A. Bohrer

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Isotope Dilution Mass Spectrometric Measurements Indicate That Arachidonylethanolamide, the Proposed Endogenous Ligand of the Cannabinoid Receptor, Accumulates in Rat Brain Tissue Post Mortem but Is Contained at Low Levels in or Is Absent from Fresh Tissue*

J. Turk, F. Hsu, J. Turk, 1996, The Journal of Biological Chemistry.

Cholesterol chlorohydrin synthesis by the myeloperoxidase-hydrogen peroxide-chloride system: potential markers for lipoproteins oxidatively damaged by phagocytes.

Wei Li, J. Turk, J. Turk, 1994, Biochemistry.

Insulin Secretory Responses and Phospholipid Composition of Pancreatic Islets from Mice That Do Not Express Group VIA Phospholipase A2 and Effects of Metabolic Stress on Glucose Homeostasis*

Haowei Song, J. Turk, A. Bohrer, 2006, Journal of Biological Chemistry.

Mass spectrometric characterization of arachidonate-containing plasmalogens in human pancreatic islets and in rat islet beta-cells and subcellular membranes.

R. Gross, J. Turk, A. Bohrer, 1993, Biochemistry.

Effects of Stable Suppression of Group VIA Phospholipase A2 Expression on Phospholipid Content and Composition, Insulin Secretion, and Proliferation of INS-1 Insulinoma Cells*

S. Zhang, J. Turk, A. Bohrer, 2006, Journal of Biological Chemistry.

Mass spectrometric identification and quantitation of arachidonate-containing phospholipids in pancreatic islets: prominence of plasmenylethanolamine molecular species.

R. Gross, J. Turk, A. Bohrer, 1993, Biochemistry.

Studies of phospholipid metabolism, proliferation, and secretion of stably transfected insulinoma cells that overexpress group VIA phospholipase A2

F. Hsu, J. Turk, Zhongming Ma, 2001, Lipids.

Quantification of distinct molecular species of the 2-lyso metabolite of platelet-activating factor by gas chromatography-negative-ion chemical ionization mass spectrometry.

J. Turk, M. Mangino, A. Bohrer, 1992, Journal of chromatography.

Electrospray ionization tandem mass spectrometric analysis of sulfatide. Determination of fragmentation patterns and characterization of molecular species expressed in brain and in pancreatic islets.

J. Turk, F. Hsu, A. Bohrer, 1998, Biochimica et biophysica acta.

Inhibition of 3-Hydroxy-3-Methylglutaryl Coenzyme A (HMG CoA) Reductase Blunts Factor VIIa/Tissue Factor and Prothrombinase Activities via Effects on Membrane Phosphatidylserine

D. Dietzen, J. Turk, A. Bohrer, 2007, Arteriosclerosis, thrombosis, and vascular biology.

THE GROUP VIA CALCIUM-INDEPENDENT PHOSPHOLIPASE A2 (iPLA2β) PARTICIPATES IN ER STRESS-INDUCED INS-1 INSULINOMA CELL APOPTOSIS BY PROMOTING CERAMIDE GENERATION VIA HYDROLYSIS OF SPHINGOMYELINS BY

A. Bohrer, X. Lei, Shunzhong S Bao, 2008 .

The group VIA calcium-independent phospholipase A2 participates in ER stress-induced INS-1 insulinoma cell apoptosis by promoting ceramide generation via hydrolysis of sphingomyelins by neutral sphingomyelinase.

Haowei Song, A. Bohrer, X. Lei, 2007, Biochemistry.

Attenuated Free Cholesterol Loading-induced Apoptosis but Preserved Phospholipid Composition of Peritoneal Macrophages from Mice That Do Not Express Group VIA Phospholipase A2*

J. Turk, C. Semenkovich, Yankun Li, 2007, Journal of Biological Chemistry.

Formation of lithiated adducts of glycerophosphocholine lipids facilitates their identification by electrospray ionization tandem mass spectrometry

F. Hsu, J. Turk, A. Bohrer, 1998, Journal of the American Society for Mass Spectrometry.

Apoptosis of insulin-secreting cells induced by endoplasmic reticulum stress is amplified by overexpression of group VIA calcium-independent phospholipase A2 (iPLA2 beta) and suppressed by inhibition of iPLA2 beta.

Haowei Song, F. Hsu, J. Turk, 2004, Biochemistry.

Studies of the Role of Group VI Phospholipase A2 in Fatty Acid Incorporation, Phospholipid Remodeling, Lysophosphatidylcholine Generation, and Secretagogue-induced Arachidonic Acid Release in Pancreatic Islets and Insulinoma Cells*

F. Hsu, J. Turk, Zhongming Ma, 1999, The Journal of Biological Chemistry.

Studies of Insulin Secretory Responses and of Arachidonic Acid Incorporation into Phospholipids of Stably Transfected Insulinoma Cells That Overexpress Group VIA Phospholipase A2(iPLA2β) Indicate a Signaling Rather Than a Housekeeping Role for iPLA2β*

F. Hsu, J. Turk, Zhongming Ma, 2001, The Journal of Biological Chemistry.

Role of calcium-independent phospholipase A(2)β in human pancreatic islet β-cell apoptosis.

S. Zhang, A. Bohrer, X. Lei, 2012, American journal of physiology. Endocrinology and metabolism.

Synthesis of the 1-O-hexadecyl molecular species of platelet-activating factor by airway epithelial and vascular endothelial cells.

J. Turk, M. Holtzman, A. Bohrer, 1991, Biochemical and biophysical research communications.

Electrospray ionization mass spectrometric analyses of phospholipids from INS-1 insulinoma cells: comparison to pancreatic islets and effects of fatty acid supplementation on phospholipid composition and insulin secretion.

J. Turk, F. Hsu, A. Bohrer, 2000, Biochimica et biophysica acta.

Δ6-, stearoyl CoA-, and Δ5-desaturase enzymes are expressed in β-cells and are altered by increases in exogenous PUFA concentrations

F. Hsu, J. Turk, Zhongming Ma, 2002 .

Distinction among eight opiate drugs in urine by gas chromatography-mass spectrometry.

J. Turk, A. Bohrer, W. Nowatzke, 1999, Journal of pharmaceutical and biomedical analysis.

Glucose homeostasis, insulin secretion, and islet phospholipids in mice that overexpress iPLA2β in pancreatic β-cells and in iPLA2β-null mice

L. Philipson, J. Turk, A. Bohrer, 2008 .

Biochemical evidence for nitric oxide formation from streptozotocin in isolated pancreatic islets.

M. Mcdaniel, J. Corbett, J. Turk, 1993, Biochemical and biophysical research communications.

Spontaneous development of ER stress that can lead to diabetes mellitus is associated with higher calcium-independent phospholipase A2 (iPLA2beta) expression: A role for regulation by SREBP-1

F. R. Papa, S. Zhang, A. Bohrer, 2009 .

Evidence of contribution of iPLA2β-mediated events during islet β-cell apoptosis due to proinflammatory cytokines suggests a role for iPLA2β in T1D development.

A. Bohrer, K. Bidasee, X. Lei, 2014, Endocrinology.

Glucose-responsitivity and expression of an ATP-stimulatable, Ca(2+)-independent phospholipase A2 enzyme in clonal insulinoma cell lines.

J. Turk, A. Bohrer, S. Ramanadham, 1997, Biochimica et biophysica acta.

Delta6-, Stearoyl CoA-, and Delta5-desaturase enzymes are expressed in beta-cells and are altered by increases in exogenous PUFA concentrations.

S. Zhang, F. Hsu, J. Turk, 2002, Biochimica et Biophysica Acta.

Arachidonic acid metabolism in isolated pancreatic islets. VI. Carbohydrate insulin secretagogues must be metabolized to induce eicosanoid release.

J. Turk, A. Bohrer, S. Ramanadham, 1992, Biochimica et biophysica acta.

Characterization of the sphingomyelin content of isolated pancreatic islets. Evaluation of the role of sphingomyelin hydrolysis in the action of interleukin-1 to induce islet overproduction of nitric oxide.

M. Mcdaniel, J. Corbett, G. Kwon, 1996, Biochimica et Biophysica Acta.

Genetic modulation of islet β-cell iPLA2β expression provides evidence for its impact on β-cell apoptosis and autophagy

J. Turk, A. Bohrer, X. Lei, 2013, Islets.

Insulin resistance in HIV protease inhibitor-associated diabetes.

K. Yarasheski, P. Cryer, P. Halban, 1999, Journal of acquired immune deficiency syndromes.

Spontaneous Development of Endoplasmic Reticulum Stress That Can Lead to Diabetes Mellitus Is Associated with Higher Calcium-independent Phospholipase A2 Expression

S. Zhang, F. R. Papa, A. Bohrer, 2009, The Journal of Biological Chemistry.

Electrospray Ionization/Mass Spectrometric Analyses of Human Promonocytic U937 Cell Glycerolipids and Evidence That Differentiation Is Associated with Membrane Lipid Composition Changes That Facilitate Phospholipase A2 Activation*

F. Hsu, J. Turk, Zhongming Ma, 2000, The Journal of Biological Chemistry.

Electrospray ionization mass spectrometric analyses of changes in tissue phospholipid molecular species during the evolution of hyperlipidemia and hyperglycemia in Zucker diabetic fatty rats

K. Yarasheski, F. Hsu, J. Turk, 2000, Lipids.

Calcium-independent Phospholipase A2 (iPLA2β)-mediated Ceramide Generation Plays a Key Role in the Cross-talk between the Endoplasmic Reticulum (ER) and Mitochondria during ER Stress-induced Insulin-secreting Cell Apoptosis*

A. Bohrer, X. Lei, S. Ramanadham, 2008, Journal of Biological Chemistry.

Interleukin-1 Reduces the Glycolytic Utilization of Glucose by Pancreatic Islets and Reduces Glucokinase mRNA Content and Protein Synthesis by a Nitric Oxide-dependent Mechanism*

D. Kipnis, J. Turk, Zhongming Ma, 1997, The Journal of Biological Chemistry.

Interleukin-1 Enhances Pancreatic Islet Arachidonic Acid 12-Lipoxygenase Product Generation by Increasing Substrate Availability through a Nitric Oxide-dependent Mechanism (*)

M. Mcdaniel, J. Corbett, R. Gross, 1996, The Journal of Biological Chemistry.

Quantitation of the putative neurotransmitter agmatine as the hexafluoroacetylacetonate derivative by stable isotope dilution gas chromatography and negative-ion chemical ionization mass spectrometry.

S. Klahr, J. Turk, J. Morrissey, 1996, Analytical biochemistry.

Mass Spectrometric Evidence That Agents That Cause Loss of Ca 2؉ from Intracellular Compartments Induce Hydrolysis of Arachidonic Acid from Pancreatic Islet Membrane Phospholipids by a Mechanism That Does Not Require a Rise in Cytosolic Ca 2؉ Concentration*

J. Turk, F. Hsu, A. Bohrer, 2022 .

Electrospray ionization mass spectrometric analyses of phospholipids from rat and human pancreatic islets and subcellular membranes: comparison to other tissues and implications for membrane fusion in insulin exocytosis.

J. Turk, F. Hsu, A. Bohrer, 1998, Biochemistry.

THE GROUP VIA CALCIUM-INDEPENDENT PHOSPHOLIPASE A 2 (iPLA 2 β ) PARTICIPATES IN ER STRESS-INDUCED INS-1 INSULINOMA CELL APOPTOSIS BY PROMOTING CERAMIDE GENERATION VIA HYDROLYSIS OF SPHINGOMYELINS BY NEUTRAL SPHINGOMYELINASE

Haowei Song, A. Bohrer, X. Lei, 2008 .

Mass spectrometric evidence that agents that cause loss of Ca2+ from intracellular compartments induce hydrolysis of arachidonic acid from pancreatic islet membrane phospholipids by a mechanism that does not require a rise in cytosolic Ca2+ concentration.

A. Bohrer, S. Ramanadham, F. Hsu, 1998, Endocrinology.

Glucose homeostasis, insulin secretion, and islet phospholipids in mice that overexpress iPLA2beta in pancreatic beta-cells and in iPLA2beta-null mice.

L. Philipson, J. Turk, A. Bohrer, 2008, American Journal of Physiology. Endocrinology and Metabolism.

Quantification of distinct molecular species of platelet activating factor in ulcerative colitis

J. Turk, A. Bohrer, W. Stenson, 2007, Lipids.