M. Broecker

发表

Interaction of human chorionic gonadotropin (hCG) and asialo-hCG with recombinant human thyrotropin receptor.

M. Grossmann, R. Hoermann, M. Broecker, 1994, The Journal of clinical endocrinology and metabolism.

Thyrotropin May Not Be the Dominant Growth Factor in Benign and Malignant Thyroid Tumors1

M. Broecker, M. Derwahl, Z. Kraiem, 1999 .

Impact of estrogen replacement therapy in a male with congenital aromatase deficiency caused by a novel mutation in the CYP19 gene.

A. Bockisch, M. Broecker, K. Mann, 2002, The Journal of clinical endocrinology and metabolism.

Excessive activation of tyrosine kinases leads to inhibition of proliferation in a thyroid carcinoma cell line.

M. Broecker, M. Derwahl, J. Hammer, 1998, Life sciences.

Mutations of the TSH receptor as cause of congenital hyperthyroidism.

R. Paschke, A. Grüters, M. Broecker, 2009, Experimental and clinical endocrinology & diabetes : official journal, German Society of Endocrinology [and] German Diabetes Association.

RT-PCR-based detection of circulating calcitonin-producing cells in patients with advanced medullary thyroid cancer.

G. Feldmann, M. Broecker, K. Mann, 2002, The Journal of clinical endocrinology and metabolism.

Thyroid nodules in recurrent multinodular goiters are predominantly polyclonal

M. Broecker, M. Derwahl, H. Schatz, 1998, Journal of endocrinological investigation.

Constitutively active germline mutation of the thyrotropin receptor gene as a cause of congenital hyperthyroidism.

M. Lohse, M. Broecker, M. Derwahl, 1997, The Journal of pediatrics.

Expression of the human TSH receptor in a human thyroid carcinoma cell line that lacks an endogenous TSH receptor: growth inhibition by cAMP.

M. Broecker, M. Derwahl, M. Kuemmel, 1993, Biochemical and biophysical research communications.

Suppression of thyrotropin receptor-G protein-phospholipase C coupling by activation of protein kinase C in thyroid carcinoma cells.

G. Mayr, M. Broecker, M. Derwahl, 1997, Endocrinology.

Constitutive activation of the Gs alpha protein-adenylate cyclase pathway may not be sufficient to generate toxic thyroid adenomas.

P. Kopp, S. Filetti, D. Russo, 1996, The Journal of clinical endocrinology and metabolism.

Pathogenesis of toxic thyroid adenomas and nodules: relevance of activating mutations in the TSH-receptor and Gs-alpha gene, the possible role of iodine deficiency and secondary and TSH-independent molecular mechanisms.

M. Broecker, M. Derwahl, A. Sobke, 2009, Experimental and clinical endocrinology & diabetes : official journal, German Society of Endocrinology [and] German Diabetes Association.

Clinical review 101: Thyrotropin may not be the dominant growth factor in benign and malignant thyroid tumors.

M. Broecker, M. Derwahl, Z. Kraiem, 1999, The Journal of clinical endocrinology and metabolism.

Malignant transformation of rat thyroid cells transfected with the human TSH receptor cDNA.

M. Broecker, M. Derwahl, H. Schatz, 1992, Biochemical and biophysical research communications.