Y. Krelin

发表

IL-1 is required for tumor invasiveness and angiogenesis

Y. Iwakura, C. Dinarello, E. Voronov, 2003, Proceedings of the National Academy of Sciences of the United States of America.

The precursor form of IL-1alpha is an intracrine proinflammatory activator of transcription.

C. Dinarello, E. Voronov, Y. Krelin, 2004, Proceedings of the National Academy of Sciences of the United States of America.

A molecular signature of lung cancer: potential biomarkers for adenocarcinoma and squamous cell carcinoma

V. Shoshan-Barmatz, Avijit Paul, Y. Krelin, 2017, Oncotarget.

Macrophages mediate gemcitabine resistance of pancreatic adenocarcinoma by upregulating cytidine deaminase

R. Wong, M. Amit, Z. Gil, 2014, Oncogene.

IL-1-induced inflammation promotes development of leishmaniasis in susceptible BALB/c mice.

M. Elkabets, E. Voronov, Y. Krelin, 2010, International immunology.

CD11b+/Gr-1+ Immature Myeloid Cells Mediate Suppression of T Cells in Mice Bearing Tumors of IL-1β-Secreting Cells1

S. Segal, C. Dinarello, E. Voronov, 2005, The Journal of Immunology.

The involvement of IL-1 in tumorigenesis, tumor invasiveness, metastasis and tumor-host interactions

Y. Carmi, M. Elkabets, E. Voronov, 2006, Cancer and Metastasis Reviews.

Opposing Effects of IL-1α and IL-1β on Malignancy Patterns

S. Argov, M. Zöller, S. Segal, 2002 .

Opposing effects of IL-1 alpha and IL-1 beta on malignancy patterns. Tumor cell-associated IL-1 alpha potentiates anti-tumor immune responses and tumor regression, whereas IL-1 beta potentiates invasiveness.

S. Argov, S. Segal, E. Voronov, 2000, Advances in experimental medicine and biology.

NOX1-induced accumulation of reactive oxygen species in abdominal fat-derived mesenchymal stromal cells impinges on long-term proliferation

Y. Krelin, D. Zipori, E. Gur, 2015, Cell Death and Disease.

Immunological and inflammatory mapping of vascularized composite allograft rejection processes in a rat model

Y. Krelin, E. Gur, A. Inbal, 2017, PloS one.

A New Role for the Mitochondrial Pro-apoptotic Protein SMAC/Diablo in Phospholipid Synthesis Associated with Tumorigenesis.

V. Shoshan-Barmatz, Avijit Paul, Y. Krelin, 2017, Molecular therapy : the journal of the American Society of Gene Therapy.

Effects of micro-environment- and malignant cell-derived interleukin-1 in carcinogenesis, tumour invasiveness and tumour-host interactions.

Y. Carmi, S. Segal, M. Elkabets, 2006, European journal of cancer.

Mitochondrial VDAC1-based peptides: Attacking oncogenic properties in glioblastoma

V. Shoshan-Barmatz, Avijit Paul, Y. Krelin, 2017, Oncotarget.

VDAC1 functions in Ca2+ homeostasis and cell life and death in health and disease.

V. Shoshan-Barmatz, Y. Krelin, Anna Shteinfer-Kuzmine, 2018, Cell calcium.

Endoneurial macrophages induce perineural invasion of pancreatic cancer cells by secretion of GDNF and activation of RET tyrosine kinase receptor.

R. Wong, M. Amit, Z. Gil, 2013, Cancer research.

The long noncoding RNA TP73‐AS1 promotes tumorigenicity of medulloblastoma cells

G. Reifenberger, A. Borkhardt, B. Rotblat, 2019, International journal of cancer.

Mitochondrial VDAC1 Silencing Leads to Metabolic Rewiring and the Reprogramming of Tumour Cells into Advanced Differentiated States

V. Shoshan-Barmatz, Avijit Paul, Y. Krelin, 2018, Cancers.

The mitochondrial voltage-dependent anion channel 1 in tumor cells.

V. Shoshan-Barmatz, D. Ben-Hail, Y. Krelin, 2015, Biochimica et biophysica acta.

VDAC1 as a Player in Mitochondria-Mediated Apoptosis and Target for Modulating Apoptosis.

V. Shoshan-Barmatz, Quan Chen, Y. Krelin, 2018, Current medicinal chemistry.

Interleukin-1beta-driven inflammation promotes the development and invasiveness of chemical carcinogen-induced tumors.

Yoichiro Iwakura, S. Segal, M. Elkabets, 2007, Cancer research.

VDAC1 at the crossroads of cell metabolism, apoptosis and cell stress

V. Shoshan-Barmatz, Y. Krelin, E. Maldonado, 2017, Cell stress.

VDAC1 is a molecular target in glioblastoma, with its depletion leading to reprogrammed metabolism and reversed oncogenic properties

V. Shoshan-Barmatz, Avijit Paul, Y. Krelin, 2017, Neuro-oncology.

Reducing VDAC1 expression induces a non-apoptotic role for pro-apoptotic proteins in cancer cell differentiation.

V. Shoshan-Barmatz, Y. Krelin, Tasleem Arif, 2016, Biochimica et biophysica acta.

Unique Versus Redundant Functions of IL-1α and IL-1β in the Tumor Microenvironment

Y. Carmi, M. Elkabets, E. Voronov, 2013, Front. Immunol..

Host-Derived Interleukin-1α Is Important in Determining the Immunogenicity of 3-Methylcholantrene Tumor Cells1

A. Porgador, A. Cerwenka, M. Elkabets, 2009, The Journal of Immunology.

3-Methylcholantrene Tumor Cells Determining the Immunogenicity of Is Important in a Host-Derived Interleukin-1

A. Porgador, A. Cerwenka, M. Elkabets, 2009 .

Targeting Liver Cancer and Associated Pathologies in Mice with a Mitochondrial VDAC1-Based Peptide

V. Shoshan-Barmatz, Y. Krelin, Srinivas Pittala, 2018, Neoplasia.

Voltage-Dependent Anion Channel 1 As an Emerging Drug Target for Novel Anti-Cancer Therapeutics

V. Shoshan-Barmatz, Y. Krelin, Tasleem Arif, 2017, Front. Oncol..

Association between Tumorigenic Potential and the Fate of Cancer Cells in a Syngeneic Melanoma Model

M. Amit, Z. Gil, Y. Krelin, 2013, PloS one.

The 106b∼25 microRNA cluster is essential for neovascularization after hindlimb ischaemia in mice.

I. Barshack, David Kain, G. Keren, 2014, European heart journal.

A Mitochondrial VDAC1-Based Peptide Greatly Suppresses Steatosis and NASH-Associated Pathologies in a Mouse Model.

V. Shoshan-Barmatz, Y. Kuperman, Y. Krelin, 2019, Molecular therapy : the journal of the American Society of Gene Therapy.

Characterization of the melanoma brain metastatic niche in mice and humans

M. Amit, Z. Gil, A. Shabtay-Orbach, 2013, Cancer medicine.

Reducing VDAC1 expression induces a non-apoptotic role for pro-apoptotic proteins in cancer cell differentiation.

V. Shoshan-Barmatz, Y. Krelin, Tasleem Arif, 2016, Biochimica et biophysica acta.