P. Sims

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A Minimal Nuclear Localization Signal (NLS) in Human Phospholipid Scramblase 4 That Binds Only the Minor NLS-binding Site of Importin α1*

G. Cingolani, A. Bhardwaj, Kaylen Lott, 2011, The Journal of Biological Chemistry.

Isolation of an Erythrocyte Membrane Protein that Mediates Ca2+-dependent Transbilayer Movement of Phospholipid*

P. Sims, F. Basse, J. Stout, 1996, The Journal of Biological Chemistry.

Palmitoylation of phospholipid scramblase is required for normal function in promoting Ca2+-activated transbilayer movement of membrane phospholipids.

P. Sims, T. Wiedmer, J. Zhao, 1998, Biochemistry.

Change in conformation of plasma membrane phospholipid scramblase induced by occupancy of its Ca2+ binding site.

P. Sims, J. Stout, T. Wiedmer, 1998, Biochemistry.

Identity of a conserved motif in phospholipid scramblase that is required for Ca2+-accelerated transbilayer movement of membrane phospholipids.

P. Sims, T. Wiedmer, Q. Zhou, 1998, Biochemistry.

Plasma membrane phospholipid scramblase 1 is enriched in lipid rafts and interacts with the epidermal growth factor receptor.

P. Sims, T. Wiedmer, Jun Sun, 2002, Biochemistry.

Palmitoylation of phospholipid scramblase 1 controls its distribution between nucleus and plasma membrane.

P. Sims, T. Wiedmer, M. Nanjundan, 2003, Biochemistry.

Phospholipid scramblase 1 is imported into the nucleus by a receptor-mediated pathway and interacts with DNA.

L. Gerace, P. Sims, T. Wiedmer, 2004, Biochemistry.

Studies on the mechanism by which cyanine dyes measure membrane potential in red blood cells and phosphatidylcholine vesicles.

C H Wang, A. Waggoner, P. Sims, 1974, Biochemistry.

Cyanine dye fluorescence used to measure membrane potential changes due to the assembly of complement proteins C5b-9

P. Sims, T. Wiedmer, 2005, The Journal of Membrane Biology.

Level of Expression of Phospholipid Scramblase Regulates Induced Movement of Phosphatidylserine to the Cell Surface*

P. Sims, T. Wiedmer, J. Zhao, 1998, The Journal of Biological Chemistry.

Molecular Cloning of Human Plasma Membrane Phospholipid Scramblase

P. Sims, J. Stout, T. Wiedmer, 1997, The Journal of Biological Chemistry.

Scott syndrome erythrocytes contain a membrane protein capable of mediating Ca2+-dependent transbilayer migration of membrane phospholipids.

P. Sims, F. Basse, J. Stout, 1997, The Journal of clinical investigation.

Production and characterization of a mutant cell line defective in aminophospholipid translocase.

P. Sims, T. Wiedmer, J. Zhao, 1997, Biochimica et biophysica acta.

Defective Ca(2+)-induced microvesiculation and deficient expression of procoagulant activity in erythrocytes from a patient with a bleeding disorder: a study of the red blood cells of Scott syndrome

P. Sims, T. Wiedmer, H. Weiss, 1992 .

Identification of a platelet dense granule membrane protein that is deficient in a patient with the Hermansky-Pudlak syndrome.

S. Israels, P. Sims, T. Wiedmer, 1991, Blood.

Complement proteins C5b-9 cause release of membrane vesicles from the platelet surface that are enriched in the membrane receptor for coagulation factor Va and express prothrombinase activity.

P. Sims, T. Wiedmer, E. Faioni, 1988, The Journal of biological chemistry.

Caspase-Independent Exposure of Aminophospholipids and Tyrosine Phosphorylation in Bicarbonate Responsive Human Sperm Cells1

P. Sims, T. Wiedmer, B. Gadella, 2003, Biology of reproduction.

Contribution of the N-linked carbohydrate of erythrocyte antigen CD59 to its complement-inhibitory activity.

A. Bothwell, P. Sims, J. Zhao, 1992, The Journal of biological chemistry.

The human complement regulatory protein CD59 binds to the alpha-chain of C8 and to the "b"domain of C9.

P. Sims, H. Ninomiya, 1992, The Journal of biological chemistry.

Overlapping but nonidentical binding sites on CD2 for CD58 and a second ligand CD59.

W. Hahn, B. Bierer, A. Bothwell, 1992, Science.

Antileukemic roles of human phospholipid scramblase 1 gene, evidence from inducible PLSCR1-expressing leukemic cells

Y. Huang, P. Sims, T. Wiedmer, 2006, Oncogene.

Expression of the phospholipid scramblase (PLSCR) gene family during the acute phase response.

K. Feingold, C. Grunfeld, A. Moser, 2007, Biochimica et biophysica acta.

Identification of phospholipid scramblase 1 as a novel interacting molecule with beta -secretase (beta -site amyloid precursor protein (APP) cleaving enzyme (BACE)).

Y. Uchiyama, S. Waguri, S. Kametaka, 2003, The Journal of biological chemistry.

The response of human platelets to activated components of the complement system.

P. Sims, T. Wiedmer, Peter J. Sims, 1991, Immunology today.

Complement proteins C5b-9 induce secretion of high molecular weight multimers of endothelial von Willebrand factor and translocation of granule membrane protein GMP-140 to the cell surface.

P. Sims, K. Hamilton, R. McEver, 1989, The Journal of biological chemistry.

Platelet-derived microparticles express high affinity receptors for factor VIII.

B. Furie, P. Sims, T. Wiedmer, 1991, The Journal of biological chemistry.

Participation of protein kinases in complement C5b-9-induced shedding of platelet plasma membrane vesicles.

P. Sims, T. Wiedmer, 1991, Blood.

Lipoprotein and Its Apolipoproteins A-I and A-Il to Human Endothelial Cells

P. Sims, K. Hamilton, 1991 .

The terminal complement proteins C5b-9 augment binding of high density lipoprotein and its apolipoproteins A-I and A-II to human endothelial cells.

P. Sims, K. Hamilton, 1991, The Journal of clinical investigation.

On the mechanism by which complement proteins C5b-9 increase platelet prothrombinase activity.

C. Esmon, P. Sims, T. Wiedmer, 1986, The Journal of biological chemistry.

The complement-inhibitory activity of CD59 resides in its capacity to block incorporation of C9 into membrane C5b-9.

P. Sims, S. Rollins, P. J. Sims, 1990, Journal of immunology.

Mechanisms by Which Elevated Intracellular Calcium Induces S49 Cell Membranes to Become Susceptible to the Action of Secretory Phospholipase A2 *

J. D. Bell, Heather A Wilson, P. Sims, 1999, The Journal of Biological Chemistry.

Human CD59 is a receptor for the cholesterol-dependent cytolysin intermedilysin

P. Sims, Ji Zhao, R. Tweten, 2004, Nature Structural &Molecular Biology.

Effect of platelet activation on the conformation of the plasma membrane glycoprotein IIb-IIIa complex.

P. Sims, M. Ginsberg, E. Plow, 1991, The Journal of biological chemistry.

Identity of the Residues Responsible for the Species-restricted Complement Inhibitory Function of Human CD59*

P. Sims, Ji Zhao, Quansheng Zhou, 1998, The Journal of Biological Chemistry.

Structure-function relationships of the complement regulatory protein, CD59.

P. Sims, J. Zhao, N. Tweedy, 1996, Blood cells, molecules & diseases.

Regulatory control of the terminal complement proteins at the surface of human endothelial cells: neutralization of a C5b-9 inhibitor by antibody to CD59.

K. Hamilton, Z. Ji, P. Sims, 1990, Blood.

Inhibition of the complement membrane attack complex by the galactose-specific adhesion of Entamoeba histolytica.

P. Sims, T. Wiedmer, S. Eacker, 1992, The Journal of clinical investigation.

Identification of a platelet dense granule membrane protein that is deficient in a patient with the Hermansky-Pudlak syndrome.

S. Israels, P. Sims, T. Wiedmer, 1991, Blood.

Binding of anticoagulant vitamin K-dependent protein S to platelet-derived microparticles.

B. Dahlbäck, P. Sims, T. Wiedmer, 1992, Biochemistry.

Expression of recombinant CD59 with an N-terminal peptide epitope facilitates analysis of residues contributing to its complement-inhibitory function.

P. Sims, Q. Zhou, J. Zhao, 1996, Molecular immunology.

Sphingomyelin Hydrolysis to Ceramide during the Execution Phase of Apoptosis Results from Phospholipid Scrambling and Alters Cell-Surface Morphology

W. V. van Blitterswijk, J. Borst, P. Sims, 2000, The Journal of cell biology.

Complement pores in erythrocyte membranes. Analysis of C8/C9 binding required for functional membrane damage.

P. Sims, 1983, Biochimica et biophysica acta.

Analysis of solute diffusion across the C5b-9 membrane lesion of complement: evidence that individual C5b-9 complexes do not function as discrete, uniform pores.

P. Sims, P. Lauf, 1980, Journal of immunology.

Steady-state analysis of tracer exchange across the C5b-9 complement lesion in a biological membrane.

P. Sims, P. Lauf, P. J. Sims, 1978, Proceedings of the National Academy of Sciences of the United States of America.

Protein kinase C (cid:1) mediates retinoic acid and phorbol myristate acetate–induced phospholipid scramblase 1 gene expression: its role in leukemic cell differentiation

P. Sims, T. Wiedmer, Ji Zhao, 2004 .

Role of calcium and calpain in complement-induced vesiculation of the platelet plasma membrane and in the exposure of the platelet factor Va receptor.

P. Sims, T. Wiedmer, S. Shattil, 1990, Biochemistry.

Effect of complement proteins C5b-9 on blood platelets. Evidence for reversible depolarization of membrane potential.

P. Sims, T. Wiedmer, 1985, The Journal of biological chemistry.

Complement proteins C5b-9 stimulate procoagulant activity through platelet prothrombinase.

C. Esmon, P. Sims, T. Wiedmer, 1986, Blood.

Kinetic aspects of the aggregation of Clostridium perfringens theta-toxin on erythrocyte membranes. A fluorescence energy transfer study.

P. Sims, R. Tweten, R. W. Harris, 1991, The Journal of biological chemistry.

Regulation of the Tyrosine Phosphorylation of Phospholipid Scramblase 1 in Mast Cells That Are Stimulated through the High-Affinity IgE Receptor

J. Rivera, P. Sims, T. Wiedmer, 2014, PloS one.

Complement proteins C5b-9 induce transbilayer migration of membrane phospholipids.

B. W. van der Meer, P. Sims, R. Fugate, 1989, Biophysical journal.

The interaction of a Ca2+-dependent monoclonal antibody with the protein C activation peptide region. Evidence for obligatory Ca2+ binding to both antigen and antibody.

C. Esmon, N. Esmon, P. Sims, 1988, The Journal of biological chemistry.

Complement proteins C5b-9 initiate secretion of platelet storage granules without increased binding of fibrinogen or von Willebrand factor to newly expressed cell surface GPIIb-IIIa.

P. Sims, T. Wiedmer, K. Hamilton, 1988, The Journal of biological chemistry.

Reprogramming of Intestinal Glucose Metabolism and Glycemic Control in Rats After Gastric Bypass

S. Nagata, P. Sims, J. Suzuki, 2013 .

Induction of cellular procoagulant activity by the membrane attack complex of complement.

P. Sims, T. Wiedmer, 1995, Seminars in cell biology.

Production and characterization of transformed B-lymphocytes expressing the membrane defect of Scott syndrome.

J. Zhao, P. Sims, T. Wiedmer, 1994, The Journal of clinical investigation.

The complex of phosphatidylinositol 4,5-bisphosphate and calcium ions is not responsible for Ca2+-induced loss of phospholipid asymmetry in the human erythrocyte: a study in Scott syndrome, a disorder of calcium-induced phospholipid scrambling.

P. Williamson, R. Schlegel, A. Schroit, 1995, Blood.

Characterization of the complement sensitivity of paroxysmal nocturnal hemoglobinuria erythrocytes.

P. Sims, T. Wiedmer, C. Parker, 1985, The Journal of clinical investigation.

Regulatory control of complement on blood platelets. Modulation of platelet procoagulant responses by a membrane inhibitor of the C5b-9 complex.

P. Sims, T. Wiedmer, S. Rollins, 1989, The Journal of biological chemistry.

The secretory release reaction initiated by complement proteins C5b-9 occurs without platelet aggregation through glycoprotein IIb-IIIa.

P. Sims, T. Wiedmer, B. Ando, 1989, Blood.

Isolation of a tryptic fragment from Clostridium perfringens theta-toxin that contains sites for membrane binding and self-aggregation.

P. Sims, R. Tweten, R. W. Harris, 1991, The Journal of biological chemistry.

Single-channel analysis of the conductance fluctuations induced in lipid bilayer membranes by complement proteins C5b-9

R. Benz, P. Sims, T. Wiedmer, 2005, The Journal of Membrane Biology.

Inhibition of homologous complement by CD59 is mediated by a species-selective recognition conferred through binding to C8 within C5b-8 or C9 within C5b-9.

P. Sims, Ji Zhao, S. Rollins, 1991, Journal of immunology.

Protein Kinase Cδ Mediates Retinoic Acid and Phorbol Myristate Acetate-induced Phospholipid Scramblase 1 Gene Expression: Its Role in Leukemic Cell Differentiation Short tilte: PKCδ regulates PLSCR1 expression

P. Sims, T. Wiedmer, Ji Zhao, 2004 .

Assembly of the platelet prothrombinase complex is linked to vesiculation of the platelet plasma membrane. Studies in Scott syndrome: an isolated defect in platelet procoagulant activity.

C. Esmon, P. Sims, T. Wiedmer, 1989, The Journal of biological chemistry.

Stimulated secretion of endothelial von Willebrand factor is accompanied by rapid redistribution to the cell surface of the intracellular granule membrane protein GMP-140.

P. Sims, R. McEver, R P McEver, 1989, The Journal of biological chemistry.

Elevated platelet-bound IgG associated with an episode of thrombotic thrombocytopenic purpura

P. Sims, E. Boswell, 1981 .

Changes in cytosolic Ca2+ associated with von Willebrand factor release in human endothelial cells exposed to histamine. Study of microcarrier cell monolayers using the fluorescent probe indo-1.

P. Sims, K. Hamilton, 1987, The Journal of clinical investigation.

Inhibition of the complement membrane attack complex by the galactose-specific adhesin of Entamoeba histolytica.

P. Sims, T. Wiedmer, C. Pham, 1992, Archives of medical research.

Award Number: W81XWH-12-1-0139 TITLE: Loss of PEDF: A Novel Mechanism of Antihormone Resistance in Breast Cancer

J. Lewis-Wambi, P. Sims, H. Choi, 2014 .

Adiposity, dyslipidemia, and insulin resistance in mice with targeted deletion of phospholipid scramblase 3 (PLSCR3).

A. Hevener, J. Olefsky, P. Sims, 2004, Proceedings of the National Academy of Sciences of the United States of America.

Regulation of glycoprotein IIb-IIIa receptor function studied with platelets permeabilized by the pore-forming complement proteins C5b-9.

L. Brass, P. Sims, T. Wiedmer, 1992, The Journal of biological chemistry.

Complement-induced vesiculation and exposure of membrane prothrombinase sites in platelets of paroxysmal nocturnal hemoglobinuria.

T. Ortel, P. Sims, T. Wiedmer, 1993, Blood.

Unraveling the Mysteries of Phospholipid Scrambling

P. Sims, T. Wiedmer, P. J. Sims, 2001, Thrombosis and Haemostasis.

Expression of proteins controlling transbilayer movement of plasma membrane phospholipids in the B lymphocytes from a patient with Scott syndrome.

P. Sims, T. Wiedmer, Q. Zhou, 1998, Blood.

Phospholipid Scramblase 1 Potentiates the Antiviral Activity of Interferon

R. N. Harty, B. Williams, R. Silverman, 2004, Journal of Virology.

Identification of Phospholipid Scramblase 1 as a Novel Interacting Molecule with β-Secretase (β-Site Amyloid Precursor Protein (APP) Cleaving Enzyme (BACE))*

Y. Uchiyama, S. Waguri, S. Kametaka, 2003, The Journal of Biological Chemistry.

Phospholipid Scramblase 1, an interferon-regulated gene located at 3q23, is regulated by SnoN/SkiL in ovarian cancer cells

P. Spellman, Pavana Anur, P. Sims, 2013, Molecular Cancer.

Interferon-α-induced Expression of Phospholipid Scramblase 1 through STAT1 Requires the Sequential Activation of Protein Kinase Cδ and JNK*

R. Silverman, P. Sims, K. Zhao, 2005, Journal of Biological Chemistry.

Normal hemostasis but defective hematopoietic response to growth factors in mice deficient in phospholipid scramblase 1.

P. Sims, T. Wiedmer, Ji Zhao, 2002, Blood.

Suppression of ovarian carcinoma cell growth in vivo by the interferon-inducible plasma membrane protein, phospholipid scramblase 1.

R. Silverman, F. Al-Zoghaibi, D. Kushner, 2002, Cancer research.

Transcriptional control of the human plasma membranephospholipid scramblase 1 gene is mediated by interferon-α

R. Silverman, F. Al-Zoghaibi, P. Sims, 2000 .

Transcriptional control of the human plasma membrane phospholipid scramblase 1 gene is mediated by interferon-alpha.

R. Silverman, F. Al-Zoghaibi, P. Sims, 2000, Blood.

The Negative c-Myc Target Onzin Affects Proliferation and Apoptosis via Its Obligate Interaction with Phospholipid Scramblase I

E. Prochownik, P. Sims, Quansheng Zhou, 2006, Molecular and Cellular Biology.

Calcium-dependent phospholipid scrambling by TMEM16F

S. Nagata, P. Sims, J. Suzuki, 2010, Nature.

Complement C5b-9 increases plasminogen binding and activation on human endothelial cells.

P. Sims, K. Hamilton, V. Christiansen, 1997, Arteriosclerosis, thrombosis, and vascular biology.

Phosphatidylserine exposure during apoptosis is a cell-type-specific event and does not correlate with plasma membrane phospholipid scramblase expression.

S. Orrenius, B. Fadeel, Afshin Samali, 1999, Biochemical and biophysical research communications.

Complement proteins C5b-9 induce vesiculation of the endothelial plasma membrane and expose catalytic surface for assembly of the prothrombinase enzyme complex.

C. Esmon, P. Sims, K. Hamilton, 1990, The Journal of biological chemistry.

Mobilization of pro-inflammatory lipids in obese Plscr3-deficient mice

W. R. Wikoff, G. Siuzdak, Grace O'Maille, 2007, Genome Biology.

Role of a disulfide-bonded peptide loop within human complement C9 in the species-selectivity of complement inhibitor CD59.

P. Sims, T. Husler, D. H. Lockert, 1996, Biochemistry.

Identification of key regulatory pathways of myeloid differentiation using an mESC-based karyotypically normal cell model.

P. Liu, R. Karni, Jiaxi Zhou, 2012, Blood.

Complement-Induced Vesiculation and Exposure of Membrane Prothrombinase Sites in Platelets of Paroxysmal Nocturnal Hemoglobinuria

T. Ortel, P. Sims, T. Wiedmer, 1993 .

Nuclear phospholipid scramblase 1 prolongs the mitotic expansion of granulocyte precursors during G‐CSF‐induced granulopoiesis

M. Sowden, P. Sims, T. Wiedmer, 2011, Journal of leukocyte biology.

Amplified gene expression in CD59-transfected Chinese hamster ovary cells confers protection against the membrane attack complex of human complement.

S. Maher, A. Bothwell, P. Sims, 1991, The Journal of biological chemistry.

Phospholipid Scramblase 1 Contains a Nonclassical Nuclear Localization Signal with Unique Binding Site in Importin α*

G. Cingolani, G. Mitrousis, N. Walker-Kopp, 2005, Journal of Biological Chemistry.

Elimination of Potential Sites of Glycosylation Fails to Abrogate Complement Regulatory Function of Cell Surface CD59*

P. Sims, Ji Zhao, Quansheng Zhou, 1996, The Journal of Biological Chemistry.

Identity of the Segment of Human Complement C8 Recognized by Complement Regulatory Protein CD59 (*)

K. Kaufman, P. Sims, J. Sodetz, 1995, The Journal of Biological Chemistry.

Chimeras of Human Complement C9 Reveal the Site Recognized by Complement Regulatory Protein CD59 (*)

K. Kaufman, P. Sims, Thomas Hüsler, 1995, The Journal of Biological Chemistry.

Identity of a peptide domain of human C9 that is bound by the cell-surface complement inhibitor, CD59.

P. Sims, T. Wiedmer, J. Zhao, 1994, The Journal of biological chemistry.

Targeting interferon response genes sensitizes aromatase inhibitor resistant breast cancer cells to estrogen-induced cell death

J. Lewis-Wambi, P. Sims, H. Choi, 2015, Breast Cancer Research.

Phospholipid Scramblase 1 Binds to the Promoter Region of the Inositol 1,4,5-Triphosphate Receptor Type 1 Gene to Enhance Its Expression*

P. Sims, T. Wiedmer, I. Ben-Efraim, 2005, Journal of Biological Chemistry.

Plasma Membrane Phospholipid Scramblase 1 Promotes EGF-dependent Activation of c-Src through the Epidermal Growth Factor Receptor*

P. Sims, T. Wiedmer, Jun Sun, 2003, Journal of Biological Chemistry.

c-Abl Tyrosine Kinase Binds and Phosphorylates Phospholipid Scramblase 1*

M. Schwartz, Jean Y. J. Wang, P. Sims, 2001, The Journal of Biological Chemistry.

Identification of three new members of the phospholipid scramblase gene family.

P. Sims, T. Wiedmer, Q. Zhou, 2000, Biochimica et biophysica acta.

Elevated platelet-bound IgG associated with an episode of thrombotic thrombocytopenic purpura.

P. Sims, E. Boswell, 1981, Blood.

Repolarization of the membrane potential of blood platelets after complement damage: evidence for a Ca++ -dependent exocytotic elimination of C5b-9 pores.

P. Sims, T. Wiedmer, 1986, Blood.

Complement protein C9 labeled with fluorescein isothiocyanate can be used to monitor C9 polymerization and formation of the cytolytic membrane lesion.

P. Sims, 1984, Biochemistry.

Repolarization of the membrane potential of blood platelets after complement damage: evidence for a Ca++ -dependent exocytotic elimination of C5b-9 pores

P. Sims, T. Wiedmer, 1986 .

Defective Ca(2+)-induced microvesiculation and deficient expression of procoagulant activity in erythrocytes from a patient with a bleeding disorder: a study of the red blood cells of Scott syndrome.

P. Sims, T. Wiedmer, H. Weiss, 1992, Blood.

Interaction between apolipoproteins A-I and A-II and the membrane attack complex of complement. Affinity of the apoproteins for polymeric C9.

Jing Zhao, P. Sims, K. Hamilton, 1993, The Journal of biological chemistry.

Phospholipid Scramblase-1-Induced Lipid Reorganization Regulates Compensatory Endocytosis in Neuroendocrine Cells

Y. Bailly, S. Chasserot-Golaz, M. Bader, 2013, The Journal of Neuroscience.

Complement C5b-9-stimulated platelet secretion is associated with a Ca2+-initiated activation of cellular protein kinases.

P. Sims, T. Wiedmer, B. Ando, 1987, The Journal of biological chemistry.

Contribution of platelet microparticle formation and granule secretion to the transmembrane migration of phosphatidylserine.

P. Sims, T. Wiedmer, J. Zhao, 1993, The Journal of biological chemistry.

Phosphatidylserine externalization and membrane blebbing are involved in the nonclassical export of FGF1

Courtney M. Mazur, F. Tarantini, P. Sims, 2012, Journal of cellular biochemistry.

Targeting interferon response genes sensitizes aromatase inhibitor resistant breast cancer cells to estrogen-induced cell death

J. Lewis-Wambi, P. Sims, H. Choi, 2015, Breast Cancer Research.

Phospholipid scramblases and Tubby-like proteins belong to a new superfamily of membrane tethered transcription factors

Robert D. Finn, Alex Bateman, Johannes Söding, 2008, Bioinform..

The influence of electrochemical gradients of Na+ and K+ upon the membrane binding and pore forming activity of the terminal complement proteins

P. Sims, T. Wiedmer, 2005, The Journal of Membrane Biology.

Protection of porcine aortic endothelial cells from complement-mediated cell lysis and activation by recombinant human CD59.

A. Bothwell, P. Sims, S. Kennedy, 1994, Transplantation.

The metabolic activation of 7‐methylbenz(a)anthracene: The induction of malignant transformation and mutation in mammalian cells by non‐K‐region dihydrodiols

P. Sims, H. Marquardt, P. Grover, 1977, International journal of cancer.

Sphingomyelin Hydrolysis to Ceramide during the Execution Phase of Apoptosis Results from Phospholipid Scrambling and Alters Cell-Surface Morphology

W. V. van Blitterswijk, J. Borst, P. Sims, 2000, The Journal of cell biology.

Mutagenicity of isomeric diol-epoxides of benzo[a]pyrene and benz[a]anthracene in S. typhimurium TA98 and TA100 and in V79 Chinese hamster cells.

H. Bartsch, T. Kuroki, P. Sims, 1977, Mutation research.

Mobilization of pro-inflammatory lipids in obese Plscr3-deficient mice

W. R. Wikoff, G. Siuzdak, Grace O'Maille, 2007, Genome Biology.

Phospholipid Scramblase 1, an interferon-regulated gene located at 3q23, is regulated by SnoN/SkiL in ovarian cancer cells

P. Spellman, Pavana Anur, P. Sims, 2013, Molecular Cancer.

Protein kinase C (cid:1) mediates retinoic acid and phorbol myristate acetate–induced phospholipid scramblase 1 gene expression: its role in leukemic cell differentiation

P. Sims, T. Wiedmer, Ji Zhao, 2022 .

Interferon-alpha-induced expression of phospholipid scramblase 1 through STAT1 requires the sequential activation of protein kinase Cdelta and JNK.

R. Silverman, P. Sims, K. Zhao, 2005, The Journal of biological chemistry.

Calcium-dependent phospholipid scrambling by TMEM16F

S. Nagata, P. Sims, M. Umeda, 2010, Nature.

The enzyme-catalysed conversion of anti-benzo[a]pyrene-7,8-diol 9,10-oxide into a glutathione conjugate.

A. Hewer, P. Sims, C. S. Cooper, 1980, Carcinogenesis.

Polycyclic hydrocarbon activation and metabolism in epithelial cell aggregates prepared from human mammary tissue

A. Neville, P. Sims, P. Grover, 1980, International journal of cancer.

Nuclear phospholipid scramblase 1 prolongs the mitotic expansion of granulocyte precursors during G‐CSF‐induced granulopoiesis

M. Sowden, P. Sims, T. Wiedmer, 2011, Journal of leukocyte biology.