Qianlei Yang

发表

Feedback circuitry via let-7c between lncRNA CCAT1 and c-Myc is involved in cigarette smoke extract-induced malignant transformation of HBE cells

Yu Qin, Lu Lu, Junchao Xue, 2017, Oncotarget.

CircLRP6 Regulation of ZEB1 via miR-455 Is Involved in the Epithelial-Mesenchymal Transition During Arsenite-Induced Malignant Transformation of Human Keratinocytes

Jiachun Lu, Ping Yang, Qizhan Liu, 2018, Toxicological sciences : an official journal of the Society of Toxicology.

In type 2 diabetes induced by cigarette smoking, activation of p38 MAPK is involved in pancreatic β-cell apoptosis

Yu Qin, Jiachun Lu, Ping Yang, 2018, Environmental Science and Pollution Research.

MicroRNA-21 activation of ERK signaling via PTEN is involved in arsenite-induced autophagy in human hepatic L-02 cells.

Qizhan Liu, Jun Li, Lu Lu, 2016, Toxicology letters.

Possible differences in the mechanism of malignant transformation of HaCaT cells by arsenite and its dimethyl metabolites, particularly dimethylthioarsenics.

Qianlei Yang, Y. An, K. Yamanaka, 2020, Journal of trace elements in medicine and biology : organ of the Society for Minerals and Trace Elements.

Impaired autophagic flux and p62‐mediated EMT are involved in arsenite‐induced transformation of L‐02 cells

Jiachun Lu, Ping Yang, Q. Bian, 2017, Toxicology and applied pharmacology.

Long-term arsenite exposure decreases autophagy by increased release of Nrf2 in transformed human keratinocytes.

Qianlei Yang, Jie Zhang, Y. An, 2020, The Science of the total environment.

The protective effect of silk fibroin on high glucose induced insulin resistance in HepG2 cells.

Qianlei Yang, Jie Zhang, Heran Li, 2019, Environmental toxicology and pharmacology.

The Potential Key Role of the NRF2/NQO1 Pathway in the Health Effects of Arsenic Pollution on SCC

Qianlei Yang, Jie Zhang, Hengdong Zhang, 2022, International journal of environmental research and public health.

miR-149 Negative Regulation of mafA Is Involved in the Arsenite-Induced Dysfunction of Insulin Synthesis and Secretion in Pancreatic Beta Cells

Xingfen Yang, Qizhan Liu, Hui Xu, 2018, Toxicological sciences : an official journal of the Society of Toxicology.

Epigenetic silencing of miR-218 by the lncRNA CCAT1, acting via BMI1, promotes an altered cell cycle transition in the malignant transformation of HBE cells induced by cigarette smoke extract.

Qizhan Liu, Lu Lu, Fei Luo, 2016, Toxicology and applied pharmacology.

NF‐κB‐regulated miR‐155, via repression of QKI, contributes to the acquisition of CSC‐like phenotype during the neoplastic transformation of hepatic cells induced by arsenite

Jiachun Lu, Ping Yang, Qizhan Liu, 2018, Molecular carcinogenesis.

MicroRNA-191 blocking the translocation of GLUT4 is involved in arsenite-induced hepatic insulin resistance through inhibiting the IRS1/AKT pathway.

Qizhan Liu, Huanwen Tang, Qianlei Yang, 2021, Ecotoxicology and environmental safety.

Exosomal circRNA_100284 from arsenite-transformed cells, via microRNA-217 regulation of EZH2, is involved in the malignant transformation of human hepatic cells by accelerating the cell cycle and promoting cell proliferation

Qizhan Liu, Fei Luo, Hui Xu, 2018, Cell Death & Disease.

MALAT1 via microRNA‐17 regulation of insulin transcription is involved in the dysfunction of pancreatic β‐cells induced by cigarette smoke extract

Jiachun Lu, Ping Yang, Qizhan Liu, 2018, Journal of Cellular Physiology.

Role of GH/IGF axis in arsenite-induced developmental toxicity in zebrafish embryos.

Qianlei Yang, Jie Zhang, Heran Li, 2020, Ecotoxicology and environmental safety.

Lead acetate induces cartilage defects and bone loss in zebrafish embryos by disrupting the GH/IGF-1 axis.

Qianlei Yang, Hengdong Zhang, Heran Li, 2023, Ecotoxicology and environmental safety.

Melatonin Prevents NaAsO2-Induced Developmental Cardiotoxicity in Zebrafish through Regulating Oxidative Stress and Apoptosis

Wenjun Ding, Qianlei Yang, Hengdong Zhang, 2022, Antioxidants.

Circ100284, via miR-217 regulation of EZH2, is involved in the arsenite-accelerated cell cycle of human keratinocytes in carcinogenesis.

Qizhan Liu, Lu Lu, Fei Luo, 2017, Biochimica et biophysica acta. Molecular basis of disease.

Qianlei Yang, Y. An, K. Yamanaka, 2019, Free radical research.

Circ008913, via miR-889 regulation of DAB2IP/ZEB1, is involved in the arsenite-induced acquisition of CSC-like properties by human keratinocytes in carcinogenesis.

Qizhan Liu, Huanwen Tang, Fei Luo, 2018, Metallomics : integrated biometal science.

MicroRNA-218 acts by repressing TNFR1-mediated activation of NF-κB, which is involved in MUC5AC hyper-production and inflammation in smoking-induced bronchiolitis of COPD.

Jiachun Lu, Ping Yang, Qizhan Liu, 2017, Toxicology letters.

Involvement of HIF-1α-regulated miR-21, acting via the Akt/NF-κB pathway, in malignant transformation of HBE cells induced by cigarette smoke extract.

Jiachun Lu, Ping Yang, Qizhan Liu, 2018, Toxicology letters.

NF-kB-regulated exosomal miR-155 promotes the inflammation associated with arsenite carcinogenesis.

Qizhan Liu, Jun Li, Lu Lu, 2017, Cancer letters.

MicroRNA-191, acting via the IRS-1/Akt signaling pathway, is involved in the hepatic insulin resistance induced by cigarette smoke extract

Qizhan Liu, Lu Lu, Fei Luo, 2018, Environmental Science and Pollution Research.

Qizhan Liu, Fei Luo, Hui Xu, 2018, Cell Death & Disease.

MicroRNA-191, regulated by HIF-2α, is involved in EMT and acquisition of a stem cell-like phenotype in arsenite-transformed human liver epithelial cells.

Jiachun Lu, Ping Yang, Qizhan Liu, 2017, Toxicology in vitro : an international journal published in association with BIBRA.

Role of mitochondrial DNA in oxidative damage induced by sodium arsenite in human bronchial epithelial cells

Qianlei Yang, J. Tong, Jie Zhang, 2019, Journal of toxicology and environmental health. Part A.

Reductive stress induced by NRF2/G6PD through glucose metabolic reprogramming promotes malignant transformation in Arsenite-exposed human keratinocytes.

Qianlei Yang, Jie Zhang, Rui Yan, 2023, The Science of the total environment.