J. Rong

发表

Reversal of Hyperlipidemia With a Genetic Switch Favorably Affects the Content and Inflammatory State of Macrophages in Atherosclerotic Plaques

S. Young, E. Fisher, Y. Vengrenyuk, 2011, Circulation.

Dramatic remodeling of advanced atherosclerotic plaques of the apolipoprotein E-deficient mouse in a novel transplantation model.

Z. Fayad, J. Aguinaldo, J. Li, 2001, Journal of vascular surgery.

Serial, noninvasive, in vivo magnetic resonance microscopy detects the development of atherosclerosis in apolipoprotein E‐deficient mice and its progression by arterial wall remodeling

Z. Fayad, J. Aguinaldo, E. Fisher, 2003, Journal of magnetic resonance imaging : JMRI.

Laser capture microdissection analysis of gene expression in macrophages from atherosclerotic lesions of apolipoprotein E-deficient mice

E. Fisher, J. Breslow, R. Choudhury, 2002, Proceedings of the National Academy of Sciences of the United States of America.

Atherosclerotic lesions in genetically modified mice quantified in vivo by non-invasive high-resolution magnetic resonance microscopy.

Zahi A Fayad, Valentin Fuster, V. Fuster, 2002, Atherosclerosis.

A transplant model to study the long term effects of apoprotein AI overexpression on large, complex atherosclerotic lesions in ApoE-deficient mice

E. Fisher, J. Fallon, R. Choudhury, 2000 .

Plaque remodeling in mouse atherosclerosis identified by high resolution in vivo magnetic resonance imaging (MRM).

Z. Fayad, J. Aguinaldo, E. Fisher, 2001 .

Serial, non-invasive, high resolution magnetic resonance microscopy measures progression of atherosclerosis in apolipoprotein E deficient mice

V. Fuster, Z. Fayad, E. Fisher, 2001 .

Arterial injury by cholesterol oxidation products causes endothelial dysfunction and arterial wall cholesterol accumulation.

A. Sevanian, G. Chisolm, H. Hodis, 1998, Arteriosclerosis, thrombosis, and vascular biology.

Comparison of mitochondrial and macrophage content between subcutaneous and visceral fat in db/db mice.

M. McVey, Michael K. Hansen, J. Rong, 2007, Experimental and molecular pathology.

Remodel Lesions Without Suppressing Indices of Inflammation or Oxidation High-Density Lipoproteins Retard the Progression of Atherosclerosis and Favorably

E. Fisher, J. Fallon, J. Breslow, 2004 .

ACAT Inhibition Reduces the Progression of Preexisting, Advanced Atherosclerotic Mouse Lesions Without Plaque or Systemic Toxicity

Christine M. Miller, E. Fisher, J. Rong, 2013, Arteriosclerosis, thrombosis, and vascular biology.

Elevating High-Density Lipoprotein Cholesterol in Apolipoprotein E—Deficient Mice Remodels Advanced Atherosclerotic Lesions by Decreasing Macrophage and Increasing Smooth Muscle Cell Content

E. Fisher, J. Fallon, J. Breslow, 2001, Circulation.

Severity of atherosclerotic lesions in ApoE-/- mice assessed by noninvasive in vivo high resolution magnetic resonance microscopy

Z. Fayad, E. Fisher, J. Fallon, 2000 .

Acyl-CoenzymeA (CoA):Cholesterol Acyltransferase Inhibition in Rat and Human Aortic Smooth Muscle Cells Is Nontoxic and Retards Foam Cell Formation

E. Fisher, J. Rong, S. Sturley, 2004, Arteriosclerosis, thrombosis, and vascular biology.

Brief Report: Increased Apoptosis in Advanced Atherosclerotic Lesions of Apoe−/− Mice Lacking Macrophage Bcl-2

E. Fisher, Yankun Li, P. M. Yao, 2009, Arteriosclerosis, thrombosis, and vascular biology.

Transdifferentiation of mouse aortic smooth muscle cells to a macrophage-like state after cholesterol loading

E. Fisher, J. Rong, M. Shapiro, 2003, Proceedings of the National Academy of Sciences of the United States of America.

Electrophysiological Characterization of Human and Mouse Sodium-Dependent Citrate Transporters (NaCT/SLC13A5) Reveal Species Differences with Respect to Substrate Sensitivity and Cation Dependence

E. Sher, J. Rong, R. Zwart, 2015, The Journal of Pharmacology and Experimental Therapeutics.

Defective enamel and bone development in sodium-dependent citrate transporter (NaCT) Slc13a5 deficient mice

J. Rong, Y. Ma, A. Irizarry, 2017, PloS one.

Selective class IIa histone deacetylase inhibition via a nonchelating zinc-binding group.

R. A. Reid, M. Head, E. Baloglu, 2013, Nature chemical biology.

The endoplasmic reticulum is the site of cholesterol-induced cytotoxicity in macrophages

George Kuriakose, E. Fisher, A. Marks, 2003, Nature Cell Biology.

Carboxyl Ester Lipase

E. Fisher, J. Rong, 2002 .

Lysophosphatidylcholine Stimulates Monocyte Chemoattractant Protein-1 Gene Expression in Rat Aortic Smooth Muscle Cells

E. Fisher, J. Berman, J. Rong, 2002, Arteriosclerosis, thrombosis, and vascular biology.

Dyslipidemia associated with atherosclerotic disease systemically alters dendritic cell mobilization.

E. Fisher, Takao Shimizu, S. Ishii, 2004, Immunity.

High-Density Lipoproteins Retard the Progression of Atherosclerosis and Favorably Remodel Lesions Without Suppressing Indices of Inflammation or Oxidation

John T. Fallon, E. Fisher, J. Fallon, 2004, Arteriosclerosis, thrombosis, and vascular biology.

Adipose Mitochondrial Biogenesis Is Suppressed in db/db and High-Fat Diet–Fed Mice and Improved by Rosiglitazone

Terence E. Ryan, Michael K. Hansen, J. Rong, 2007, Diabetes.

Eliminating Atherogenesis in Mice by Switching Off Hepatic Lipoprotein Secretion

S. Young, E. Fisher, R. Walzem, 2003, Circulation.

HDL promotes rapid atherosclerosis regression in mice and alters inflammatory properties of plaque monocyte-derived cells

K. Moore, K. Rayner, E. Fisher, 2011, Proceedings of the National Academy of Sciences.

High-density lipoprotein: gene-based approaches to the prevention of atherosclerosis

E. Fisher, J. Rong, 2000, Annals of medicine.

Optimization of nutrition: polyphenols and vascular protection.

F. Ursini, A. Sevanian, J. Rong, 2009, Nutrition reviews.

Cholesterol oxidation products induce vascular foam cell lesion formation in hypercholesterolemic New Zealand white rabbits.

A. Sevanian, H. Hodis, J. Rong, 1999, Arteriosclerosis, thrombosis, and vascular biology.

Rosiglitazone Induces Mitochondrial Biogenesis in Differentiated Murine 3T3-L1 and C3H/10T1/2 Adipocytes

K. Waters, T. Ryan, S. Witherspoon, 2011, PPAR research.

Dyslipidemia associated with atherosclerotic disease systemically alters dendritic cell mobilization.

E. Fisher, Takao Shimizu, S. Ishii, 2004, Immunity.