J. Bundzikova

发表

Role of Prefrontal Cortex Glucocorticoid Receptors in Stress and Emotion

James P. Herman, J. Herman, J. McKlveen, 2013, Biological Psychiatry.

Different antipsychotics elicit different effects on magnocellular oxytocinergic and vasopressinergic neurons as revealed by Fos immunohistochemistry

J. Bundzikova, A. Kiss, J. Mikkelsen, 2009, Journal of neuroscience research.

Activation of Different Neuronal Phenotypes in the Rat Brain Induced by Liver Ischemia–Reperfusion Injury: Dual Fos/Neuropeptide Immunohistochemistry

J. Bundzikova, A. Kiss, B. Mravec, 2011, Cellular and Molecular Neurobiology.

Response of Substances Co-Expressed in Hypothalamic Magnocellular Neurons to Osmotic Challenges in Normal and Brattleboro Rats

J. Bundzikova, A. Kiss, J. Mikkelsen, 2008, Cellular and Molecular Neurobiology.

GABA Regulates the Rat Hypothalamic‐Pituitary‐Adrenocortical Axis via Different GABA‐A Receptor α‐Subtypes

J. Bundzikova, A. Kiss, J. Mikkelsen, 2008, Annals of the New York Academy of Sciences.

Ghrelin agonists impact on Fos protein expression in brain areas related to food intake regulation in male C57BL/6 mice

J. Fehrentz, J. Bundzikova, A. Kiss, 2011, Neurochemistry International.

Brain response to induced peripheral cancer development in rats: dual fos-tyrosine hydroxylase and fos-oxytocin immunohistochemistry.

J. Bundzikova, A. Kiss, I. Hulín, 2009, Endocrine regulations.

The α2-adrenoceptors do not modify the activity of tyrosine hydroxylase, corticoliberine, and neuropeptide Y producing hypothalamic magnocellular neurons ion the Long Evans and Brattleboro rats.

J. Bundzikova, A. Kiss, J. Mikkelsen, 2010, Journal of physiology and pharmacology : an official journal of the Polish Physiological Society.

α2-Adrenergic Impact on Hypothalamic Magnocellular Oxytocinergic Neurons in Long Evans and Brattleboro Rats: Effects of Agonist and Antagonists

J. Bundzikova, A. Kiss, J. Mikkelsen, 2009, Cellular and Molecular Neurobiology.

Effect of Liver Ischemia–Reperfusion Injury on the Activity of Neurons in the Rat Brain

J. Bundzikova, A. Kiss, B. Mravec, 2009, Cellular and Molecular Neurobiology.

Response of hypothalamic oxytocinergic neurons to immobilization stress is not dependent on the presence of corticotrophin releasing hormone (CRH): a CRH knock-out mouse study.

R. Kvetňanský, J. Bundzikova, A. Kiss, 2009, Journal of physiology and pharmacology : an official journal of the Polish Physiological Society.

Chemical sympathectomy suppresses fibrosarcoma development and improves survival of tumor-bearing rats.

J. Bundzikova, A. Kiss, B. Mravec, 2011, Neoplasma.

Fos expression in tyrosine hydroxylase containing hypothalamic neurons in CRH-KO mice: effect of immobilization stress.

R. Kvetňanský, J. Bundzikova, A. Kiss, 2010, Endocrine regulations.

Fos expression in hypocretinergic neurons in C57B1/6 male and female mice after long-term consumption of high fat diet.

J. Bundzikova, A. Kiss, J. Mikkelsen, 2008, Endocrine regulations.

Zolpidem, a selective GABAA receptor α1 subunit agonist, induces comparable Fos expression in oxytocinergic neurons of the hypothalamic paraventricular and accessory but not supraoptic nuclei in the rat

J. Bundzikova, A. Kiss, J. Mikkelsen, 2006, Brain Research Bulletin.

Improvement in the evaluation of Fos-neuropeptide colocalization by employing free-floating cryosections of delicate thickness: double and triple colored immunohistochemistry.

J. Bundzikova, A. Kiss, Z. Pirník, 2007, Endocrine regulations.

Activity of Brain Stem Groups of Catecholaminergic Cells in Tumor‐Bearing Rats

J. Bundzikova, A. Kiss, I. Hulín, 2008, Annals of the New York Academy of Sciences.

Different antipsychotics elicit different effects on magnocellular oxytocinergic and vasopressinergic neurons as revealed by Fos immunohistochemistry

J. Bundzikova, Z. Pirnik, Alexander Kiss, 2009, Journal of neuroscience research.

Activity of Oxytocinergic Neurons in the Supraoptic Nucleus under Stimulation of α2‐Adrenoceptors in Brattleboro Rats

J. Bundzikova, A. Kiss, J. Mikkelsen, 2008, Annals of the New York Academy of Sciences.