W. Dolci

发表

Increasing GLP-1–Induced β-Cell Proliferation by Silencing the Negative Regulators of Signaling cAMP Response Element Modulator-α and DUSP14

P. Halban, B. Thorens, C. Poussin, 2008, Diabetes.

Dominant negative MyD88 proteins inhibit interleukin-1beta /interferon-gamma -mediated induction of nuclear factor kappa B-dependent nitrite production and apoptosis in beta cells.

B. Thorens, W. Dolci, P. Dupraz, 2000, The Journal of biological chemistry.

Dominant Negative MyD88 Proteins Inhibit Interleukin-1β/Interferon-γ-mediated Induction of Nuclear Factor κB-dependent Nitrite Production and Apoptosis in β Cells*

B. Thorens, W. Dolci, P. Dupraz, 2000, The Journal of Biological Chemistry.

Sodium/hydrogen exchanger NHA2 is critical for insulin secretion in β-cells

H. Moch, B. Thorens, W. Dolci, 2013, Proceedings of the National Academy of Sciences.

Regulated exocytosis of an H+/myo‐inositol symporter at synapses and growth cones

P. Steiner, M. Uldry, B. Thorens, 2004, The EMBO journal.

Differential Sensitivity of GLUT1- and GLUT2-Expressing β Cells to Streptozotocin

B. Thorens, W. Dolci, M. Hosokawa, 2001 .

Different behaviour of mouse‐human chimeric antibody F(ab')2 fragments of IgG1, IgG2 and IgG4 sub‐class in vivo

C. Heusser, F. Buchegger, W. Dolci, 1992, International journal of cancer.

Encapsulated, Genetically Engineered Cells, Secreting Glucagon‐like Peptide‐1 for the Treatment of Non‐insulin‐dependent Diabetes Mellitus

S. Germain, R. Burcelin, B. Thorens, 1999, Annals of the New York Academy of Sciences.

Heterologous Desensitization of the Glucagon-like Peptide-1 Receptor by Phorbol Esters Requires Phosphorylation of the Cytoplasmic Tail at Four Different Sites*

C. Widmann, B. Thorens, W. Dolci, 1996, The Journal of Biological Chemistry.

Exendin-(9-39) is an inverse agonist of the murine glucagon-like peptide-1 receptor: implications for basal intracellular cyclic adenosine 3',5'-monophosphate levels and beta-cell glucose competence.

D. Drucker, B. Thorens, S. Efrat, 1998, Endocrinology.

Exendin-(9-39) Is an Inverse Agonist of the Murine Glucagon-Like Peptide-1 Receptor: Implications for Basal Intracellular Cyclic Adenosine 3',5'-Monophosphate Levels and β-Cell Glucose Competence* *This work was supported by Grant 31-46958.96 from the Swiss National Science Foundation (to B.T.).

D. Drucker, B. Thorens, S. Efrat, 1998, Endocrinology.

Desensitization and phosphorylation of the glucagon-like peptide-1 (GLP-1) receptor by GLP-1 and 4-phorbol 12-myristate 13-acetate.

C. Widmann, B. Thorens, W. Dolci, 1996, Molecular endocrinology.

Internalization and homologous desensitization of the GLP-1 receptor depend on phosphorylation of the receptor carboxyl tail at the same three sites.

C. Widmann, B. Thorens, W. Dolci, 1997, Molecular endocrinology.

Agonist-induced internalization and recycling of the glucagon-like peptide-1 receptor in transfected fibroblasts and in insulinomas.

B. Thorens, W. Dolci, C. Widmann, 1995, The Biochemical journal.

Portal glucose infusion in the mouse induces hypoglycemia: evidence that the hepatoportal glucose sensor stimulates glucose utilization.

R. Burcelin, B. Thorens, W. Dolci, 2000, Diabetes.

Signal transduction by the cloned glucagon-like peptide-1 receptor: comparison with signaling by the endogenous receptors of beta cell lines.

B. Thorens, W. Dolci, E. Bürki, 1994, Molecular pharmacology.

Effect of Different Calcium Channel Blockers on Angiotensin II‐ and Vasopressin‐Induced Prostacyclin Biosynthesis in Vascular Smooth Muscle Cells

W. Dolci, M. Vallotton, R. Rivest, 1990, Journal of cardiovascular pharmacology.

Interaction of vasopressin and angiotensin II in stimulation of prostacyclin synthesis in vascular smooth muscle cells.

R. Wüthrich, R. P. Wuthrich, W. Dolci, 1989, The American journal of physiology.

cFLIP protein prevents tumor necrosis factor-alpha-mediated induction of caspase-8-dependent apoptosis in insulin-secreting betaTc-Tet cells.

B. Thorens, W. Dolci, P. Dupraz, 2002, Diabetes.

cFLIP protein prevents tumor necrosis factor-alpha-mediated induction of caspase-8-dependent apoptosis in insulin-secreting betaTc-Tet cells.

B. Thorens, W. Dolci, P. Dupraz, 2002, Diabetes.

SOCS-1 Protein Prevents Janus Kinase/STAT-dependent Inhibition of β Cell Insulin Gene Transcription and Secretion in Response to Interferon-γ*

B. Thorens, W. Dolci, P. Dupraz, 2001, The Journal of Biological Chemistry.

Fgf15 Neurons of the Dorsomedial Hypothalamus Control Glucagon Secretion and Hepatic Gluconeogenesis

G. Labouèbe, B. Thorens, S. Croizier, 2021, Diabetes.

Long-lasting antidiabetic effect of a dipeptidyl peptidase IV-resistant analog of glucagon-like peptide-1.

R. Burcelin, B. Thorens, W. Dolci, 1999, Metabolism: clinical and experimental.

α-cell glucokinase suppresses glucose-regulated glucagon secretion

P. Rorsman, B. Thorens, P. Herrera, 2018, Nature Communications.

Differential sensitivity of GLUT1- and GLUT2-expressing beta cells to streptozotocin.

B. Thorens, W. Dolci, M. Hosokawa, 2001, Biochemical and biophysical research communications.

Glucose sensing by the hepatoportal sensor is GLUT2-dependent: in vivo analysis in GLUT2-null mice.

R. Burcelin, B. Thorens, W. Dolci, 2000, Diabetes.

Action of calcitonin gene-related peptide on rat aortic smooth muscle.

W. Dolci, M. Vallotton, J. Fischer, 1989, European journal of pharmacology.

Anatomical localization of corticotropin-releasing activity in the human brain.

A. F. Muller, W. Dolci, R. Gaillard, 1986, Life sciences.

α-cell glucokinase suppresses glucose-regulated glucagon secretion

P. Rorsman, B. Thorens, P. Herrera, 2018, Nature Communications.

Plasma vasopressin levels after infusions of hypertonic saline solutions into the renal, portal, carotid, or systemic circulation in conscious dogs.

W. Dolci, M. Vallotton, J. Liard, 1984, Endocrinology.

Agonist-induced internalization and recycling of the glucagon-like peptide-1 receptor in transfected fibroblasts and in insulinomas.

C. Widmann, B. Thorens, W. Dolci, 1995, The Biochemical journal.

D. Drucker, B. Thorens, S. Efrat, 1998, Endocrinology.

Exendin-(9-39) is an inverse agonist of the murine glucagon-like peptide-1 receptor: implications for basal intracellular cyclic adenosine 3',5'-monophosphate levels and beta-cell glucose competence.

W. Dolci, B. Thorens, V. Serre, 1998, Endocrinology.