L. Brooks

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p53 mutation with frequent novel codons but not a mutator phenotype in BRCA1- and BRCA2-associated breast tumours

J. Peto, B. Gusterson, Paul D. Smith, 1998, Oncogene.

Epstein-Barr virus and Hodgkin's disease: transcriptional analysis of virus latency in the malignant cells

L. Young, G. Niedobitek, A. Rickinson, 1993, The Journal of experimental medicine.

Novel p53 mutants selected in BRCA-associated tumours which dissociate transformation suppression from other wild-type p53 functions

B. Gusterson, Paul D. Smith, T. Crook, 1999, Oncogene.

Epstein-Barr virus infection in oral hairy leukoplakia: virus replication in the absence of a detectable latent phase.

L. Young, G. Niedobitek, L. Brooks, 1991, The Journal of general virology.

Coincident inactivation of 14-3-3σ and p16INK4a is an early event in vulval squamous neoplasia

B. Gusterson, T. Crook, A. Sullivan, 2002, Oncogene.

Distribution and phenotype of Epstein-Barr virus-harboring cells in Hodgkin's disease.

Young, H. Stein, H. Herbst, 1992, Blood.

Expression of Epstein-Barr virus genes and of lymphocyte activation molecules in undifferentiated nasopharyngeal carcinomas.

U. Prasad, C. Sam, L. Young, 1992, The American journal of pathology.

Epstein-Barr virus gene expression in malignant lymphomas induced by experimental virus infection of cottontop tamarins

Young, L. Young, A. Rickinson, 1989, Journal of virology.

Analysis of epstein‐barr virus infection in nasopharyngeal biopsies from a group at high risk of nasopharyngeal carcinoma

U. Prasad, C. Sam, L. Young, 2009, International journal of cancer.

Persistent epstein-barr virus infection: unrestricted latent and lytic viral gene expression in healthy immunosuppressed transplant recipients1

L. Brooks, D. Crawford, P. Hopwood, 2002, Transplantation.

Spectrum of p53 gene mutations suggests a possible role for ultraviolet radiation in the pathogenesis of advanced cutaneous lymphomas.

J. McGregor, S. Whittaker, L. Brooks, 1999, The Journal of investigative dermatology.

Patterns of Epstein-Barr virus infection in non-neoplastic lymphoid tissue.

H. Stein, H. Herbst, M. Masucci, 1992, Blood.

Restricted Epstein-Barr virus protein expression in Burkitt lymphoma is due to a different Epstein-Barr nuclear antigen 1 transcriptional initiation site.

E. Kieff, C. Sample, J. Sample, 1991, Proceedings of the National Academy of Sciences of the United States of America.

Transcripts from the Epstein-Barr virus BamHI A fragment are detectable in all three forms of virus latency

L. Young, A. Rickinson, L. Brooks, 1993, Journal of virology.

Epstein‐Barr virus (EBV) latent membrane protein 1 increases HLA class II expression in an EBV‐negative B cell line

T. Tursz, A. Rickinson, L. Brooks, 1994, European journal of immunology.

Kaposi's sarcoma‐associated herpesvirus (KSHV)/human herpesvirus 8 (HHV8)—a new human tumour virus

Andrew J. Wilson, A. Wilson, L. Brooks, 1997, The Journal of pathology.

Epstein-Barr virus latent gene transcription in nasopharyngeal carcinoma cells: coexpression of EBNA1, LMP1, and LMP2 transcripts

Q Y Yao, L. Young, A. Rickinson, 1992, Journal of virology.

A fragment are detectable in all three forms of Transcripts from the Epstein-Barr virus BamHI

A. Rickinson, L. Brooks, A. Lear, 2014 .

Transcripts from the Epstein-Barr virus BamHI A fragment are detectable in all three forms of virus latency

L. Young, A. Rickinson, L. Brooks, 1993, Journal of virology.

The Expression of Variant CD 44 in Nasopharyngeal Carcinoma Is Unrelated to Expression of LMP1

P. Farrell, G. Niedobitek, L. Brooks, 2007 .

Epstein-Barr Virus Replication and Gene Expression in Oral “Hairy” Leukoplakia

L. Resnick, L. Young, N. Raab-Traub, 1989 .

Epstein-Barr Virus Gene Expression in Lymphomas Induced by the Virus in the Cottontop Tamarin

L. Young, A. Rickinson, L. Brooks, 1989 .

A common polymorphism acts as an intragenic modifier of mutant p53 behaviour

B. Gusterson, W. Kaelin, T. Crook, 2000, Nature Genetics.

TCL1 is activated by chromosomal rearrangement or by hypomethylation

D. Catovsky, L. Brooks, A. Condie, 2001, Genes, chromosomes & cancer.

High level expression of ΔN-p63: a mechanism for the inactivation of p53 in undifferentiated nasopharyngeal carcinoma (NPC)?

T. Crook, J. Nicholls, L. Brooks, 2000, Oncogene.

Epstein-Barr virus and lymphomas

L. Brooks, D. Crawford, T. Crook, 1999 .

A strategy for specific targeting of therapeutic agents to tumour cells of virus-associated cancers

T. Evans, L. Brooks, P. Farrell, 1997, Gene Therapy.

Preferential retention of codon 72 arginine p53 in squamous cell carcinomas of the vulva occurs in cancers positive and negative for human papillomavirus.

B. Gusterson, W. Kaelin, T. Crook, 2000, Cancer research.

Relationship Between p 53 Codon 72 Polymorphism and Susceptibility to Sunburn and Skin Cancer

S. Fisher, C. Lewis, J. McGregor, 2010 .

Relationship between p53 codon 72 polymorphism and susceptibility to sunburn and skin cancer.

S. Fisher, C. Lewis, J. McGregor, 2002, The Journal of investigative dermatology.

p73 is over-expressed in vulval cancer principally as the Δ2 isoform

B. Gusterson, T. Crook, A. Sullivan, 2001, British Journal of Cancer.

p53 mutations implicate sunlight in post-transplant skin cancer irrespective of human papillomarivus status

T. Crook, J. McGregor, L. Brooks, 1997, Oncogene.

E7 proteins from oncogenic human papillomavirus types transactivate p73: role in cervical intraepithelial neoplasia

B. Gusterson, T. Sakai, T. Crook, 2002, British Journal of Cancer.

The expression of variant CD44 in nasopharyngeal carcinoma is unrelated to expression of LMP-1.

G. Niedobitek, L. Brooks, P. Farrell, 1995, The American journal of pathology.

Evidence against a major genetic basis for combined breast and colorectal cancer susceptibility

I. Tomlinson, S. Hodgson, A. Rowan, 2006, Clinical genetics.