J. Boehm

发表

Glutamate Receptor Exocytosis and Spine Enlargement during Chemically Induced Long-Term Potentiation

Roberto Malinow, Bo Li, Charles D. Kopec, 2006, The Journal of Neuroscience.

AMPAR Removal Underlies Aβ-Induced Synaptic Depression and Dendritic Spine Loss

R. Malinow, T. Iwatsubo, C. Sato, 2006, Neuron.

A ‘danse macabre’: tau and Fyn in STEP with amyloid beta to facilitate induction of synaptic depression and excitotoxicity

J. Boehm, Jannic Boehm, 2013, The European journal of neuroscience.

AMPA receptor phosphorylation during synaptic plasticity.

R. Malinow, J. Boehm, J Boehm, 2005, Biochemical Society transactions.

Synaptic Incorporation of AMPA Receptors during LTP Is Controlled by a PKC Phosphorylation Site on GluR1

Roberto Malinow, Richard L. Huganir, Myoung-Goo Kang, 2006, Neuron.

NMDA receptor–dependent dephosphorylation of serine 387 in Argonaute 2 increases its degradation and affects dendritic spine density and maturation

J. Boehm, Nicolas Paradis-Isler, Jannic Boehm, 2018, The Journal of Biological Chemistry.

Interaction of Endogenous Tau Protein with Synaptic Proteins Is Regulated by N-Methyl-d-aspartate Receptor-dependent Tau Phosphorylation*

N. Leclerc, J. Boehm, Emilie Trillaud-Doppia, 2012, The Journal of Biological Chemistry.

Amyloid Beta and Tau Proteins as Therapeutic Targets for Alzheimer's Disease Treatment: Rethinking the Current Strategy

Xiongwei Zhu, J. Boehm, S. Mondragón-Rodríguez, 2012, International journal of Alzheimer's disease.

Regulation of BOB.1/OBF.1 stability by SIAH

L. Staudt, T. Wirth, A. Greiner, 2001, The EMBO journal.

The Amyloid Precursor Protein Intracellular Domain Is an Effector Molecule of Metaplasticity

J. Boehm, Emilie Trillaud-Doppia, Jannic Boehm, 2016, Biological Psychiatry.

A single amino acid difference between the intracellular domains of amyloid precursor protein and amyloid-like precursor protein 2 enables induction of synaptic depression and block of long-term potentiation

J. Boehm, Emilie Trillaud-Doppia, Nicolas Paradis-Isler, 2016, Neurobiology of Disease.

RCAN1 Regulates Bidirectional Synaptic Plasticity

J. Boehm, Emilie Trillaud-Doppia, Anthony Dudilot, 2020, Current Biology.

NMDA-Receptor Activation but Not Ion Flux Is Required for Amyloid-Beta Induced Synaptic Depression

J. Boehm, Sara J. Licea, Anthony Dudilot, 2013, PloS one.

Two mutations preventing PDZ-protein interactions of GluR1 have opposite effects on synaptic plasticity.

R. Malinow, I. Ehrlich, J. Boehm, 2006, Learning & memory.