N. J. Gibbons

发表

Computer simulation of native epidermal enzyme structures in the presence and absence of hydrogen peroxide (H2O2): potential and pitfalls.

N. J. Gibbons, J. Wood, H. Rokos, 2006, The Journal of investigative dermatology.

Structural and functional alterations in the β2‐adrenoceptor are caused by a point mutation in patients with atopic eczema

M. Pittelkow, N. J. Gibbons, J. Wood, 2007, Experimental dermatology.

The Ca2+ binding capacity of epidermal furin is disrupted by H2O2-mediated oxidation in vitiligo

N. J. Gibbons, K. Schallreuter, M. Böhm, 2008 .

Molecular modeling of human chorionic gonadotropin glycosylation, nicking, and carboxyl terminal extension

N. J. Gibbons, R. Iles, S. Butler, 2020 .

The Ca2+-binding capacity of epidermal furin is disrupted by H2O2-mediated oxidation in vitiligo.

N. J. Gibbons, K. Schallreuter, M. Böhm, 2008, Endocrinology.

In situ and in vitro evidence for DCoH/HNF-1 alpha transcription of tyrosinase in human skin melanocytes.

N. J. Gibbons, J. Wood, K. Schallreuter, 2003, Biochemical and biophysical research communications.

Presence of epidermal allantoin further supports oxidative stress in vitiligo

N. J. Gibbons, J. Wood, H. Rokos, 2008, Experimental dermatology.

Methionine sulfoxide reductases A and B are deactivated by hydrogen peroxide (H2O2) in the epidermis of patients with vitiligo.

N. J. Gibbons, J. Wood, H. Rokos, 2008, The Journal of investigative dermatology.

Hydrogen peroxide-mediated oxidative stress disrupts calcium binding on calmodulin: more evidence for oxidative stress in vitiligo.

N. J. Gibbons, J. Wood, K. Schallreuter, 2007, Biochemical and biophysical research communications.

Calcium-activated butyrylcholinesterase in human skin protects acetylcholinesterase against suicide inhibition by neurotoxic organophosphates.

N. J. Gibbons, J. Wood, K. Schallreuter, 2007, Biochemical and biophysical research communications.

KdPT, a Tripeptide Derivative of α-Melanocyte–Stimulating Hormone, Suppresses IL-1β–Mediated Cytokine Expression and Signaling in Human Sebocytes

M. Picardo, T. Luger, C. Zouboulis, 2010, The Journal of Immunology.

Enhanced DNA binding capacity on up‐regulated epidermal wild‐type p53 in vitiligo by H2O2‐mediated oxidation: a possible repair mechanism for DNA damage

N. J. Gibbons, K. Schallreuter, M. Shalbaf, 2009, FASEB journal : official publication of the Federation of American Societies for Experimental Biology.

Blunted epidermal L‐tryptophan metabolism in vitiligo affects immune response and ROS scavenging by Fenton chemistry, part 2: epidermal H2O2/ONOO–‐mediated stress in vitiligo hampers indoleamine 2,3‐dioxygenase and aryl hydrocarbon receptor‐mediated immune response signaling

N. J. Gibbons, K. Schallreuter, E. Marsch, 2012, FASEB journal : official publication of the Federation of American Societies for Experimental Biology.

Oxidative stress via hydrogen peroxide affects proopiomelanocortin peptides directly in the epidermis of patients with vitiligo.

E. Peters, N. J. Gibbons, J. Wood, 2007, The Journal of investigative dermatology.

Blunted epidermal L‐tryptophan metabolism in vitiligo affects immune response and ROS scavenging by Fenton chemistry, part 1: epidermal H2O2/ONOO–‐mediated stress abrogates tryptophan hydroxylase and dopa decarboxylase activities, leading to low serotonin and melatonin levels

Aurora Martínez, N. J. Gibbons, H. Rokos, 2012, FASEB journal : official publication of the Federation of American Societies for Experimental Biology.

Butyrylcholinesterase is present in the human epidermis and is regulated by H2O2: more evidence for oxidative stress in vitiligo.

N. J. Gibbons, J. Wood, H. Rokos, 2006, Biochemical and biophysical research communications.

Perturbed 6-tetrahydrobiopterin recycling via decreased dihydropteridine reductase in vitiligo: more evidence for H2O2 stress.

N. J. Gibbons, H. Rokos, K. Schallreuter, 2004, The Journal of investigative dermatology.

α‐MSH Can Control the Essential Cofactor 6‐Tetrahydrobiopterin in Melanogenesis

D. Tobin, N. J. Gibbons, J. Wood, 1999, Annals of the New York Academy of Sciences.

Human phenylalanine hydroxylase is activated by H2O2: a novel mechanism for increasing the L-tyrosine supply for melanogenesis in melanocytes.

U. Wazir, N. J. Gibbons, J. Wood, 2004, Biochemical and biophysical research communications.

Activation/deactivation of acetylcholinesterase by H2O2: more evidence for oxidative stress in vitiligo.

N. J. Gibbons, J. Wood, H. Rokos, 2004, Biochemical and biophysical research communications.