M. Hillaire

发表

Binding of DC-SIGN to the Hemagglutinin of Influenza A Viruses Supports Virus Replication in DC-SIGN Expressing Cells

A. Osterhaus, G. Rimmelzwaan, R. Fouchier, 2013, PloS one.

Human Cytotoxic T Lymphocytes Directed to Seasonal Influenza A Viruses Cross-React with the Newly Emerging H7N9 Virus

A. Osterhaus, G. Rimmelzwaan, R. Fouchier, 2013, Journal of Virology.

Human T-cells directed to seasonal influenza A virus cross-react with 2009 pandemic influenza A (H1N1) and swine-origin triple-reassortant H3N2 influenza viruses.

A. Osterhaus, G. Rimmelzwaan, R. Fouchier, 2013, The Journal of general virology.

Infection of the Upper Respiratory Tract with Seasonal Influenza A(H3N2) Virus Induces Protective Immunity in Ferrets against Infection with A(H1N1)pdm09 Virus after Intranasal, but Not Intratracheal, Inoculation

T. Kuiken, A. Osterhaus, G. Rimmelzwaan, 2013, Journal of Virology.

Assessment of the antiviral properties of recombinant surfactant protein D against influenza B virus in vitro.

T. Kuiken, A. Osterhaus, G. Rimmelzwaan, 2015, Virus research.

Vaccination with whole inactivated virus vaccine affects the induction of heterosubtypic immunity against influenza virus A/H5N1 and immunodominance of virus-specific CD8+ T-cell responses in mice.

A. Osterhaus, G. Rimmelzwaan, R. Fouchier, 2010, The Journal of general virology.

Characterization of the Human CD8+ T Cell Response following Infection with 2009 Pandemic Influenza H1N1 Virus

A. Osterhaus, M. Koopmans, G. Rimmelzwaan, 2011, Journal of Virology.

Human Influenza A Virus-Specific CD8+ T-Cell Response Is Long-lived.

A. Osterhaus, G. Rimmelzwaan, R. Fouchier, 2015, The Journal of infectious diseases.

Autophagy: A Home Remodeler for Hepatitis C Virus

M. Hillaire, M. Dreux, E. Décembre, 2014 .

Assessment of the Antiviral Properties of Recombinant Porcine SP-D against Various Influenza A Viruses In Vitro

T. Kuiken, A. Osterhaus, R. Romijn, 2011, PloS one.

Influenza B virus-specific CD8+ T-lymphocytes strongly cross-react with viruses of the opposing influenza B lineage.

A. Osterhaus, G. Rimmelzwaan, R. Fouchier, 2015, The Journal of general virology.

Clearance of influenza virus infections by T cells: risk of collateral damage?

G. Rimmelzwaan, J. Kreijtz, M. Hillaire, 2013, Current opinion in virology.

Induction of Virus-Specific Cytotoxic T Lymphocytes as a Basis for the Development of Broadly Protective Influenza Vaccines

A. Osterhaus, G. Rimmelzwaan, M. Hillaire, 2011, Journal of biomedicine & biotechnology.

Cross-protective immunity against influenza pH1N1 2009 viruses induced by seasonal influenza A (H3N2) virus is mediated by virus-specific T-cells.

T. Kuiken, A. Osterhaus, G. Rimmelzwaan, 2011, The Journal of general virology.

Sensing of Immature Particles Produced by Dengue Virus Infected Cells Induces an Antiviral Response by Plasmacytoid Dendritic Cells

G. Screaton, A. Davidson, M. Hillaire, 2014, PLoS pathogens.

Enhanced Antiviral Activity of Human Surfactant Protein D by Site-Specific Engineering of the Carbohydrate Recognition Domain

M. Rynkiewicz, G. Rimmelzwaan, K. Hartshorn, 2019, Front. Immunol..

Recombinant porcine surfactant protein D inhibits influenza A virus replication ex vivo.

A. Osterhaus, G. Rimmelzwaan, R. Fouchier, 2014, Virus research.

Pulmonary Surfactant Protein D in First-Line Innate Defence against Influenza A Virus Infections

A. Osterhaus, G. Rimmelzwaan, M. Hillaire, 2013, Journal of Innate Immunity.

The number and position of N-linked glycosylation sites in the hemagglutinin determine differential recognition of seasonal and 2009 pandemic H1N1 influenza virus by porcine surfactant protein D.

A. Osterhaus, G. Rimmelzwaan, R. Fouchier, 2012, Virus research.

Cross-protective immunity against influenza pH1N1 2009 viruses induced by seasonal influenza A (H3N2) virus is mediated by virus-specific T-cells.

T. Kuiken, A. Osterhaus, G. Rimmelzwaan, 2011, The Journal of general virology.

IFN-γ: A Crucial Player in the Fight Against HBV Infection?

M. Hillaire, P. Lawrence, Brice Lagrange, 2023, Immune network.