A. Sassi

发表

Infection of the equine population by Leishmania parasites.

M. Mhadhbi, A. Sassi, 2019, Equine veterinary journal.

The extended clinical phenotype of 64 patients with dedicator of cytokinesis 8 deficiency.

A. Schäffer, V. Lougaris, R. Geha, 2015, The Journal of allergy and clinical immunology.

The pathological effects of Heminecrolysin, a dermonecrotic toxin from Hemiscorpius lepturus scorpion venom are mediated through its lysophospholipase D activity.

D. Shahbazzadeh, I. Safra, A. Sassi, 2013, Toxicon : official journal of the International Society on Toxinology.

Leishmania major Amastigotes Induce p50/c-Rel NF-κΒ Transcription Factor in Human Macrophages: Involvement in Cytokine Synthesis

L. Guizani-Tabbane, K. Dellagi, A. Sassi, 2004, Infection and Immunity.

Heminecrolysin, the first hemolytic dermonecrotic toxin purified from scorpion venom.

A. van Dorsselaer, J. Strub, D. Shahbazzadeh, 2011, Toxicon : official journal of the International Society on Toxinology.

Leishmanin skin test lymphoproliferative responses and cytokine production after symptomatic or asymptomatic Leishmania major infection in Tunisia

K. Dellagi, M. Mokni, A. Ben Salah, 1999, Clinical and experimental immunology.

Development of antibodies to the iron-binding proteins transferrin and ferritin in dogs and mice infected with Leishmania parasites.

A. Sassi, 2022, Acta tropica.

Comparative analysis of the Leishmania infantum-specific antibody repertoires and the autoantibody repertoires between asymptomatic and symptomatic dogs.

M. Mhadhbi, A. Sassi, M. Gharbi, 2018, Veterinary parasitology.

Large deletions and point mutations involving the dedicator of cytokinesis 8 (DOCK8) in the autosomal-recessive form of hyper-IgE syndrome.

Edward R B McCabe, Bodo Grimbacher, Christoph Klein, 2009, The Journal of allergy and clinical immunology.

Large deletions and point mutations involving the dedicator of cytokinesis 8 (DOCK8) in the autosomal-recessive form of hyper-IgE syndrome.

T. Chatila, S. Holland, C. Klein, 2009, The Journal of allergy and clinical immunology.

Hypomorphic homozygous mutations in phosphoglucomutase 3 (PGM3) impair immunity and increase serum IgE levels.

A. Schäffer, U. Kölsch, T. Jakob, 2014, The Journal of allergy and clinical immunology.

Mechanisms of the Natural Reactivity of Lymphocytes from Noninfected Individuals to Membrane-Associated Leishmania infantum Antigens1

K. Dellagi, P. Cazenave, A. Sassi, 2005, The Journal of Immunology.

[Primary immunodeficiency in Tunisia: study of 152 cases].

M. Barbouche, A. Sassi, M. Bejaoui, 1997, Archives de pediatrie : organe officiel de la Societe francaise de pediatrie.

Leishmania major induces deactivation of extracellular signal regulated kinases 2 in human U937 macrophage like cells.

L. Guizani-Tabbane, K. Dellagi, A. Sassi, 2000, Archives de l'Institut Pasteur de Tunis.

Identification of immunodominant Leishmania major antigenic markers of the early C57BL/6 and BALB/c mice infection stages

A. Sassi, A. Ben Ammar Elgaied, O. Kaak, 2015, Parasite immunology.

Age related efficiency of the leishmanin skin test as a marker of immunity to human visceral leishmaniasis.

A. Ben Salah, A. Sassi, N. Hamida, 2012, Archives de l'Institut Pasteur de Tunis.

Toxoplasma gondii inhibits R5 HIV-1 replication in human lymphoid tissues ex vivo.

A. Sher, J. Grivel, L. Margolis, 2009, Microbes and infection.

Heminecrolysin, a potential immunogen for monospecific antivenom production against Hemiscorpius lepturus scorpion.

I. Safra, A. Sassi, M. El Ayeb, 2011, Toxicon : official journal of the International Society on Toxinology.

Natural autoantibodies, IgG antibodies to tetanus toxoid and CD5+ B cells in patients with Mediterranean visceral leishmaniasis

K. Dellagi, A. Sassi, H. Louzir, 1994, Clinical and experimental immunology.

Immunoregulatory role for a public IgM idiotype in the induction of autoimmune diseases in Mycoplasma pneumoniae infection.

A. Sassi, A. Benammar-Elgaaied, F. Ben Aissa-Fennira, 2011, Immunology letters.

Large deletions and point mutations involving the dedicator of cytokinesis 8 (DOCK8) in the autosomal-recessive form of hyper-IgE syndrome (vol 124, pg 1289, 2009)

T. Chatila, J. Puck, S. Ehl, 2010 .