B. Vohra

发表

The Timing and Location of Glial Cell Line-Derived Neurotrophic Factor Expression Determine Enteric Nervous System Structure and Function

C. Keller-Peck, R. Heuckeroth, J. Grider, 2010, Journal of Neuroscience.

Transgenic Mice Expressing the Nmnat1 Protein Manifest Robust Delay in Axonal Degeneration In Vivo

J. Milbrandt, Y. Sasaki, B. Vohra, 2009, The Journal of Neuroscience.

Tributyltin alters calcium levels, mitochondrial dynamics, and activates calpains within dorsal root ganglion neurons.

B. Vohra, Shaneann Fross, Clayton Mansel, 2021, Toxicological sciences : an official journal of the Society of Toxicology.

Axon degeneration is key component of neuronal death in amyloid-β toxicity

W. Xia, B. Vohra, W. Alobuia, 2013, Neurochemistry International.

Mitochondrial Dynamics Decrease Prior to Axon Degeneration Induced by Vincristine and are Partially Rescued by Overexpressed cytNmnat1

B. Vohra, Kari Naylor, Gregory W. Berbusse, 2016, Front. Cell. Neurosci..

Lead exposure reduces survival, neuronal determination, and differentiation of P19 stem cells.

B. Vohra, Shaneann Fross, H. Hart, 2019, Neurotoxicology and teratology.

Reduced endothelin converting enzyme‐1 and endothelin‐3 mRNA in the developing bowel of male mice may increase expressivity and penetrance of Hirschsprung disease–like distal intestinal aganglionosis

R. Heuckeroth, Sanjay Jain, W. Planer, 2007, Developmental dynamics : an official publication of the American Association of Anatomists.

Organotypic specificity of key RET adaptor-docking sites in the pathogenesis of neurocristopathies and renal malformations in mice.

J. Milbrandt, R. Heuckeroth, Sanjay Jain, 2010, The Journal of clinical investigation.

BMP signaling regulates murine enteric nervous system precursor migration, neurite fasciculation, and patterning via altered Ncam1 polysialic acid addition.

R. Heuckeroth, B. Vohra, M. Fu, 2006, Developmental biology.

Amyloid Precursor Protein Cleavage-Dependent and -Independent Axonal Degeneration Programs Share a Common Nicotinamide Mononucleotide Adenylyltransferase 1-Sensitive Pathway

J. Milbrandt, A. Diantonio, Y. Sasaki, 2010, The Journal of Neuroscience.

Withania somnifera (ASHWAGANDHA) ATTENUATES ANTIOXIDANT DEFENSE IN AGED SPINAL CORD AND INHIBITS COPPER INDUCED LIPID PEROXIDATION AND PROTEIN OXIDATIVE MODIFICATIONS

B. Vohra, A. Dua, Sanjeev K. Gupta, 2003, Drug metabolism and drug interactions.

Activation of apoptotic pathways at muscle fiber synapses is circumscribed and reversible in a slow-channel syndrome model

C. Gomez, R. Wollmann, B. Vohra, 2006, Neurobiology of Disease.

Transcriptional profiling after bile duct ligation identifies PAI‐1 as a contributor to cholestatic injury in mice

R. Heuckeroth, Hongtao Wang, B. Vohra, 2005, Hepatology.

Ca2+ toxicity due to reverse Na+/Ca2+ exchange contributes to degeneration of neurites of DRG neurons induced by a neuropathy-associated Nav1.7 mutation.

S. Waxman, J. A. Black, G. Lauria, 2015, Journal of neurophysiology.

Ca 2 toxicity due to reverse Na / Ca 2 exchange contributes to degeneration of neurites of DRG neurons induced by a neuropathy-associated Nav 1 . 7 mutation

S. Waxman, J. A. Black, G. Lauria, 2015 .

Differential Regional and Subtype-Specific Vulnerability of Enteric Neurons to Mitochondrial Dysfunction

J. Milbrandt, R. Heuckeroth, B. Vohra, 2011, PloS one.

Protein Kinase Cζ and Glycogen Synthase Kinase-3β Control Neuronal Polarity in Developing Rodent Enteric Neurons, whereas SMAD Specific E3 Ubiquitin Protein Ligase 1 Promotes Neurite Growth But Does Not Influence Polarity

R. Heuckeroth, B. Vohra, M. Fu, 2007, The Journal of Neuroscience.

Enteric Neurodegeneration is Mediated Through Independent Neuritic and Somal Mechanisms in Rotenone and MPP+ Toxicity

B. Vohra, D. Virga, Jessica Capps, 2018, Neurochemical Research.

Calpain activation impairs neuromuscular transmission in a mouse model of the slow-channel myasthenic syndrome.

C. Gomez, R. Wollmann, E. Kudryashova, 2007, The Journal of clinical investigation.

Nicotinamide Mononucleotide Adenylyl Transferase-Mediated Axonal Protection Requires Enzymatic Activity But Not Increased Levels of Neuronal Nicotinamide Adenine Dinucleotide

J. Milbrandt, Y. Sasaki, B. Vohra, 2009, The Journal of Neuroscience.

Image-based Screening Identifies Novel Roles for IκB Kinase and Glycogen Synthase Kinase 3 in Axonal Degeneration*

J. Milbrandt, Josiah Gerdts, Y. Sasaki, 2011, The Journal of Biological Chemistry.

Role of oxygen consumption in hypoxia protection by translation factor depletion

J. Milbrandt, B. Vohra, X. Mao, 2013, Journal of Experimental Biology.

Differential gene expression and functional analysis implicate novel mechanisms in enteric nervous system precursor migration and neuritogenesis.

Daniel Wind, Hideki Enomoto, H. Enomoto, 2006, Developmental biology.

Improvement of Impaired Memory in Mice by Taurine

B. Vohra, Xiang Hui, 2000, Neural plasticity.

Nicotinamide Mononucleotide Adenylyltransferase 2 maintains neuronal structural integrity through the maintenance of golgi structure

B. Vohra, Shaneann Fross, Tana S. Pottorf, 2018, Neurochemistry International.

Novel delta subunit mutation in slow‐channel syndrome causes severe weakness by novel mechanisms

J. Stiles, L. Rojas, J. Lasalde-Dominicci, 2002, Annals of neurology.

Enteric Neurodegeneration is Mediated Through Independent Neuritic and Somal Mechanisms in Rotenone and MPP+ Toxicity

B. Vohra, D. Virga, Jessica Capps, 2018, Neurochemical Research.

The Translocase of the Outer Mitochondrial Membrane (TOM40) is required for mitochondrial dynamics and neuronal integrity in Dorsal Root Ganglion Neurons

B. Vohra, Caitlin Overmeyer, Kylie Jorgensen, 2023, Molecular and Cellular Neuroscience.

Focal caspase activation underlies the endplate myopathy in slow‐channel syndrome

R. Maselli, R. Wollmann, B. Vohra, 2004, Annals of neurology.

The Timing and Location of Glial Cell Line-Derived Neurotrophic Factor Expression Determine Enteric Nervous System Structure and Function

C. Keller-Peck, R. Heuckeroth, J. Grider, 2010, The Journal of Neuroscience.

Organotypic specificity of key RET adaptor-docking sites in the pathogenesis of neurocristopathies and renal malformations in mice.

J. Milbrandt, R. Heuckeroth, Hongtao Wang, 2010, The Journal of clinical investigation.

Organotypic Specificity of Key Ret Adaptor- Docking Sites in the Pathogenesis of Neurocristopathies and Renal Malformations in Mice Recommended Citation Organotypic Specificity of Key Ret Adaptor-docking Sites in the Pathogenesis of Neurocristopathies and Renal Malformations in Mice

R. Heuckeroth, B. Vohra, Amanda Knoten, 2022 .