P. M. Yao

发表

Free Cholesterol-loaded Macrophages Are an Abundant Source of Tumor Necrosis Factor-α and Interleukin-6

David A. Brenner, R. Schwabe, D. Brenner, 2005, Journal of Biological Chemistry.

Free Cholesterol Loading of Macrophages Is Associated with Widespread Mitochondrial Dysfunction and Activation of the Mitochondrial Apoptosis Pathway*

P. M. Yao, I. Tabas, 2001, The Journal of Biological Chemistry.

Human Cathepsin F

H. Chapman, D. Brömme, P. M. Yao, 1998, The Journal of Biological Chemistry.

Free Cholesterol Loading of Macrophages Induces Apoptosis Involving the Fas Pathway*

P. M. Yao, I. Tabas, 2000, The Journal of Biological Chemistry.

Dok1 and SHIP Act as Negative Regulators of v-Abl-Induced Pre-B Cell Transformation, Proliferation and Ras/Erk Activation

P. Pandolfi, P. Rothman, P. M. Yao, 2005, Cell cycle.

Divergent regulation of 92-kDa gelatinase and TIMP-1 by HBECs in response to IL-1β and TNF-α.

J. Buhler, C. Delacourt, A. Harf, 1997, American journal of physiology. Lung cellular and molecular physiology.

Expression of Matrix Metalloproteinase Gelatinases A and B by Cultured Epithelial Cells from Human Bronchial Explants*

J. Buhler, A. Harf, M. d’Ortho, 1996, The Journal of Biological Chemistry.

Divergent regulation of 92-kDa gelatinase and TIMP-1 by HBECs in response to IL-1beta and TNF-alpha.

J. Buhler, C. Delacourt, A. Harf, 1997, The American journal of physiology.

Balance between MMP‐9 and TIMP‐1 Expressed by Human Bronchial Epithelial Cells: Relevance to Asthma

M. d’Ortho, B. Wallaert, P. M. Yao, 1999, Annals of the New York Academy of Sciences.

Macrophages deficient in CTP:Phosphocholine cytidylyltransferase-alpha are viable under normal culture conditions but are highly susceptible to free cholesterol-induced death. Molecular genetic evidence that the induction of phosphatidylcholine biosynthesis in free cholesterol-loaded macrophages is

P. M. Yao, I. Tabas, S. Jackowski, 2000, The Journal of biological chemistry.

Brief Report: Increased Apoptosis in Advanced Atherosclerotic Lesions of Apoe−/− Mice Lacking Macrophage Bcl-2

E. Fisher, Yankun Li, P. M. Yao, 2009, Arteriosclerosis, thrombosis, and vascular biology.

The endoplasmic reticulum is the site of cholesterol-induced cytotoxicity in macrophages

George Kuriakose, E. Fisher, A. Marks, 2003, Nature Cell Biology.

Macrophages Deficient in CTP:Phosphocholine Cytidylyltransferase-α Are Viable under Normal Culture Conditions but Are Highly Susceptible to Free Cholesterol-induced Death

P. M. Yao, I. Tabas, S. Jackowski, 2000, The Journal of Biological Chemistry.

Free cholesterol-loaded macrophages are an abundant source of tumor necrosis factor-alpha and interleukin-6: model of NF-kappaB- and map kinase-dependent inflammation in advanced atherosclerosis.

R. Schwabe, D. Brenner, A. Tall, 2005, The Journal of biological chemistry.

Cholesterol-induced macrophage apoptosis requires ER stress pathways and engagement of the type A scavenger receptor

Yibin Wang, R. Flavell, Roger J. Davis, 2005, The Journal of cell biology.

Cell-matrix interactions modulate 92-kD gelatinase expression by human bronchial epithelial cells.

A. Harf, M. d’Ortho, C. Delclaux, 1998, American journal of respiratory cell and molecular biology.

Alveolar epithelial cells in vitro produce gelatinases and tissue inhibitor of matrix metalloproteinase-2.

C. Delacourt, A. Harf, M. d’Ortho, 1997, The American journal of physiology.

Human Cathepsin F. Molecular Cloning, Functional Expression, Tissue Localization, and Enzymatic Characterization

H. Chapman, D. Brömme, P. M. Yao, 1998 .

Cholesterol-induced macrophage apoptosis requires ER stress pathways and engagement of the type A scavenger receptor

R. Flavell, Yibin Wang, Yankun Li, 2005, The Journal of cell biology.