L. Roussel

发表

The Mycobacterium tuberculosis Cell-surface Glycoprotein Apa as a Potential Adhesin to Colonize Target Cells via the Innate Immune System Pulmonary C-type Lectin Surfactant Protein A*

M. Rivière, G. Puzo, A. Ragas, 2007, Journal of Biological Chemistry.

The level of p38α mitogen-activated protein kinase activation in airway epithelial cells determines the onset of innate immune responses to planktonic and biofilm Pseudomonas aeruginosa.

M. Desrosiers, L. Roussel, S. Rousseau, 2013, The Journal of infectious diseases.

Steroid-insensitive ERK1/2 activity drives CXCL8 synthesis and neutrophilia by airway smooth muscle.

Q. Hamid, R. Olivenstein, L. Roussel, 2011, American journal of respiratory cell and molecular biology.

IL-17 Promotes p38 MAPK-Dependent Endothelial Activation Enhancing Neutrophil Recruitment to Sites of Inflammation

Q. Hamid, J. Huot, F. Houle, 2010, The Journal of Immunology.

Vaccine breakthrough hypoxemic COVID-19 pneumonia in patients with auto-Abs neutralizing type I IFNs

Mark S. Anderson, Chun Jimmie Ye, Gabriela K. Fragiadakis, 2022, Science immunology.

Differential Contribution of the Aryl-Hydrocarbon Receptor and Toll-Like Receptor Pathways to IL-8 Expression in Normal and Cystic Fibrosis Airway Epithelial Cells Exposed to Pseudomonas aeruginosa

C. Baglole, L. Roussel, S. Rousseau, 2016, Front. Cell Dev. Biol..

Real-world serologic responses to Extended-interval and Heterologous COVID-19 mRNA vaccination in Frail Elderly - Interim report from a prospective observational cohort study

D. Vinh, X. Zhang, Y. Sun, 2021, medRxiv.

The aryl hydrocarbon receptor suppresses cigarette-smoke-induced oxidative stress in association with dioxin response element (DRE)-independent regulation of sulfiredoxin 1.

Q. Hamid, P. Nair, C. Baglole, 2015, Free radical biology & medicine.

P. aeruginosa drives CXCL8 synthesis via redundant toll-like receptors and NADPH oxidase in CFTR∆F508 airway epithelial cells.

L. Roussel, S. Rousseau, J. Bérubé, 2011, Journal of cystic fibrosis : official journal of the European Cystic Fibrosis Society.

TH17 cytokines induce human airway smooth muscle cell migration.

Q. Hamid, A. Halayko, D. Eidelman, 2011, The Journal of allergy and clinical immunology.

The risk of COVID-19 death is much greater and age dependent with type I IFN autoantibodies

P. Gregersen, J. Casanova, L. Rowen, 2022, Proceedings of the National Academy of Sciences of the United States of America.

The risk of COVID-19 death is much greater and age-dependent with type I IFN autoantibodies

Mark S. Anderson, J. Casanova, L. Rowen, 2022, Research square.

IL-17 primes airway epithelial cells lacking functional Cystic Fibrosis Transmembrane conductance Regulator (CFTR) to increase NOD1 responses.

L. Roussel, S. Rousseau, Lucie Roussel, 2010, Biochemical and biophysical research communications.

Molecular mimicry between IL‐33 and KSHV for attachment to chromatin through the H2A–H2B acidic pocket

L. Roussel, J. Girard, C. Cayrol, 2008, EMBO reports.

IL-33 is expressed in epithelia from patients with cystic fibrosis and potentiates neutrophil recruitment.

L. Roussel, S. Rousseau, R. Farias, 2013, The Journal of allergy and clinical immunology.

Decreasing SMPD1 activity in BEAS-2B bronchial airway epithelial cells results in increased NRF2 activity, cytokine synthesis and neutrophil recruitment.

L. Roussel, S. Rousseau, G. Martel, 2017, Biochemical and biophysical research communications.

Autoantibodies against type I IFNs in patients with life-threatening COVID-19

Steven M. Holland, Amelia Licari, Gian Luigi Marseglia, 2020, Science.