D. Lin

发表

Human steroidogenic acute regulatory protein: functional activity in COS-1 cells, tissue-specific expression, and mapping of the structural gene to 8p11.2 and a pseudogene to chromosome 13.

D. Driscoll, W. Miller, T. Sugawara, 1995, Proceedings of the National Academy of Sciences of the United States of America.

Stimulation of Cell Elongation in Teleost Rod Photoreceptors by Distinct Protein Kinase Inhibitors

B. Burnside, D. Lin, K. Pagh-Roehl, 1996, Journal of neurochemistry.

Mutations in the human sterol delta7-reductase gene at 11q12-13 cause Smith-Lemli-Opitz syndrome.

D. S. Lin, C. Maslen, R. Steiner, 1998, American journal of human genetics.

T-->A transversion 11 bp from a splice acceptor site in the human gene for steroidogenic acute regulatory protein causes congenital lipoid adrenal hyperplasia.

W. Miller, T. Sugawara, J. Holt, 1995, Human molecular genetics.

Physical and cDNA Mapping in the DBH Region of Human Chromosome 9q34

J. Gilbert, M. Pericak-Vance, P. Xu, 2000, Human Heredity.

Mutation of the serine 15 phosphorylation site of human p53 reduces the ability of p53 to inhibit cell cycle progression.

M. Fiscella, E. Appella, N. Zambrano, 1993, Oncogene.

Steroid 17 alpha-hydroxylase and 17,20-lyase activities of P450c17: contributions of serine106 and P450 reductase.

S. Black, W. Miller, D. Lin, 1993, Endocrinology.

Structure of the human steroidogenic acute regulatory protein (StAR) gene: StAR stimulates mitochondrial cholesterol 27-hydroxylase activity.

W. Miller, T. Sugawara, J. Holt, 1995, Biochemistry.

The carboxy-terminal serine 392 phosphorylation site of human p53 is not required for wild-type activities.

M. Fiscella, E. Appella, T. Unger, 1994, Oncogene.

Missense mutation serine106----proline causes 17 alpha-hydroxylase deficiency.

K. Jones, J. Harikrishna, W. Miller, 1991, The Journal of biological chemistry.

Deletion of amino acids Asp487-Ser488-Phe489 in human cytochrome P450c17 causes severe 17 alpha-hydroxylase deficiency.

W. Miller, D. Lin, C. Fardella, 1993, The Journal of clinical endocrinology and metabolism.

Normal genes for the cholesterol side chain cleavage enzyme, P450scc, in congenital lipoid adrenal hyperplasia.

S. Gitelman, W. Miller, D. Lin, 1991, The Journal of clinical investigation.

Congenital lipoid adrenal hyperplasia—Genes for P450scc, side chain cleavage enzyme, are normal

S. Gitelman, W. Miller, D. Lin, 1993, The Journal of Steroid Biochemistry and Molecular Biology.

Cloning and sequence of the human adrenodoxin reductase gene.

W. Miller, D. Lin, Y. F. Shi, 1990, Proceedings of the National Academy of Sciences of the United States of America.

Induction of WAF1/CIP1 by a p53-independent pathway.

D. Givol, W. Mercer, J. Pierce, 1994, Cancer research.

Characterization of the Protein Responsible for the Acute Regulation of Steroidogenesis in Mouse Leydig Tumor Cells

D. Stocco, W. Miller, T. Sugawara, 1996 .

Cloning of human 25-hydroxyvitamin D-1 alpha-hydroxylase and mutations causing vitamin D-dependent rickets type 1.

W. Miller, C. Shackleton, D. Lin, 1997, Molecular endocrinology.

Growth arrest induced by wild-type p53 protein blocks cells prior to or near the restriction point in late G1 phase.

E. Appella, D. Lin, W. Mercer, 1992, Proceedings of the National Academy of Sciences of the United States of America.

Phosducin and PP33 are in vivo targets of PKA and type 1 or 2A phosphatases, regulators of cell elongation in teleost rod inner-outer segments

B. Burnside, L. Su, D. Lin, 1995, The Journal of neuroscience : the official journal of the Society for Neuroscience.

Constitutive expression of B-myb can bypass p53-induced Waf1/Cip1-mediated G1 arrest.

M. Fiscella, E. Appella, P. O'Connor, 1994, Proceedings of the National Academy of Sciences of the United States of America.

Growth suppression induced by wild-type p53 protein is accompanied by selective down-regulation of proliferating-cell nuclear antigen expression.

E. Appella, W. Mercer, S. Ullrich, 1991, Proceedings of the National Academy of Sciences of the United States of America.

Modeling and mutagenesis of the active site of human P450c17.

E. Chiao, W. Miller, D. Lin, 1994, Molecular endocrinology.

The human peripheral benzodiazepine receptor gene: cloning and characterization of alternative splicing in normal tissues and in a patient with congenital lipoid adrenal hyperplasia.

W L Miller, W. Miller, J. Strauss, 1993, Genomics.

Chk1, but not Chk2, is responsible for G2/M phase arrest induced by diallyl disulfide in human gastric cancer BGC823 cells.

D. Lin, S. Qi, Xiaohong Ai, 2014, Food and chemical toxicology : an international journal published for the British Industrial Biological Research Association.

Role of steroidogenic acute regulatory protein in adrenal and gonadal steroidogenesis.

A. Rogol, D. Stocco, W. Miller, 1995, Science.

Human severe combined immunodeficiency due to a defect in ZAP-70, a T cell tyrosine kinase.

T. Hope, A. Weiss, T. Parslow, 1994, Science.

cAMP post-transcriptionally diminishes the abundance of adrenodoxin reductase mRNA.

S. Black, W. Miller, D. Lin, 1992, Proceedings of the National Academy of Sciences of the United States of America.