P. Benatti

发表

NF-YC Complexity Is Generated by Dual Promoters and Alternative Splicing

R. Mantovani, C. Imbriano, M. Ceribelli, 2009, The Journal of Biological Chemistry.

NF-Y activates genes of metabolic pathways altered in cancer cells

N. Gnesutta, R. Mantovani, C. Imbriano, 2015, Oncotarget.

NF-Y coassociates with FOS at promoters, enhancers, repetitive elements, and inactive chromatin regions, and is stereo-positioned with growth-controlling transcription factors

Kevin Struhl, Giulio Pavesi, Carol Imbriano, 2013, Genome research.

Specific inhibition of NF-Y subunits triggers different cell proliferation defects

A. Weisz, C. Imbriano, M. Ravo, 2011, Nucleic acids research.

A balance between NF-Y and p53 governs the pro- and anti-apoptotic transcriptional response

Daniele Merico, Carol Imbriano, Enrico Tagliafico, 2008, Nucleic acids research.

Direct non transcriptional role of NF-Y in DNA replication.

B. Miotto, R. Mantovani, V. Basile, 2016, Biochimica et biophysica acta.

NF-Y loss triggers p53 stabilization and apoptosis in HPV18-positive cells by affecting E6 transcription

V. Basile, C. Imbriano, P. Benatti, 2016, Oncotarget.

NF-YA splice variants have different roles on muscle differentiation.

R. Mantovani, V. Basile, C. Imbriano, 2016, Biochimica et biophysica acta.

Concurrent inhibition of enzymatic activity and NF-Y-mediated transcription of Topoisomerase-IIα by bis-DemethoxyCurcumin in cancer cells

V. Basile, C. Imbriano, P. Benatti, 2013, Cell Death and Disease.