L. Piconi

发表

Oscillating Glucose Is More Deleterious to Endothelial Function and Oxidative Stress Than Mean Glucose in Normal and Type 2 Diabetic Patients

R. Testa, M. Boemi, D. Giugliano, 2008, Diabetes.

Pramlintide reduced markers of oxidative stress in the postprandial period in patients with type 2 diabetes

J. Frias, T. Darsow, D. Maggs, 2008, Diabetes/metabolism research and reviews.

Attenuated Superoxide Dismutase Induction in Retinal Cells in Response to Intermittent High Versus Continuous High Glucose

M. Ihnat, A. Ceriello, L. Piconi, 2007 .

Effects of pramlintide on postprandial glucose excursions and measures of oxidative stress in patients with type 1 diabetes.

C. Weyer, C. Schnabel, J. Ruggles, 2005, Diabetes care.

Intermittent high glucose enhances ICAM-1, VCAM-1 and E-selectin expression in human umbilical vein endothelial cells in culture: the distinct role of protein kinase C and mitochondrial superoxide production.

A. Ceriello, L. Piconi, L. Quagliaro, 2005, Atherosclerosis.

Intermittent high glucose enhances ICAM‐1, VCAM‐1, E‐selectin and interleukin‐6 expression in human umbilical endothelial cells in culture: the role of poly(ADP‐ribose) polymerase

D. Giugliano, K. Esposito, C. Szabó, 2004, Journal of thrombosis and haemostasis : JTH.

Effect of irbesartan on nitrotyrosine generation in non-hypertensive diabetic patients

D. Giugliano, K. Esposito, A. Ceriello, 2004, Diabetologia.

Oxidative Stress, Diabetes, and Its Complications

A. Ceriello, L. Piconi, 2007 .

Reactive oxygen species mediate a cellular ‘memory’ of high glucose stress signalling

C. Szabó, M. Ihnat, A. Ceriello, 2007, Diabetologia.

The protective effect of rosuvastatin in human umbilical endothelial cells exposed to constant or intermittent high glucose.

A. Ceriello, L. Piconi, R. Da Ros, 2008, Journal of diabetes and its complications.

Primary role of superoxide anion generation in the cascade of events leading to endothelial dysfunction and damage in high glucose treated HUVEC.

C. Szabó, A. Ceriello, L. Piconi, 2007, Nutrition, metabolism, and cardiovascular diseases : NMCD.

Evaluation of gliclazide ability to attenuate the hyperglycaemic ‘memory’ induced by high glucose in isolated human endothelial cells

M. Ihnat, A. Ceriello, L. Piconi, 2008, Diabetes/metabolism research and reviews.

Effect of postprandial hypertriglyceridemia and hyperglycemia on circulating adhesion molecules and oxidative stress generation and the possible role of simvastatin treatment.

D. Giugliano, K. Esposito, A. Ceriello, 2004, Diabetes.

Evidence for an Independent and Cumulative Effect of Postprandial Hypertriglyceridemia and Hyperglycemia on Endothelial Dysfunction and Oxidative Stress Generation: Effects of Short- and Long-Term Simvastatin Treatment

A. Ceriello, L. Piconi, L. Quagliaro, 2002, Circulation.

Intermittent high glucose enhances apoptosis related to oxidative stress in human umbilical vein endothelial cells: the role of protein kinase C and NAD(P)H-oxidase activation.

Antonio Ceriello, A. Ceriello, L. Piconi, 2003, Diabetes.

Effects of S21403 (mitiglinide) on postprandial generation of oxidative stress and inflammation in type 2 diabetic patients

A. Ceriello, L. Piconi, L. Quagliaro, 2005, Diabetologia.

The post‐prandial state in Type 2 diabetes and endothelial dysfunction: effects of insulin aspart

A. Cavarape, A. Ceriello, L. Piconi, 2004, Diabetic medicine : a journal of the British Diabetic Association.

Constant and intermittent high glucose enhances endothelial cell apoptosis through mitochondrial superoxide overproduction

A. Ceriello, L. Piconi, L. Quagliaro, 2006, Diabetes/metabolism research and reviews.

Oxidative Stress in Diabetes

Antonio Ceriello, A. Ceriello, L. Piconi, 2003, Clinical chemistry and laboratory medicine.

Glucose "peak" and glucose "spike": Impact on endothelial function and oxidative stress.

R. Testa, A. Bonfigli, D. Giugliano, 2008, Diabetes research and clinical practice.

Effect of Atorvastatin and Irbesartan, Alone and in Combination, on Postprandial Endothelial Dysfunction, Oxidative Stress, and Inflammation in Type 2 Diabetic Patients