J. Weiss

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The bactericidal/permeability-increasing protein (BPI), a potent element in host-defense against gram-negative bacteria and lipopolysaccharide.

P. Elsbach, J. Weiss, Jerrold Weiss, 1993, Immunobiology.

Prospects for use of recombinant BPI in the treatment of gram-negative bacterial infections.

P. Elsbach, J. Weiss, 1995, Infectious agents and disease.

Lipopolysaccharide (LPS)-binding Proteins BPI and LBP Form Different Types of Complexes with LPS*

P. Tobias, P. Elsbach, J. Weiss, 1997, The Journal of Biological Chemistry.

Detecting lipopolysaccharide in the cytosol of mammalian cells: Lessons from MD‐2/TLR4

J. Weiss, Jason Barker, Jason H. Barker, 2019, Journal of leukocyte biology.

Isolation of an endotoxin–MD-2 complex that produces Toll-like receptor 4-dependent cell activation at picomolar concentrations

N. Coussens, S. Ramaswamy, J. Weiss, 2004, Proceedings of the National Academy of Sciences of the United States of America.

Wall Teichoic Acid Deficiency in Staphylococcus aureus Confers Selective Resistance to Mammalian Group IIA Phospholipase A2 and Human β-Defensin 3

C. Weidenmaier, A. Peschel, J. Weiss, 2008, Infection and Immunity.

Endotoxin·albumin complexes transfer endotoxin monomers to MD-2 resulting in activation of TLR4

J. Weiss, T. Gioannini, A. Teghanemt, 2012, Innate immunity.

Specific High Affinity Interactions of Monomeric Endotoxin·Protein Complexes with Toll-like Receptor 4 Ectodomain*

F. Re, J. Weiss, T. Gioannini, 2007, Journal of Biological Chemistry.

Uniform lipopolysaccharide (LPS)-loaded magnetic nanoparticles for the investigation of LPS-TLR4 signaling.

F. Granucci, M. Colombo, P. Tortora, 2011, Angewandte Chemie.

Molecular Basis of the Functional Differences between Soluble Human Versus Murine MD-2: Role of Val135 in Transfer of Lipopolysaccharide from CD14 to MD-2

J. Weiss, R. Jerala, T. Gioannini, 2016, The Journal of Immunology.

A 25-kDa NH2-terminal fragment carries all the antibacterial activities of the human neutrophil 60-kDa bactericidal/permeability-increasing protein.

C. E. Ooi, P. Elsbach, J. Weiss, 1987, The Journal of biological chemistry.

Bactericidal/permeability increasing protein and host defense against gram-negative bacteria and endotoxin.

P. Elsbach, J. Weiss, Jerrold Weiss, 1993, Current opinion in immunology.

The TLR4 antagonist Eritoran protects mice from lethal influenza infection

Alison J. Scott, F. Gusovsky, C. Karp, 2013, Nature.

A Novel Role for the Bactericidal/Permeability Increasing Protein in Interactions of Gram-Negative Bacterial Outer Membrane Blebs with Dendritic Cells1

J. Weiss, T. Gioannini, De-sheng Zhang, 2007, The Journal of Immunology.

Separation and purification of a potent bactericidal/permeability-increasing protein and a closely associated phospholipase A2 from rabbit polymorphonuclear leukocytes. Observations on their relationship.

A. Schneider, P. Elsbach, J. Weiss, 1979, The Journal of biological chemistry.

The potent anti-Staphylococcus aureus activity of a sterile rabbit inflammatory fluid is due to a 14-kD phospholipase A2.

P. Elsbach, J. Weiss, Y. Weinrauch, 1996, The Journal of clinical investigation.

Cation-Induced Transcriptional Regulation of the dlt Operon of Staphylococcus aureus

A. Peschel, J. Weiss, T. Koprivnjak, 2006, Journal of bacteriology.

Purification and characterization of a potent bactericidal and membrane active protein from the granules of human polymorphonuclear leukocytes.

H. Odeberg, I. Olsson, P. Elsbach, 1978, The Journal of biological chemistry.

Deacylation of Purified Lipopolysaccharides by Cellular and Extracellular Components of a Sterile Rabbit Peritoneal Inflammatory Exudate

R. Munford, P. Elsbach, J. Weiss, 1999, Infection and Immunity.

A Synthetic Lipid A Mimetic Modulates Human TLR4 Activity

F. Peri, J. Weiss, T. Gioannini, 2012, ChemMedChem.

Biochemical Transformation of Bacterial Lipopolysaccharide by acyloxyacyl hydrolase reduces host injury and promotes recovery.

R. Munford, J. Weiss, Mingfang Lu, 2020, The Journal of biological chemistry.

LBP/BPI proteins and their relatives: conservation over evolution and roles in mutualism.

M. McFall-Ngai, J. Weiss, Benjamin C. Krasity, 2011, Biochemical Society transactions.

Phospholipid Synthesis by Staphylococcus aureus during (Sub)Lethal Attack by Mammalian 14-Kilodalton Group IIA Phospholipase A2

P. Elsbach, J. Weiss, A. K. Foreman-Wykert, 2000, Infection and Immunity.

Neutrophils and after Phagocytosis by Human Action before 2 Secretory Phospholipase A on Host Staphylococcus aureus Acids of Effect of d-Alanylation of ( Lipo ) Teichoic

W. Nauseef, J. Weiss, C. L. Hunt, 2006 .

Neutrophil Bleaching of GFP-Expressing Staphylococci: Probing the Intraphagosomal Fate of Individual Bacteria1

W. Nauseef, J. Weiss, J. Femling, 2009, The Journal of Immunology.

Effect of d-Alanylation of (Lipo)Teichoic Acids of Staphylococcus aureus on Host Secretory Phospholipase A2 Action before and after Phagocytosis by Human Neutrophils1

W. Nauseef, J. Weiss, C. L. Hunt, 2006, The Journal of Immunology.

17(R)‐Resolvin D1 differentially regulates TLR4‐mediated responses of primary human macrophages to purified LPS and live E. coli

O. Levy, E. Guinan, C. Serhan, 2011, Journal of leukocyte biology.

Role of the bactericidal/permeability-increasing protein in host defence.

P. Elsbach, J. Weiss, 1998, Current opinion in immunology.

The Carboxyl-terminal Domain of Closely Related Endotoxin-binding Proteins Determines the Target of Protein-Lipopolysaccharide Complexes*

P. Elsbach, J. Weiss, N. Iovine, 2002, The Journal of Biological Chemistry.

An opsonic function of the neutrophil bactericidal/permeability-increasing protein depends on both its N- and C-terminal domains.

P. Elsbach, J. Weiss, N. Iovine, 1997, Proceedings of the National Academy of Sciences of the United States of America.

Both N- and C-terminal regions of the bioactive N-terminal fragment of the neutrophil granule bactericidal/permeability-increasing protein are required for stability and function.

J. Weiss, C. Capodici, 1996, Journal of immunology.

Regulation of interactions of Gram-negative bacterial endotoxins with mammalian cells

J. Weiss, T. Gioannini, 2007, Immunologic research.

Lipid Metabolism by Phagocytic Cells

P. Elsbach, J. Weiss, 1980 .

Lipopolysaccharide deacylation by an endogenous lipase controls innate antibody responses to Gram-negative bacteria

Xia Li, R. Munford, M. Apicella, 2005, Nature Immunology.

Molecular Basis of Reduced Potency of Underacylated Endotoxins1

J. Weiss, T. Gioannini, A. Teghanemt, 2005, The Journal of Immunology.

Interactions Conferred through Hydrophobic TLR4/MD-2 Paclitaxel-Selective Activation of Tetraacylated Lipid A and

J. Weiss, T. Gioannini, A. Oblak, 2014 .

Bactericidal/permeability-increasing protein (BPI) and lipopolysaccharide-binding protein (LBP): structure, function and regulation in host defence against Gram-negative bacteria.

J. Weiss, Jerrold Weiss, 2003, Biochemical Society transactions.

Role of Charge Properties of Bacterial Envelope in Bactericidal Action of Human Group IIA Phospholipase A2against Staphylococcus aureus*

M. Gelb, A. Peschel, J. Weiss, 2002, The Journal of Biological Chemistry.

Mobilization and function of extracellular phospholipase A2 in inflammation.

J. Weiss, G. Wright, 1990, Advances in experimental medicine and biology.

Radioiodination of an endotoxin·MD-2 complex generates a novel sensitive, high-affinity ligand for TLR4

J. Weiss, T. Gioannini, A. Teghanemt, 2013, Innate immunity.

Isolation of Monomeric and Dimeric Secreted MD-2

J. Weiss, T. Gioannini, A. Teghanemt, 2008, Journal of Biological Chemistry.

Functional Activity of MD-2 Polymorphic Variant Is Significantly Different in Soluble and TLR4-Bound Forms: Decreased Endotoxin Binding by G56R MD-2 and Its Rescue by TLR4 Ectodomain1

J. Weiss, R. Jerala, T. Gioannini, 2008, The Journal of Immunology.

Novel Roles in Human MD-2 of Phenylalanines 121 and 126 and Tyrosine 131 in Activation of Toll-like Receptor 4 by Endotoxin*

F. Re, J. Weiss, T. Gioannini, 2008, Journal of Biological Chemistry.

Transfer of Monomeric Endotoxin from MD-2 to CD14

J. Weiss, T. Gioannini, A. Teghanemt, 2007, Journal of Biological Chemistry.

Antibacterial proteins of granulocytes differ in interaction with endotoxin. Comparison of bactericidal/permeability-increasing protein, p15s, and defensins.

M. Doerfler, C. E. Ooi, O. Levy, 1995, Journal of immunology.

Phospholipase A2 activity in the plasma membrane of human polymorphonuclear leukocytes

M. Klempner, P. Elsbach, J. Weiss, 1981, FEBS letters.

Individual and synergistic effects of rabbit granulocyte proteins on Escherichia coli.

C. E. Ooi, O. Levy, R. Lehrer, 1994, The Journal of clinical investigation.

Biochemical and Functional Characterization of Membrane Blebs Purified from Neisseria meningitidis Serogroup B*

B. Gibson, J. Weiss, A. Teghanemt, 2005, Journal of Biological Chemistry.

Antimicrobial peptides of phagocytes and epithelia.

T. Ganz, J. Weiss, Tomas Ganz, 1997, Seminars in hematology.

Oxygen-dependent and oxygen-independent mechanisms of microbicidal activity of neutrophils.

P. Elsbach, J. Weiss, 1985, Immunology letters.

A reevaluation of the roles of the O2-dependent and O2-independent microbicidal systems of phagocytes.

P. Elsbach, J. Weiss, 1983, Reviews of infectious diseases.

Integration of antimicrobial host defenses: role of the bactericidal/permeability-increasing protein.

O. Levy, P. Elsbach, J. Weiss, 1994, Trends in microbiology.

Partial characterization and purification of a rabbit granulocyte factor that increases permeability of Escherichia coli.

P. Elsbach, J. Weiss, R. Franson, 1975, The Journal of clinical investigation.

Oxygen-independent intracellular and oxygen-dependent extracellular killing of Escherichia coli S15 by human polymorphonuclear leukocytes.

P. Elsbach, J. Weiss, L. Kao, 1985, The Journal of clinical investigation.

Resistance of gram-negative bacteria to purified bactericidal leukocyte proteins: relation to binding and bacterial lipopolysaccharide structure.

P. Elsbach, J. Weiss, S. Beckerdite-Quagliata, 1980, The Journal of clinical investigation.

Low concentrations of indomethacin inhibit phospholipase A2 of rabbit polymorphonuclear leukocytes.

P. Elsbach, J. Weiss, L. Kaplan, 1978, Proceedings of the National Academy of Sciences of the United States of America.

Cellular and Extracellular Defenses against Staphylococcal Infections

J. Weiss, A. Bayer, M. Yeaman, 2006 .

Deacylation of Lipopolysaccharide in Whole Escherichia coli during Destruction by Cellular and Extracellular Components of a Rabbit Peritoneal Inflammatory Exudate*

R. Munford, P. Elsbach, J. Weiss, 1999, The Journal of Biological Chemistry.

Shigella flexneri is trapped in polymorphonuclear leukocyte vacuoles and efficiently killed

I. Mandic-Mulec, A. Zychlinsky, J. Weiss, 1997, Infection and immunity.

Separation of sublethal and lethal effects of the bactericidal/permeability increasing protein on Escherichia coli.

P. Elsbach, J. Weiss, B. Mannion, 1990, The Journal of clinical investigation.

p15s (15-kD antimicrobial proteins) are stored in the secondary granules of Rabbit granulocytes: implications for antibacterial synergy with the bactericidal/permeability-increasing protein in inflammatory fluids.

P. Elsbach, J. Weiss, K. Zarember, 1997, Blood.

Leukocyte-derived antimicrobial proteins.

J. Weiss, 1994, Current opinion in hematology.

Antibacterial 15-kDa protein isoforms (p15s) are members of a novel family of leukocyte proteins.

C. E. Ooi, O. Levy, P. Elsbach, 1993, The Journal of biological chemistry.

Host Defense Functions of Proteolytically Processed and Parent (Unprocessed) Cathelicidins of Rabbit Granulocytes

P. Elsbach, J. Weiss, K. Zarember, 2002, Infection and Immunity.

Cutting Edge: Mitochondrial Assembly of the NLRP3 Inflammasome Complex Is Initiated at Priming

F. Sutterwala, R. Gottlieb, S. Cassel, 2018, The Journal of Immunology.

High-affinity caspase-4 binding to LPS presented as high molecular mass aggregates or in outer membrane vesicles

J. Weiss, Jason Barker, A. Teghanemt, 2017, Innate immunity.

The role of intramembrane Ca2+ in the hydrolysis of the phospholipids of Escherichia coli by Ca2+-dependent phospholipases.

P. Elsbach, J. Weiss, L. Kao, 1985, The Journal of biological chemistry.

Role of charge and hydrophobic interactions in the action of the bactericidal/permeability-increasing protein of neutrophils on gram-negative bacteria.

P. Elsbach, J. Weiss, M. Victor, 1983, The Journal of clinical investigation.

The role of complement opsonization in interactions between F. tularensis subsp. novicida and human neutrophils.

W. Nauseef, J. Weiss, Jason Barker, 2009, Microbes and infection.

Evidence Suggesting That Francisella tularensis O-Antigen Capsule Contains a Lipid A-Like Molecule That Is Structurally Distinct from the More Abundant Free Lipid A

M. Apicella, J. Weiss, Jason Barker, 2016, PloS one.

Metabolic labeling to characterize the overall composition of Francisella lipid A and LPS grown in broth and in human phagocytes

J. Weiss, Jason Barker, De-sheng Zhang, 2014, Innate immunity.

Environmental modulation of lipopolysaccharide chain length alters the sensitivity of Escherichia coli to the neutrophil bactericidal/permeability-increasing protein

M. Hutzler, J. Weiss, L. Kao, 1986, Infection and immunity.

An Essential Role for Albumin in the Interaction of Endotoxin with Lipopolysaccharide-binding Protein and sCD14 and Resultant Cell Activation*

J. Weiss, T. Gioannini, A. Teghanemt, 2002, The Journal of Biological Chemistry.

MD-2-dependent pulmonary immune responses to inhaled lipooligosaccharides: effect of acylation state.

P. McCray, P. Thorne, J. Weiss, 2008, American journal of respiratory cell and molecular biology.

Purified monomeric ligand.MD-2 complexes reveal molecular and structural requirements for activation and antagonism of TLR4 by Gram-negative bacterial endotoxins

Liping Yu, J. Weiss, T. Gioannini, 2014, Immunologic Research.

Expression of Functional D299G.T399I Polymorphic Variant of TLR4 Depends More on Coexpression of MD-2 Than Does Wild-Type TLR4

J. Weiss, T. Gioannini, Polonca Prohinar, 2010, The Journal of Immunology.

Wild-Type TLR4 on Coexpression of MD-2 Than Does More Polymorphic Variant of TLR4 Depends Expression of Functional D299G.T399I

J. Weiss, T. Gioannini, P. Rallabhandi, 2010 .

Bacterial lipopolysaccharide primes human neutrophils for enhanced release of arachidonic acid and causes phosphorylation of an 85-kD cytosolic phospholipase A2.

M. Doerfler, P. Elsbach, J. Weiss, 1994, The Journal of clinical investigation.

The role of lipopolysaccharides in the action of the bactericidal/permeability-increasing neutrophil protein on the bacterial envelope.

P. Elsbach, J. Weiss, K. Muello, 1984, Journal of immunology.

Human bactericidal/permeability-increasing protein and a recombinant NH2-terminal fragment cause killing of serum-resistant gram-negative bacteria in whole blood and inhibit tumor necrosis factor release induced by the bacteria.

P. Elsbach, J. Weiss, A. Horwitz, 1992, The Journal of clinical investigation.

Isolation of two isoforms of a novel 15-kDa protein from rabbit polymorphonuclear leukocytes that modulate the antibacterial actions of other leukocyte proteins.

C. E. Ooi, O. Levy, P. Elsbach, 1990, The Journal of biological chemistry.

Potent CD14-mediated signalling of human leukocytes by Escherichia coli can be mediated by interaction of whole bacteria and host cells without extensive prior release of endotoxin

M. Doerfler, P. Elsbach, J. Weiss, 1996, Infection and immunity.

Purification and assay of bactericidal/permeability-increasing protein.

J. Weiss, 1994, Methods in enzymology.

Synergy between Extracellular Group IIA Phospholipase A2 and Phagocyte NADPH Oxidase in Digestion of Phospholipids of Staphylococcus aureus Ingested by Human Neutrophils1

W. Nauseef, J. Weiss, J. Femling, 2005, The Journal of Immunology.

Novel Roles of Lysines 122, 125, and 58 in Functional Differences between Human and Murine MD-21

J. Weiss, R. Jerala, T. Gioannini, 2009, The Journal of Immunology.

Murine MD2 Functional Differences between Human and Novel Roles of Lysines 122 , 125 , and 58 in Weiss and Roman Jerala

J. Weiss, R. Jerala, T. Gioannini, 2009 .

Regulation of interactions of endotoxin with host cells

J. Weiss, T. Gioannini, A. Teghanemt, 2003, Journal of endotoxin research.

Phagocytosis of bacteria and phospholipid degradation.

P. Elsbach, J. Weiss, Jerrold Weiss, 1988, Biochimica et biophysica acta.

Bactericidal/Permeability-Increasing Protein (rBPI21) and Fluoroquinolone Mitigate Radiation-Induced Bone Marrow Aplasia and Death

L. Kalish, O. Levy, E. Guinan, 2011, Science Translational Medicine.

Purification of a cellular (granulocyte) and an extracellular (serum) phospholipase A2 that participate in the destruction of Escherichia coli in a rabbit inflammatory exudate.

C. E. Ooi, P. Elsbach, J. Weiss, 1990, The Journal of biological chemistry.

Utilization of labeled Escherichia coli as phospholipase substrate.

P. Elsbach, J. Weiss, 1991, Methods in enzymology.

EARLY AND DISCRETE CHANGES IN PERMEABILITY OF ESCHERICHIA COLI AND CERTAIN OTHER GRAM-NEGATIVE BACTERIA DURING KILLING BY GRANULOCYTES

P. Elsbach, J. Weiss, R. Franson, 1974, The Journal of experimental medicine.

Modification of sample processing for the Limulus amebocyte lysate assay enhances detection of inflammogenic endotoxin in intact bacteria and organic dust

P. Thorne, J. Weiss, B. Kilburg-Basnyat, 2017, Innate immunity.

Respiratory burst facilitates the digestion of Escherichia coli killed by polymorphonuclear leukocytes

P. Elsbach, J. Weiss, L. Kao, 1987, Infection and immunity.

Monomeric endotoxin:protein complexes are essential for TLR4-dependent cell activation

J. Weiss, T. Gioannini, A. Teghanemt, 2005, Journal of endotoxin research.

Identification of single nucleotide polymorphisms in hematopoietic cell transplant patients affecting early recognition of, and response to, endotoxin

L. Kalish, O. Levy, E. Guinan, 2014, Innate immunity.

The use of a phospholipase A-less Escherichia coli mutant to establish the action of granulocyte phospholipase A on bacterial phospholipids during killing by a highly purified granulocyte fraction.

P. Elsbach, J. Weiss, 1977, Biochimica et biophysica acta.

Structural determinants of the action against Escherichia coli of a human inflammatory fluid phospholipase A2 in concert with polymorphonuclear leukocytes.

P. Elsbach, J. Weiss, R. Crowl, 1994, The Journal of biological chemistry.

Mobilization of potent plasma bactericidal activity during systemic bacterial challenge. Role of group IIA phospholipase A2.

J. Weiss, Y. Weinrauch, N. Liang, 1998, The Journal of clinical investigation.

COOPERATIVE ACTION OF PURIFIED BACTERICIDAL/PERMEABILITY INCREASING PROTEIN AND PHOSPHOLIPASE A2 FROM RABBIT POLYMORPHONUCLEAR LEUKOCYTES

P. Elsbach, J. Weiss, 1980 .

Isolation and characterization of a phospholipase A2 from an inflammatory exudate.

P. Elsbach, J. Weiss, R. Dobrow, 1978, Journal of lipid research.

Structural and functional organization of the human neutrophil 60 kDa bactericidal/permeability-increasing protein

C. E. Ooi, P. Elsbach, J. Weiss, 1991, Agents and Actions.

Sensitivity of K1-Encapsulated Escherichia coli to Killing by the Bactericidal/Permeability-Increasing Protein of Rabbit and Human Neutrophils

A. Cross, P. Elsbach, J. Weiss, 1982, Infection and immunity.

The cationic amphiphile 3,4-bis(tetradecyloxy)benzylamine inhibits LPS signaling by competing with endotoxin for CD14 binding.

F. Peri, J. Weiss, T. Gioannini, 2010, Biochemical pharmacology.

Neutrophils exposed to bacterial lipopolysaccharide upregulate NADPH oxidase assembly.

F. DeLeo, M. Apicella, W. Nauseef, 1998, The Journal of clinical investigation.

Extracellular accumulation of potently microbicidal bactericidal/permeability-increasing protein and p15s in an evolving sterile rabbit peritoneal inflammatory exudate.

O. Levy, P. Elsbach, J. Weiss, 1995, The Journal of clinical investigation.

Cellular and subcellular localization of the bactericidal/permeability-increasing protein of neutrophils.

I. Olsson, J. Weiss, 1987, Blood.

Cell-wall determinants of the bactericidal action of group IIA phospholipase A2 against Gram-positive bacteria.

P. Elsbach, J. Weiss, Y. Weinrauch, 1999, The Journal of clinical investigation.

Killing of gram-negative bacteria by polymorphonuclear leukocytes: role of an O2-independent bactericidal system.

P. Elsbach, J. Weiss, M. Victor, 1982, The Journal of clinical investigation.

Amino acid sequence of a basic Agkistrodon halys blomhoffii phospholipase A2. Possible role of NH2-terminal lysines in action on phospholipids of Escherichia coli.

P. Elsbach, J. Weiss, S. Forst, 1986, Biochemistry.

The action of human and rabbit serum phospholipase A2 on Escherichia coli phospholipids.

P. Elsbach, J. Weiss, S. Beckerdite-Quagliata, 1980, Journal of lipid research.

Reversible envelope effects during and after killing of Escherichia coli w by a highly-purified rabbit polymorpho-nuclear leukocyte fraction.

P. Elsbach, J. Weiss, K. Schmeidler, 1976, Biochimica et biophysica acta.

Phospholipase A activity associated with membranes of human polymorphonuclear leucocytes.

P. Elsbach, J. Weiss, J. Spitznagel, 1977, The Biochemical journal.

High-mobility group nucleosome-binding protein 1 acts as an alarmin and is critical for lipopolysaccharide-induced immune responses

M. Bustin, G. de la Rosa, P. Tewary, 2012, The Journal of experimental medicine.

Biochemical transformation of bacterial lipopolysaccharides by acyloxyacyl hydrolase reduces host injury and promotes recovery.

R. Munford, J. Weiss, Mingfang Lu, 2020, The Journal of biological chemistry.

Purified monomeric ligand.MD-2 complexes reveal molecular and structural requirements for activation and antagonism of TLR4 by Gram-negative bacterial endotoxins

Liping Yu, J. Weiss, T. Gioannini, 2014, Immunologic Research.

Tetraacylated Lipid A and Paclitaxel-Selective Activation of TLR4/MD-2 Conferred through Hydrophobic Interactions

J. Weiss, R. Jerala, T. Gioannini, 2014, The Journal of Immunology.

Respiratory Syncytial Virus Fusion Protein-Induced Toll-Like Receptor 4 (TLR4) Signaling Is Inhibited by the TLR4 Antagonists Rhodobacter sphaeroides Lipopolysaccharide and Eritoran (E5564) and Requires Direct Interaction with MD-2

M. Boukhvalova, S. Vogel, J. Weiss, 2012, mBio.

NMR Studies of Hexaacylated Endotoxin Bound to Wild-type and F126A Mutant MD-2 and MD-2·TLR4 Ectodomain Complexes*

Liping Yu, J. Weiss, T. Gioannini, 2012, The Journal of Biological Chemistry.

Evidence of a specific interaction between new synthetic antisepsis agents and CD14.

Liping Yu, F. Peri, J. Weiss, 2009, Biochemistry.

Endotoxin-binding Proteins Modulate the Susceptibility of Bacterial Endotoxin to Deacylation by Acyloxyacyl Hydrolase*

R. Munford, J. Weiss, T. Gioannini, 2007, Journal of Biological Chemistry.

Effect of lipopolysaccharide (LPS) chain length on interactions of bactericidal/permeability-increasing protein and its bioactive 23-kilodalton NH2-terminal fragment with isolated LPS and intact Proteus mirabilis and Escherichia coli

P. Elsbach, J. Weiss, C. Capodici, 1994, Infection and immunity.

High-affinity binding of the bactericidal/permeability-increasing protein and a recombinant amino-terminal fragment to the lipid A region of lipopolysaccharide

P. Elsbach, J. Weiss, P. Conlon, 1992, Infection and immunity.

Isolation and characterization of a phospholipase A2 from an inflammatory exudate.

P. Elsbach, J. Weiss, R. Dobrow, 1978, Journal of lipid research.

26 Antimicrobial activity of host cells

F. DeLeo, W. Nauseef, J. Weiss, 2002 .

Determinants of activation by complement of group II phospholipase A2 acting against Escherichia coli

J. Weiss, M. Inada, L. Madsen, 1996, Infection and immunity.

IFN-α and Lipopolysaccharide Upregulate APOBEC3 mRNA through Different Signaling Pathways

P. Jones, J. Weiss, C. Okeoma, 2012, The Journal of Immunology.

Endotoxin responsiveness of human airway epithelia is limited by low expression of MD-2.

J. Kline, P. McCray, M. Apicella, 2004, American journal of physiology. Lung cellular and molecular physiology.

OmpR‐dependent and OmpR‐independent responses of Escherichia coli to sublethal attack by the neutrophil bactericidal/permeability increasing protein

I. Mandic-Mulec, J. Weiss, S. Forst, 2002, Molecular microbiology.

The endotoxin‐binding bactericidal/permeability‐increasing protein (BPI): a target antigen of autoantibodies

S. Carroll, J. Weiss, H. Schultz, 2001, Journal of leukocyte biology.

Structural and functional properties of a phospholipase A2 purified from an inflammatory exudate.

I. Reardon, R. Heinrikson, J. Maraganore, 1986, Biochemistry.

Hemin and a metabolic derivative coprohemin modulate the TLR4 pathway differently through different molecular targets

B. Costa, F. Peri, J. Weiss, 2011, Innate immunity.

The bactericidal/permeability-increasing protein (BPI) in infection and inflammatory disease.

J. Weiss, Hendrik Schultz, Jerrold P Weiss, 2007, Clinica chimica acta; international journal of clinical chemistry.

Determinants of the inhibitory action of purified 14-kDa phospholipases A2 on cell-free prothrombinase complex.

J. Weiss, H. Verheij, R. Crowl, 1994, The Journal of biological chemistry.

Molecular determinants of bacterial sensitivity and resistance to mammalian Group IIA phospholipase A2.

J. Weiss, 2015, Biochimica et biophysica acta.

Essential Roles of Hydrophobic Residues in Both MD-2 and Toll-like Receptor 4 in Activation by Endotoxin*

P. Pristovsek, J. Weiss, R. Jerala, 2009, Journal of Biological Chemistry.

Endotoxin-neutralizing properties of the 25 kD N-terminal fragment and a newly isolated 30 kD C-terminal fragment of the 55-60 kD bactericidal/permeability-increasing protein of human neutrophils

M. Doerfler, C. E. Ooi, P. Elsbach, 1991, The Journal of experimental medicine.

Relation between binding and the action of phospholipases A2 on Escherichia coli exposed to the bactericidal/permeability-increasing protein of neutrophils.

R. Heinrikson, J. Maraganore, P. Elsbach, 1987, Biochimica et biophysica acta.

Determinants of the action of phospholipases A on the envelope phospholipids of Escherichia coli.

P. Elsbach, J. Weiss, S. Beckerdite-Quagliata, 1979, The Journal of biological chemistry.

Human granulocytes express a 55-kDa lipopolysaccharide-binding protein on the cell surface that is identical to the bactericidal/permeability-increasing protein.

J. Verhoef, P. Elsbach, J. Weiss, 1993, Journal of immunology.

Cloning of the cDNA of a human neutrophil bactericidal protein. Structural and functional correlations.

C. E. Ooi, P. Gray, P. Elsbach, 1989, The Journal of biological chemistry.

Separation of sublethal and lethal effects of polymorphonuclear leukocytes on Escherichia coli.

P. Elsbach, J. Weiss, B. Mannion, 1990, The Journal of clinical investigation.

Organism-Specific Neutrophil-Endothelial Cell Interactions in Response to Escherichia coli, Streptococcus pneumoniae, and Staphylococcus aureus1

W. Nauseef, J. Weiss, J. Moreland, 2004, The Journal of Immunology.

Low concentrations of indomethacin inhibit phospholipase A 2 of rabbit polymorphonuclear leukocytes ( inflammation / prostaglandin synthesis

P. Elsbach, J. Weiss, L. Kaplan, 2022 .

The phospholipid-repair system LplT/Aas in Gram-negative bacteria protects the bacterial membrane envelope from host phospholipase A2 attack

Z. Guan, W. Margolin, M. Bogdanov, 2018, The Journal of Biological Chemistry.

Characterization of Staphylococcus aureus Cardiolipin Synthases 1 and 2 and Their Contribution to Accumulation of Cardiolipin in Stationary Phase and within Phagocytes

A. Peschel, W. Nauseef, J. Weiss, 2011, Journal of bacteriology.

Monitoring of Endotoxin-Induced Pulmonary Inflammation In Vivo in NF-kB Luciferase Transgenic Mice

C. Wohlford-Lenane, P. McCray, P. Thorne, 2006 .

How bacteria initiate inflammation: aspects of the emerging story.

A. Zychlinsky, J. Weiss, D. Hersh, 1998, Current opinion in microbiology.

Structural and Functional Features of a Developmentally Regulated Lipopolysaccharide-Binding Protein

W. Goldman, E. Lehnert, M. McFall-Ngai, 2015, mBio.

Preferential binding of the neutrophil cytoplasmic granule-derived bactericidal/permeability increasing protein to target bacteria. Implications and use as a means of purification.

P. Elsbach, J. Weiss, B. Mannion, 1989, Journal of immunology.

Isolation of the bactericidal/permeability-increasing protein from polymorphonuclear leukocytes by reversible binding to target bacteria.

J. Weiss, 2000, Methods in Molecular Medicine.

The role of phospholipase A2 lysines in phospholipolysis of Escherichia coli killed by a membrane-active neutrophil protein.

P. Elsbach, J. Weiss, S. Forst, 1982, The Journal of biological chemistry.

A recombinant amino terminal fragment of bactericidal/permeability‐increasing protein inhibits the induction of leukocyte responses by LPS

P. Elsbach, J. Weiss, P. Conlon, 1993, Journal of leukocyte biology.

Myeloablative Hematopoietic Stem Cell Transplantation (HSCT) Is Accompanied by Endotoxemia, Activation of Endotoxin-Directed Innate Immunity, and Deficiency of Endogenous Proteins That Limit Endotoxin- Induced TNF Production

L. Kalish, O. Levy, E. Guinan, 2008 .

The bactericidal/permeability increasing protein of neutrophils is a potent antibacterial and anti-endotoxin agent in vitro and in vivo.

M. Doerfler, F. Kohn, A. Kung, 1994, Progress in clinical and biological research.

Bacterial phospholipid hydrolysis enhances the destruction of Escherichia coli ingested by rabbit neutrophils. Role of cellular and extracellular phospholipases.

G. C. Wright, P. Elsbach, J. Weiss, 1990, The Journal of clinical investigation.

Endotoxin-neutralizing properties of the 25 kD N-terminal fragment and a newly isolated 30 kD C-terminal fragment of the 55-60 kD bactericidal/permeability-increasing protein of human neutrophils

M. Doerfler, P. Elsbach, J. Weiss, 1991, The Journal of experimental medicine.

Killing of gram-negative bacteria by neutrophils: role of O2-independent system in intracellular killing and evidence of O2-dependent extracellular killing.

P. Elsbach, J. Weiss, L. Kao, 1985, Advances in experimental medicine and biology.

Antibacterial Action of Extracellular Mammalian Group IIA Phospholipase A2 against Grossly ClumpedStaphylococcus aureus

J. Weiss, Mary E. Dominiecki, 1999, Infection and Immunity.

This information is current as Signaling Pathways APOBEC 3 mRNA through Different and Lipopolysaccharide Upregulate α IFN -

P. Jones, J. Weiss, C. Okeoma, 2012 .

Early Deficiency of Endogenous Proteins Inhibiting LPS-Induced TNF-α Production Correlates with Acute Graft vs Host Disease (aGVHD) after Myeloablative Stem Cell Transplantation (SCT).

L. Kalish, O. Levy, E. Guinan, 2007 .

Effects of the bactericidal/permeability-increasing protein of polymorphonuclear leukocytes on isolated bacterial cytoplasmic membrane vesicles

P. Elsbach, J. Weiss, B. Mannion, 1988, Infection and immunity.

Heparin inhibits phagocytosis by polymorphonuclear leukocytes

P. Elsbach, J. Weiss, M. Victor, 1981, Infection and immunity.

O2-independent killing of gram-negative bacteria by intact granulocytes. The role of a potent bactericidal membrane-perturbing protein.

P. Elsbach, J. Weiss, O. Stendahl, 1982, Advances in experimental medicine and biology.

Evidence of a specific interaction between new synthetic antisepsis agents and CD14.

Liping Yu, F. Peri, J. Weiss, 2009, Biochemistry.

Cellular and subcellular localization of the bactericidal/permeability-increasing protein of neutrophils.

I. Olsson, J. Weiss, 1987, Blood.