J. Shan

发表

ER-stress-induced transcriptional regulation increases protein synthesis leading to cell death

R. Kaufman, J. Hur, M. Hatzoglou, 2013, Nature Cell Biology.

CHOP induces activating transcription factor 5 (ATF5) to trigger apoptosis in response to perturbations in protein homeostasis

J. McClintick, Donghui Zhou, R. Wek, 2013, Molecular biology of the cell.

A Novel DNMT3B Splice Variant Expressed in Tumor and Pluripotent Cells Modulates Genomic DNA Methylation Patterns and Displays Altered DNA Binding

Keith D Robertson, Naohiro Terada, Philip H Schwartz, 2009, Molecular Cancer Research.

Influence of Amino Acid Metabolism on Embryonic Stem Cell Function and Differentiation.

N. Terada, J. Shan, M. Kilberg, 2016, Advances in nutrition.

The transcription factor network associated with the amino acid response in mammalian cells.

J. Shan, M. Kilberg, M. Balasubramanian, 2012, Advances in nutrition.

Human CHAC1 Protein Degrades Glutathione, and mRNA Induction Is Regulated by the Transcription Factors ATF4 and ATF3 and a Bipartite ATF/CRE Regulatory Element*

I. Mungrue, J. Shan, M. Kilberg, 2015, The Journal of Biological Chemistry.

The C/ebp-Atf response element (CARE) location reveals two distinct Atf4-dependent, elongation-mediated mechanisms for transcriptional induction of aminoacyl-tRNA synthetase genes in response to amino acid limitation

Fan Zhang, J. Shan, M. Kilberg, 2016, Nucleic acids research.

DOE-NSF-NIH Workshop on Opportunities in THz Science, February 12-14, 2004

Qing Hu, Peter Uhd Jepsen, Peter H. Siegel, 2004 .

Induction of early growth response gene 1 (EGR1) by endoplasmic reticulum stress is mediated by the extracellular regulated kinase (ERK) arm of the MAPK pathways.

E. Dudenhausen, J. Shan, M. Kilberg, 2019, Biochimica et biophysica acta. Molecular cell research.

A Mitogen-activated Protein Kinase/Extracellular Signal-regulated Kinase Kinase (MEK)-dependent Transcriptional Program Controls Activation of the Early Growth Response 1 (EGR1) Gene during Amino Acid Limitation*

H. Baker, J. Shan, M. Kilberg, 2014, The Journal of Biological Chemistry.

Nutrient Control of Metabolism and Cell Signaling The Transcription Factor Network Associated With the Amino Acid Response in Mammalian Cells 1 , 2

J. Shan, M. Kilberg, M. Balasubramanian, 2012 .

Elevated ATF4 Expression, in the Absence of Other Signals, Is Sufficient for Transcriptional Induction via CCAAT Enhancer-binding Protein-activating Transcription Factor Response Elements*

J. Shan, M. Kilberg, Daima Örd, 2009, The Journal of Biological Chemistry.

MAPK signaling triggers transcriptional induction of cFOS during amino acid limitation of HepG2 cells.

E. Dudenhausen, Fan Zhang, J. Shan, 2015, Biochimica et biophysica acta.

Auto-activation of c-JUN Gene by Amino Acid Deprivation of Hepatocellular Carcinoma Cells Reveals a Novel c-JUN-mediated Signaling Pathway*

E. Dudenhausen, J. Shan, M. Kilberg, 2011, The Journal of Biological Chemistry.

Myeloma Cells Deplete Bone Marrow Glutamine and Inhibit Osteoblast Differentiation Limiting Asparagine Availability

G. Pruneri, C. Mancini, V. Franceschi, 2020, Cancers.

Activation of the amino acid response modulates lineage specification during differentiation of murine embryonic stem cells.

N. Terada, J. Shan, M. Kilberg, 2013, American journal of physiology. Endocrinology and metabolism.

Sequencing of Argonaute-bound microRNA/mRNA hybrids reveals regulation of the unfolded protein response by microRNA-320a

Tongjun Gu, Mingyi Xie, J. Shan, 2021, PLoS genetics.

Sequencing of Argonaute-bound miRNA/mRNA hybrids reveals regulation of the unfolded protein response by microRNA-320a

Tongjun Gu, Mingyi Xie, J. Shan, 2021, bioRxiv.

Regulation of the ATF3 gene by a single promoter in response to amino acid availability and endoplasmic reticulum stress in human primary hepatocytes and hepatoma cells.

J. Shan, M. Kilberg, Jaclyn N. Hayner, 2018, Biochimica et biophysica acta. Gene regulatory mechanisms.

Regulatory Elements Associated with Paternally-Expressed Genes in the Imprinted Murine Angelman/Prader-Willi Syndrome Domain

J. Khadake, Thomas P. Yang, J. Shan, 2013, PloS one.

ATF4-dependent transcription mediates signaling of amino acid limitation

N. Su, J. Shan, M. Kilberg, 2009, Trends in Endocrinology & Metabolism.

Expression profiling after activation of amino acid deprivation response in HepG2 human hepatoma cells.

H. Baker, J. Shan, M. Kilberg, 2010, Physiological genomics.

ATF4-dependent Regulation of the JMJD3 Gene during Amino Acid Deprivation Can Be Rescued in Atf4-deficient Cells by Inhibition of Deacetylation*

T. Anthony, J. Shan, M. Kilberg, 2012, The Journal of Biological Chemistry.

Parkin is transcriptionally regulated by ATF4: evidence for an interconnection between mitochondrial stress and ER stress

David S. Park, W. Wurst, D. Trümbach, 2011, Cell Death and Differentiation.

Tumor suppressor BTG1 promotes PRMT1-mediated ATF4 function in response to cellular stress

B. Scheijen, G. Poelmans, J. Shan, 2015, Oncotarget.

Dynamic changes in genomic histone association and modification during activation of the ASNS and ATF3 genes by amino acid limitation.

J. Shan, M. Kilberg, M. Balasubramanian, 2013, The Biochemical journal.

Despite Increased ATF4 Binding at the C/EBP-ATF Composite Site following Activation of the Unfolded Protein Response, System A Transporter 2 (SNAT2) Transcription Activity Is Repressed in HepG2 Cells*

J. Shan, M. Kilberg, S. Palii, 2008, Journal of Biological Chemistry.

Expression, refolding, and activation of the catalytic domain of human blood coagulation factor XII.

J. Shan, Li Zheng, R. Krishnamoorthi, 2003, Protein expression and purification.