E. Hoogervorst

发表

Chapter 4 Lack of p 53 . S 389 phosphorylation predisposes mice to develop 2-acetylaminoflurene-induced bladder tumors but not ionizing radiation-induced lymphomas

T. Jacks, E. Zwart, A. D. Vries, 2007 .

Lack of p53 Ser389 phosphorylation predisposes mice to develop 2-acetylaminofluorene-induced bladder tumors but not ionizing radiation-induced lymphomas.

T. Jacks, E. Zwart, H. van Steeg, 2005, Cancer research.

p53 Heterozygosity Results in an Increased 2-Acetylaminofluorene-Induced Urinary Bladder but not Liver Tumor Response in DNA Repair-Deficient Xpa Mice

H. van Steeg, A. de Vries, C. V. van Oostrom, 2004, Cancer Research.

Tissue specific mutagenic and carcinogenic responses in NER defective mouse models.

H. van Steeg, G. V. D. van der Horst, E. Hoogervorst, 2007, Mutation research.

Delayed expression of apoptotic and cell-cycle control genes in carcinogen-exposed bladders of mice lacking p53.S389 phosphorylation.

J. Pennings, T. Breit, M. Jonker, 2007, Carcinogenesis.

The novel MMS‐inducible gene Mif1/KIAA0025 is a target of the unfolded protein response pathway

T. van Laar, C. Terleth, A. van der Eb, 2000, FEBS letters.

Combined oral benzo[a]pyrene and inhalatory ozone exposure have no effect on lung tumor development in DNA repair-deficient Xpa mice.

F. Cassee, J. Vijg, H. van Steeg, 2003, Carcinogenesis.

2-AAF-induced tumor development in nucleotide excision repair-deficient mice is associated with a defect in global genome repair but not with transcription coupled repair.

H. van Steeg, A. de Vries, C. V. van Oostrom, 2005, DNA repair.

Increased Sensitivity to UV Radiation in Mice with a p53 Point Mutation at Ser389

T. Jacks, T. Iwakuma, G. Lozano, 2004, Molecular and Cellular Biology.

Genotoxic exposure: novel cause of selection for a functional ΔN-p53 isoform

J. Pennings, T. Breit, E. Zwart, 2011, Oncogene.

Nucleotide excision repair- and p53-deficient mouse models in cancer research.

H. van Steeg, A. de Vries, E. Hoogervorst, 2005, Mutation research.

Dominant-negative but not gain-of-function effects of a p53.R270H mutation in mouse epithelium tissue after DNA damage.

T. Jacks, J. Jonkers, L. Attardi, 2007, Cancer research.

Growth stimulation of UV-induced DNA damage retaining epidermal basal cells gives rise to clusters of p53 overexpressing cells.

L. Mullenders, F. D. de Gruijl, E. Speksnijder, 2007, DNA repair.