visintin, 2000, The Journal of Immunology.

Mechanisms of TLR9 activation

D. Golenbock, E. Latz, T. Espevik, 2004, Journal of endotoxin research.

Inhibition of TLR-4/MD-2 signaling by RP105/MD-1

A. Tarakhovsky, Y. Belkaid, D. Golenbock, 2005, Journal of endotoxin research.

The functional repertoire of rabbit antibodies and antibody discovery via next-generation sequencing

C. Huard, J. C. Almagro, A. Visintin, 2014, mAbs.

Novel Anti-TM4SF1 Antibody–Drug Conjugates with Activity against Tumor Cells and Tumor Vasculature

Anson K. Abraham, H. Dvorak, L. Zawel, 2015, Molecular Cancer Therapeutics.

Corrigendum: Ligand-induced conformational changes allosterically activate Toll-like receptor 9

B. Monks, D. Golenbock, E. Latz, 2007, Nature Immunology.

248 Empiric profiling of peripheral T cell recall responses to tumor mutanomes versus in silico predictions in NSCLC patients undergoing pembrolizumab treatment ± chemotherapy

A. Visintin, Tulin Dadali, M. Awad, 2021, Journal for ImmunoTherapy of Cancer.

Targeting of HLA‐DR molecules transduces agonistic functional signals in cutaneous melanoma

A. Leonardi, M. Maio, C. Pucillo, 2004, Journal of cellular physiology.

A Novel Factor H–Fc Chimeric Immunotherapeutic Molecule against Neisseria gonorrhoeae

M. Unemo, M. Ohnishi, S. Ram, 2012, The Journal of Immunology.

Neisseria gonorrhoeae Immunotherapeutic Molecule against Fc Chimeric - A Novel Factor H

M. Unemo, M. Ohnishi, S. Gulati, 2016 .

Bacterial Fimbriae Stimulate Proinflammatory Activation in the Endothelium through Distinct TLRs1

F. C. Gibson, D. Golenbock, A. Visintin, 2008, The Journal of Immunology.

Species-Specific Complement Evasion Neisseria gonorrhoeae and Results in Human Factor H Interacts Selectively with

S. Ram, S. Gulati, B. Monks, 2013 .

Human Factor H Interacts Selectively with Neisseria gonorrhoeae and Results in Species-Specific Complement Evasion1

S. Ram, S. Gulati, B. Monks, 2008, The Journal of Immunology.

Species-Specific Complement Evasion and Results in Neisseria gonorrhoeae Human Factor H Interacts Selectively with

S. Ram, S. Gulati, B. Monks, 2008 .

A coding mutation within the first exon of the human MD-2 gene results in decreased lipopolysaccharide-induced signaling

P. Schlag, M. Müller, R. Schumann, 2004, Genes and Immunity.

A Point Mutation in the Amino Terminus of TLR7 Abolishes Signaling without Affecting Ligand Binding

I. Leykin, B. Beutler, M. Mbow, 2011, The Journal of Immunology.

The LPS receptor generates inflammatory signals from the cell surface

D. Golenbock, E. Latz, T. Espevik, 2003, Journal of endotoxin research.

Molecular Characterization of the Interaction between Sialylated Neisseria gonorrhoeae and Factor H*

S. Ram, B. Monks, P. Rice, 2011, The Journal of Biological Chemistry.

Mechanisms of TLR9 activation

D. Golenbock, E. Latz, T. Espevik, 2004, Journal of endotoxin research.