Sangmin Lee

发表

Degenerative Neurological and Neuromuscular Disease Pathogenic mechanisms of neurodegeneration based on the phenotypic expression of progressive forms of immune-mediated neurologic disease

M. Levin, Sangmin Lee, L. Gardner, 2012 .

A potential link between autoimmunity and neurodegeneration in immune-mediated neurological disease

R. Homayouni, C. Raine, M. Levin, 2011, Journal of Neuroimmunology.

Antibodies to the RNA binding protein heterogeneous nuclear ribonucleoprotein A1 contribute to neuronal cell loss in an animal model of multiple sclerosis

M. Levin, Sangmin Lee, Cole D. Libner, 2019, The Journal of comparative neurology.

Novel somatic single nucleotide variants within the RNA binding protein hnRNP A1 in multiple sclerosis patients

M. Levin, Sangmin Lee, 2014, F1000Research.

Novel somatic single nucleotide variants within the RNA binding protein hnRNP A1 in multiple sclerosis patients

M. Levin, Sangmin Lee, 2014, F1000Research.

Cross-Reactive Antibodies to Target Proteins are Dependent upon Oligomannose Glycosylated Epitopes in HTLV-1 Associated Neurological Disease

E. Gotuzzo, M. Levin, Sangmin Lee, 2012, Journal of Clinical Immunology.

Post-translational glycosylation of target proteins implicate molecular mimicry in the pathogenesis of HTLV-1 associated neurological disease

M. Levin, Sangmin Lee, Yoojin Shin, 2008, Journal of Neuroimmunology.

A role for Apolipoprotein A-I in the pathogenesis of multiple sclerosis

M. Levin, Sangmin Lee, L. Gardner, 2014, Journal of Neuroimmunology.

Localization of near-infrared labeled antibodies to the central nervous system in experimental autoimmune encephalomyelitis

M. Levin, Sangmin Lee, Hannah E. Salapa, 2019, PloS one.

Antibody transfection into neurons as a tool to study disease pathogenesis.

M. Levin, Sangmin Lee, L. Gardner, 2012, Journal of visualized experiments : JoVE.

Cross-Reactive Antibodies to Target Proteins are Dependent upon Oligomannose Glycosylated Epitopes in HTLV-1 Associated Neurological Disease

E. Gotuzzo, M. Levin, Sangmin Lee, 2012, Journal of Clinical Immunology.

Contribution of the Degeneration of the Neuro-Axonal Unit to the Pathogenesis of Multiple Sclerosis

M. Levin, Sangmin Lee, Yoojin Shin, 2017, Brain sciences.

Neurodegeneration in multiple sclerosis involves multiple pathogenic mechanisms

M. Levin, Sangmin Lee, L. Gardner, 2014, Degenerative neurological and neuromuscular disease.

Autoantibodies to Non-myelin Antigens as Contributors to the Pathogenesis of Multiple Sclerosis.

M. Levin, Sangmin Lee, L. Gardner, 2013, Journal of clinical & cellular immunology.

Autoantibodies to heterogeneous nuclear ribonuclear protein A1 (hnRNPA1) cause altered ‘ribostasis’ and neurodegeneration; the legacy of HAM/TSP as a model of progressive multiple sclerosis

M. Levin, Sangmin Lee, L. Gardner, 2017, Journal of Neuroimmunology.

Antibodies to the RNA-binding protein hnRNP A1 contribute to neurodegeneration in a model of central nervous system autoimmune inflammatory disease

P. Sawchenko, M. Levin, Sangmin Lee, 2016, Journal of Neuroinflammation.

Antibodies to the RNA-binding protein hnRNP A1 contribute to neurodegeneration in a model of central nervous system autoimmune inflammatory disease

P. Sawchenko, M. Levin, Sangmin Lee, 2016, Journal of Neuroinflammation.

Novel somatic single nucleotide variants within the RNA binding protein hnRNP A1 in multiple sclerosis patients

M. Levin, Sangmin Lee, 2014, F1000Research.

Neurodegeneration in multiple sclerosis involves multiple pathogenic mechanisms

M. Levin, Sangmin Lee, Yoojin Shin, 2014, Degenerative Neurological and Neuromuscular Disease.

Degenerative Neurological and Neuromuscular Disease Dovepress Neurodegeneration in Multiple Sclerosis Involves Multiple Pathogenic Mechanisms

M. Levin, Sangmin Lee, Yoojin Shin, 2022 .

Pathogenic mechanisms of neurodegeneration based on the phenotypic expression of progressive forms of immune-mediated neurologic disease.

Sangmin Lee, Yoojin Shin, Chassidy J. Groover, 2012, Degenerative neurological and neuromuscular disease.