B. LaMarca

发表

Pathophysiology and Current Clinical Management of Preeclampsia

M. Owens, L. Amaral, K. Wallace, 2017, Current Hypertension Reports.

Risk of cardiovascular disease, end-stage renal disease, and stroke in postpartum women and their fetuses after a hypertensive pregnancy.

B. LaMarca, Mark W. Cunningham, 2018, American journal of physiology. Regulatory, integrative and comparative physiology.

Tumor Necrosis Factor-&agr; Antagonist Etanercept Decreases Blood Pressure and Protects the Kidney in a Mouse Model of Systemic Lupus Erythematosus

H. Drummond, B. LaMarca, L. Racusen, 2010, Hypertension.

Vitamin D supplementation reduces some AT1-AA-induced downstream targets implicated in preeclampsia including hypertension.

R. Dechend, L. Amaral, K. Wallace, 2017, American journal of physiology. Regulatory, integrative and comparative physiology.

Effects of hyperhomocysteinemia on arterial pressure and nitric oxide production in pregnant rats.

J. Granger, B. LaMarca, J. Reckelhoff, 2009, American journal of hypertension.

Characterization of Mitochondrial Bioenergetics in Preeclampsia

L. Amaral, K. Wallace, B. LaMarca, 2021, Journal of clinical medicine.

Renal natural killer cell activation and mitochondrial oxidative stress; new mechanisms in AT1-AA mediated hypertensive pregnancy.

G. Wallukat, R. Dechend, L. Amaral, 2018, Pregnancy hypertension.

A model of preeclampsia in rats: the reduced uterine perfusion pressure (RUPP) model.

B. LaMarca, Jing Li, J. Reckelhoff, 2012, American journal of physiology. Heart and circulatory physiology.

Effects of reduced uterine perfusion pressure on blood pressure and metabolic factors in the pregnant rat

J. Granger, B. LaMarca, J. Gilbert, 2007, American journal of hypertension.

Pathophysiology of hypertension during preeclampsia: linking placental ischemia with endothelial dysfunction.

J. Granger, B. LaMarca, J. Gilbert, 2008, American journal of physiology. Heart and circulatory physiology.

Blockade of CD40 ligand for intercellular communication reduces hypertension, placental oxidative stress, and AT1-AA in response to adoptive transfer of CD4+ T lymphocytes from RUPP rats.

G. Wallukat, R. Dechend, L. Amaral, 2015, American journal of physiology. Regulatory, integrative and comparative physiology.

Hypertension Produced by Reductions in Uterine Perfusion in the Pregnant Rat: Role of Tumor Necrosis Factor-&agr;

J. Granger, B. LaMarca, B. Alexander, 2005, Hypertension.

Changes in cardiac structure in hypertension produced by placental ischemia in pregnant rats: effect of tumor necrosis factor blockade

J. Granger, B. LaMarca, J. Gutkowska, 2011, Journal of hypertension.

Cytochrome P450 Subfamily 2J Polypeptide 2 Expression and Circulating Epoxyeicosatrienoic Metabolites in Preeclampsia

G. Wallukat, B. Huppertz, E. Ekholm, 2012, Circulation.

Hypertension Produced by Reductions in Uterine Perfusion in the Pregnant Rat: Role of Interleukin 6

J. Granger, B. LaMarca, G. Gadonski, 2006, Hypertension.

Natural killer cells contribute to mitochondrial dysfunction in response to placental ischemia in reduced uterine perfusion pressure rats.

L. Amaral, B. LaMarca, D. Cornelius, 2019, American journal of physiology. Regulatory, integrative and comparative physiology.

Angiotensin II Type 1 Receptor Autoantibody (AT1‐AA)‐Mediated Pregnancy Hypertension

B. LaMarca, F. Herse, 2013, American journal of reproductive immunology.

Hypertension in Response to Chronic Reductions in Uterine Perfusion in Pregnant Rats: Effect of Tumor Necrosis Factor-&agr; Blockade

J. Granger, B. LaMarca, Sara A. Babcock, 2008, Hypertension.

Reduced uterine perfusion pressure (RUPP) model for studying cardiovascular-renal dysfunction in response to placental ischemia.

J. Granger, B. LaMarca, M. Sedeek, 2006, Methods in molecular medicine.

Vitamin D supplementation improves pathophysiology in a rat model of preeclampsia.

G. Wallukat, R. Dechend, L. Amaral, 2016, American journal of physiology. Regulatory, integrative and comparative physiology.

Vitamin D supplementation improves pathophysiology in a rat

G. Wallukat, R. Dechend, L. Amaral, 2015 .

Vitamin D supplementation improves pathophysiology in a rat 1 model of preeclampsia

G. Wallukat, R. Dechend, L. Amaral, 2015 .

Vitamin D Supplementation Suppresses Hypoxia-Stimulated Placental Cytokine Secretion, Hypertension and CD4+ T Cell Stimulation in Response to Placental Ischemia.

J. Martin, K. Wallace, B. LaMarca, 2013, Medical journal of obstetrics and gynecology.

CYP2J2 Expression and Circulating Epoxyeicosatrienoic Metabolites in Preeclampsia

G. Wallukat, B. Huppertz, E. Ekholm, 2012 .

Placental Ischemia Impairs Middle Cerebral Artery Myogenic Responses in the Pregnant Rat

H. Drummond, J. Granger, B. LaMarca, 2011, Hypertension.

Rosiglitazone reduces blood pressure in female Dahl salt-sensitive rats

B. LaMarca, L. Racusen, D. Romero, 2010, Steroids.

Angiotensin II type 1 receptor autoantibody blockade improves cerebral blood flow autoregulation and hypertension in a preclinical model of preeclampsia

G. Booz, B. LaMarca, J. Williams, 2020, Hypertension in pregnancy.

The Role of Agonistic Autoantibodies to the Angiotensin II Type 1 Receptor (AT1-AA) in Pathophysiology of Preeclampsia.

B. LaMarca, Mark W. Cunningham, Nathan Campbell, 2018, Current pharmaceutical biotechnology.

A complicated role for the renin–angiotensin system during pregnancy: highlighting the importance of drug discovery

J. Martin, B. LaMarca, Marie M Darby, 2013, Expert opinion on drug safety.

Arachidonic acid metabolites of CYP4A and CYP4F are altered in women with preeclampsia.

K. Wallace, B. LaMarca, S. Spencer, 2018, Prostaglandins & other lipid mediators.

AT1-AA (Angiotensin II Type 1 Receptor Agonistic Autoantibody) Blockade Prevents Preeclamptic Symptoms in Placental Ischemic Rats

L. Amaral, B. LaMarca, J. Williams, 2018, Hypertension.

Hypoxia-Induced Pathways Associated With Hypertension During Pregnancy

K. Wallace, B. LaMarca, D. Cornelius, 2013 .

Endothelial dysfunction. An important mediator in the pathophysiology of hypertension during pre-eclampsia.

B. LaMarca, 2012, Minerva ginecologica.

Hypertension in Response to Autoantibodies to the Angiotensin II Type I Receptor (AT1-AA) in Pregnant Rats: Role of Endothelin-1

G. Wallukat, R. Dechend, J. Martin, 2009, Hypertension.

CALL FOR PAPERS Integrative and Translational Physiology: Inﬂammation and Immunity in Organ System Physiology IL-17-mediated oxidative stress is an important stimulator of AT1-AA and hypertension during pregnancy

G. Wallukat, R. Dechend, J. Martin, 2012 .

The effect of immune factors, tumor necrosis factor-alpha, and agonistic autoantibodies to the angiotensin II type I receptor on soluble fms-like tyrosine-1 and soluble endoglin production in response to hypertension during pregnancy.

G. Wallukat, J. Granger, R. Dechend, 2010, American journal of hypertension.

Interleukin-4 supplementation improves the pathophysiology of hypertension in response to placental ischemia in RUPP rats.

G. Wallukat, R. Dechend, L. Amaral, 2019, American journal of physiology. Regulatory, integrative and comparative physiology.

Reduced uterine perfusion pressure T-helper 17 cells cause pathophysiology associated with preeclampsia during pregnancy.

G. Wallukat, R. Dechend, L. Amaral, 2016, American journal of physiology. Regulatory, integrative and comparative physiology.

Preventing Autoimmunity Protects Against the Development of Hypertension and Renal Injury

K. Wallace, B. LaMarca, Christine Maric-Bilkan, 2014, Hypertension.

Progesterone supplementation attenuates hypertension and the autoantibody to the angiotensin II type I receptor in response to elevated interleukin-6 during pregnancy.

G. Wallukat, R. Dechend, J. Martin, 2014, American journal of obstetrics and gynecology.

Control of soluble fms-like tyrosine-1 (sFlt-1) production response to placental ischemia/hypoxia: role of tumor necrosis factor-α.

J. Granger, B. LaMarca, M. Parrish, 2013, American journal of physiology. Regulatory, integrative and comparative physiology.

The Effect of Immune Factors, Tumor Necrosis Factor- α (TNF- α ), and Agonistic Autoantibodies to the Angiotensin II Type I Receptor (AT1-AA) on Soluble fms-Like Tyrosine-1 (sFlt-1) and Soluble Endoglin (sEng) Production in Response to Hypertension During Pregnancy

Katrin, G. Wallukat, J. Granger, 2012 .

The role of immune activation in contributing to vascular dysfunction and the pathophysiology of hypertension during preeclampsia.

B. LaMarca, Babbette LaMarca, 2010, Minerva ginecologica.

Babbette LaMarca Autoantibody to the Angiotensin II Type I Receptor Progress Toward Identifying Potential Markers for Preeclampsia : Role of Agonistic

B. LaMarca, 2010 .

ACE2 and ANG-(1-7) in the gravid uterus: the new players on the block.

J. Granger, B. LaMarca, J. Gilbert, 2008, American Journal of Physiology. Regulatory Integrative and Comparative Physiology.

CD4+ T Cells Are Important Mediators of Oxidative Stress That Cause Hypertension in Response to Placental Ischemia

K. Wallace, B. LaMarca, D. Cornelius, 2014, Hypertension.

Hypertension in response to AT1-AA: role of reactive oxygen species in pregnancy-induced hypertension.

G. Wallukat, R. Dechend, J. Martin, 2011, American journal of hypertension.

Angiotensin II type 1 autoantibody induced hypertension during pregnancy is associated with renal endothelial dysfunction.

R. Dechend, J. Martin, B. LaMarca, 2011, Gender medicine.

Posterior reversible encephalopathy syndrome in 46 of 47 patients with eclampsia.

M. Owens, J. Martin, M. Khan, 2013, American journal of obstetrics and gynecology.

An increased population of regulatory T cells improves the pathophysiology of placental ischemia in a rat model of preeclampsia.

G. Wallukat, R. Dechend, L. Amaral, 2015, American journal of physiology. Regulatory, integrative and comparative physiology.

Renal medullary endothelin-1 is decreased in Dahl salt-sensitive rats.

J. Granger, B. LaMarca, E. George, 2011, American journal of physiology. Regulatory, integrative and comparative physiology.

Randomized, placebo controlled, double blind trial evaluating early pregnancy phytonutrient supplementation in the prevention of preeclampsia

J. N. Martin, M. Owens, B. LaMarca, 2013, Journal of Perinatology.

Continued Investigation Into 17-OHPC: Results From the Preclinical RUPP Rat Model of Preeclampsia

L. Amaral, B. LaMarca, D. Cornelius, 2017, Hypertension.

Role of Endothelin in Mediating Soluble fms-Like Tyrosine Kinase 1–Induced Hypertension in Pregnant Rats

J. Granger, B. LaMarca, Sydney R. Murphy, 2010, HYPERTENSION.

The role of tumor necrosis factor in triggering activation of natural killer cell, multi-organ mitochondrial dysfunction and hypertension during pregnancy.

L. Amaral, B. LaMarca, D. Cornelius, 2021, Pregnancy hypertension.

Progesterone blunts vascular endothelial cell secretion of endothelin-1 in response to placental ischemia.

J. Martin, K. Wallace, B. LaMarca, 2013, American journal of obstetrics and gynecology.

Novel treatment avenues for uterine leiomyoma: a new implication for endothelin?

K. Wallace, B. LaMarca, J. Rushing, 2018, Clinical science.

TH17- and IL-17- mediated autoantibodies and placental oxidative stress play a role in the pathophysiology of pre-eclampsia.

B. LaMarca, D. Cornelius, 2014, Minerva ginecologica.

Placental CD4+ T cells isolated from preeclamptic women cause preeclampsia-like symptoms in pregnant nude-athymic rats.

L. Amaral, K. Wallace, B. LaMarca, 2019, Pregnancy hypertension.

CD4+ T-Helper Cells Stimulated in Response to Placental Ischemia Mediate Hypertension During Pregnancy

J. Martin, K. Wallace, B. LaMarca, 2011, Hypertension.

Agonistic autoantibodies to the angiotensin II type I receptor cause pathophysiologic characteristics of preeclampsia.

K. Wallace, B. LaMarca, M. Parrish, 2012, Gender medicine.

Hellp syndrome and composite major maternal morbidity: importance of Mississippi classification system

M. Owens, J. Martin, K. Wallace, 2013, The journal of maternal-fetal & neonatal medicine : the official journal of the European Association of Perinatal Medicine, the Federation of Asia and Oceania Perinatal Societies, the International Society of Perinatal Obstetricians.

Participation of fad and mbt Genes in Synthesis of Mycobactin in Mycobacterium smegmatis

M. Lundrigan, Wenming Zhu, B. LaMarca, 2004, Journal of bacteriology.

Hypertension, inflammation and T lymphocytes are increased in a rat model of HELLP syndrome

P. Kyle, K. Wallace, B. LaMarca, 2014, Hypertension in pregnancy.

765: Agonistic autoantibodies to the angiotensin II type I receptor (AT1-AA) mediate endothelial dysfunction associated with hypertension in response to placental ischemia during pregnancy

G. Wallukat, R. Dechend, J. Martin, 2011 .

Preeclampsia: long-term consequences for vascular health

L. Amaral, B. LaMarca, D. Cornelius, 2015, Vascular health and risk management.

Inflammatory mediators: a causal link to hypertension during preeclampsia

L. Amaral, B. LaMarca, D. Cornelius, 2018, British journal of pharmacology.

Identifying immune mechanisms mediating the hypertension during preeclampsia.

L. Amaral, K. Wallace, B. LaMarca, 2016, American journal of physiology. Regulatory, integrative and comparative physiology.

Hypertension in Response to Placental Ischemia During Pregnancy: Role of B Lymphocytes

G. Wallukat, R. Dechend, J. Martin, 2011, Hypertension.

IL-6-induced pathophysiology during pre-eclampsia: potential therapeutic role for magnesium sulfate?

K. Wallace, B. LaMarca, J. Brewer, 2011, International journal of interferon, cytokine and mediator research.

Activating autoantibodies to the angiotensin II type I receptor play an important role in mediating hypertension in response to adoptive transfer of CD4+ T lymphocytes from placental ischemic rats.

G. Wallukat, R. Dechend, J. Martin, 2012, American journal of physiology. Regulatory, integrative and comparative physiology.

Placental Ischemia and Resultant Phenotype in Animal Models of Preeclampsia

L. Amaral, B. LaMarca, D. Cornelius, 2016, Current Hypertension Reports.

Hypertension in response to CD4(+) T cells from reduced uterine perfusion pregnant rats is associated with activation of the endothelin-1 system.

M. Owens, J. Martin, K. Wallace, 2012, American journal of physiology. Regulatory, integrative and comparative physiology.

Role of angiotensin II type I receptor agonistic autoantibodies (AT1-AA) in preeclampsia.

J. Granger, K. Wallace, B. LaMarca, 2011, Current opinion in pharmacology.

Elucidating immune mechanisms causing hypertension during pregnancy.

K. Wallace, B. LaMarca, D. Cornelius, 2013, Physiology.

A Model of Pre-eclampsia in Rats : The Reduced Uterine Perfusion Pressure 2 ( RUPP ) Model 3 4 5

B. LaMarca, Jing Li, J. Reckelhoff, 2012 .

Proliferation of endogenous regulatory T cells improve the pathophysiology associated with placental ischaemia of pregnancy

G. Wallukat, R. Dechend, L. Amaral, 2017, American journal of reproductive immunology.

Magnesium Sulfate Treatment Reverses Seizure Susceptibility and Decreases Neuroinflammation in a Rat Model of Severe Preeclampsia

M. Cipolla, K. Nagle, B. LaMarca, 2014, PloS one.

Role of reactive oxygen species in hypertension produced by reduced uterine perfusion in pregnant rats.

J. Granger, B. LaMarca, J. Gilbert, 2008, American journal of hypertension.

[35-OR]: A role for TH17 cells and IL-17 in mediating the pathophysiology associated with preeclampsia

K. Wallace, B. LaMarca, D. Cornelius, 2015 .

Identifying immune mechanisms mediating the hypertension during preeclampsia.

L. Amaral, K. Wallace, B. LaMarca, 2016, American journal of physiology. Regulatory, integrative and comparative physiology.

Blockade of CD40 ligand for intercellular communication reduces hypertension, placental oxidative stress, and AT1-AA in response to adoptive transfer of CD4+ T lymphocytes from RUPP rats.

G. Wallukat, R. Dechend, L. Amaral, 2015, American journal of physiology. Regulatory, integrative and comparative physiology.

Blockade of Cd40 Ligand for Intercellular Communication Reduces Hypertension, Placental Oxidative Stress, and at 1 -aa in Response to Adoptive Transfer of Cd4 Ϩ T Lymphocytes from Rupp Rats

G. Wallukat, R. Dechend, L. Amaral, 2022 .

Hypertension in response to CD4(+) T cells from reduced uterine perfusion pregnant rats is associated with activation of the endothelin-1 system.

M. Owens, J. Martin, K. Wallace, 2012, American journal of physiology. Regulatory, integrative and comparative physiology.

A model of preeclampsia in rats: the reduced uterine perfusion pressure (RUPP) model.

B. LaMarca, Jing Li, J. Reckelhoff, 2012, American journal of physiology. Heart and circulatory physiology.

Role of Endothelin in Mediating Tumor Necrosis Factor-Induced Hypertension in Pregnant Rats

J. Granger, B. LaMarca, K. Cockrell, 2005, Hypertension.

ANTIHYPERTENSIVE AGENTS : MECHANISMS OF DRUG ACTION ( ME ERNST , SECTION EDITOR ) Pathophysiology and Current Clinical Management of Preeclampsia

M. Owens, L. Amaral, K. Wallace, 2017 .

An increased population of regulatory T cells improves the pathophysiology of placental ischemia in a rat model of preeclampsia.

G. Wallukat, R. Dechend, L. Amaral, 2015, American journal of physiology. Regulatory, integrative and comparative physiology.

Vascular Health and Risk Management Dovepress Preeclampsia: Long-term Consequences for Vascular Health

L. Amaral, B. LaMarca, D. Cornelius, 2022 .

Renal natural killer cell activation and mitochondrial oxidative stress; new mechanisms in AT1-AA mediated hypertensive pregnancy.

G. Wallukat, R. Dechend, L. Amaral, 2019, Pregnancy hypertension.

Hypertension in Response to Autoantibodies to the Angiotensin II Type I Receptor (AT1-AA) in Pregnant Rats: Role of Endothelin-1

G. Wallukat, R. Dechend, J. Martin, 2009, Hypertension.

Inhibition of the AT1R agonistic autoantibody in a rat model of preeclampsia improves fetal growth in late gestation.

G. Bidwell, B. LaMarca, D. Romero, 2022, American journal of physiology. Regulatory, integrative and comparative physiology.

Angiotensin II type 1 autoantibody induced hypertension during pregnancy is associated with renal endothelial dysfunction.

R. Dechend, J. Martin, B. LaMarca, 2011, Gender medicine.