H. Viberg

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Paracetamol (acetaminophen) administration during neonatal brain development affects cognitive function and alters its analgesic and anxiolytic response in adult male mice.

T. Gordh, H. Viberg, P. Eriksson, 2014, Toxicological sciences : an official journal of the Society of Toxicology.

A brominated flame retardant, 2,2',4,4',5-pentabromodiphenyl ether: uptake, retention, and induction of neurobehavioral alterations in mice during a critical phase of neonatal brain development.

E. Jakobsson, H. Viberg, P. Eriksson, 2002, Toxicological sciences : an official journal of the Society of Toxicology.

Polybrominated diphenylethers (PBDEs): A novel calss of developmental neurotoxicants in our environment.

E. Jakobsson, H. Viberg, P. Eriksson, 2001 .

Perfluorooctane sulfonate and perfluorooctanoic acid

H. Viberg, P. Eriksson, 2017 .

Perfluorooctane sulfonate (PFOS) and perfluorooctanoic acid (PFOA)

H. Viberg, P. Eriksson, 2011 .

Differences in neonatal neurotoxicity of brominated flame retardants, PBDE 99 and TBBPA, in mice.

H. Viberg, P. Eriksson, 2011, Toxicology.

Changes in spontaneous behaviour and altered response to nicotine in the adult rat, after neonatal exposure to the brominated flame retardant, decabrominated diphenyl ether (PBDE 209).

H. Viberg, P. Eriksson, A. Fredriksson, 2007, Neurotoxicology.

Neontal PBDE 99 exposure causes dose-response related behavioural derangements that are not sex or strain specific in mice

H. Viberg, P. Eriksson, A. Fredriksson, 2003 .

Postnatal exposure to PFOS, but not PBDE 99, disturb dopaminergic gene transcription in the mouse CNS.

H. Viberg, S. Hallgren, 2016, Environmental toxicology and pharmacology.

Developmental exposure to PBDE 209 : sex, neuroprotein and neurobehavioural analyses

H. Viberg, P. Eriksson, A. Fredriksson, 2012 .

Neonatal exposure to propofol affects BDNF but not CaMKII, GAP-43, synaptophysin and tau in the neonatal brain and causes an altered behavioural response to diazepam in the adult mouse brain

T. Gordh, H. Viberg, P. Eriksson, 2011, Behavioural Brain Research.

Developmental toxicology in the neonatal mouse: The use of randomly selected individuals as statistical unit compared to the litter in mice neonatally exposed to PBDE 99

D. Rosen, H. Viberg, P. Eriksson, 2004 .

Clonidine abolishes the adverse effects on apoptosis and behaviour after neonatal ketamine exposure in mice

T. Gordh, H. Viberg, P. Eriksson, 2012, Acta anaesthesiologica Scandinavica.

Proteomic Evaluation of Neonatal Exposure to 2,2′,4,4′,5-Pentabromodiphenyl Ether

Henrik Alm, Kim Kultima, Lennart Dencker, 2005, Environmental health perspectives.

Effects of neonatal exposure to the flame retardant tetrabromobisphenol-A, aluminum diethylphosphinate or zinc stannate on long-term potentiation and synaptic protein levels in mice

G. Ramakers, M. Dingemans, H. Viberg, 2015, Archives of Toxicology.

Neurobehavioral derangements in adult mice receiving decabrominated diphenyl ether (PBDE 209) during a defined period of neonatal brain development.

Eva Jakobsson, Anders Fredriksson, E. Jakobsson, 2003, Toxicological sciences : an official journal of the Society of Toxicology.

Neonatal exposure to PFOS and PFOA in mice results in changes in proteins which are important for neuronal growth and synaptogenesis in the developing brain.

H. Viberg, P. Eriksson, N. Johansson, 2009, Toxicological sciences : an official journal of the Society of Toxicology.

Neonatal ketamine exposure results in changes in biochemical substrates of neuronal growth and synaptogenesis, and alters adult behavior irreversibly.

T. Gordh, H. Viberg, P. Eriksson, 2008, Toxicology.

Neonatal exposure to decabrominated diphenyl ether (PBDE 209) results in changes in BDNF, CaMKII and GAP-43, biochemical substrates of neuronal survival, growth, and synaptogenesis.

H. Viberg, P. Eriksson, W. Mundy, 2008, Neurotoxicology.

Neonatal exposure to the brominated flame-retardant, 2,2',4,4',5-pentabromodiphenyl ether, decreases cholinergic nicotinic receptors in hippocampus and affects spontaneous behaviour in the adult mouse.

Anders Fredriksson, H. Viberg, P. Eriksson, 2004, Environmental toxicology and pharmacology.

Neonatal exposure to polybrominated diphenyl ether (PBDE 153) disrupts spontaneous behaviour, impairs learning and memory, and decreases hippocampal cholinergic receptors in adult mice.

Anders Fredriksson, H. Viberg, P. Eriksson, 2003, Toxicology and applied pharmacology.

Neonatal ontogeny and neurotoxic effect of decabrominated diphenyl ether (PBDE 209) on levels of synaptophysin and tau

H. Viberg, 2009, International Journal of Developmental Neuroscience.

The developing cholinergic system as target for environmental toxicants, nicotine and polychlorinated biphenyls (PCBs): Implications for neurotoxicological processes in mice

H. Viberg, P. Eriksson, A. Fredriksson, 2009, Neurotoxicity Research.

Investigations of strain and/or gender differences in developmental neurotoxic effects of polybrominated diphenyl ethers in mice.

Anders Fredriksson, H. Viberg, P. Eriksson, 2004, Toxicological sciences : an official journal of the Society of Toxicology.

Developmental neurotoxic effects of two pesticides: Behavior and neuroprotein studies on endosulfan and cypermethrin.

Anders Fredriksson, H. Viberg, P. Eriksson, 2015, Toxicology.

Etomidate exposure in early infant mice (P10) does not induce apoptosis or affect behaviour

H. Viberg, A. Fredriksson, P. Lönnqvist, 2016, Acta anaesthesiologica Scandinavica.

Adult neurobehavioral alterations in male and female mice following developmental exposure to paracetamol (acetaminophen): characterization of a critical period

T. Gordh, H. Viberg, A. Fredriksson, 2017, Journal of applied toxicology : JAT.

Adult dose-dependent behavioral and cognitive disturbances after a single neonatal PFHxS dose.

H. Viberg, P. Eriksson, Iwa Lee, 2013, Toxicology.

A Single δ9-Tetrahydrocannabinol (THC) Dose During Brain Development Affects Markers of Neurotrophy, Oxidative Stress, and Apoptosis

E. Forsberg, R. Fredriksson, H. Viberg, 2019, Front. Pharmacol..

Short-term exposure and long-term consequences of neonatal exposure to Δ9-tetrahydrocannabinol (THC) and ibuprofen in mice

T. Gordh, H. Viberg, A. Fredriksson, 2016, Behavioural Brain Research.

A single neonatal exposure to perfluorohexane sulfonate (PFHxS) affects the levels of important neuroproteins in the developing mouse brain.

H. Viberg, Iwa Lee, 2013, Neurotoxicology.

Neonatal exposure to deca-brominated diphenyl ether (PBDE 209) causes dose-response changes in spontaneous behaviour and cholinergic susceptibility in adult mice.

H. Viberg, P. Eriksson, A. Fredriksson, 2008, Neurotoxicology.

Neonatal Developmental Neurotoxicity of Brominated Flame Retardants, the Polybrominated Diphenyl Ethers (PBDEs)

H. Viberg, 2004 .

Dose-response modeling and benchmark calculations from spontaneous behavior data on mice neonatally exposed to 2,2',4,4',5-pentabromodiphenyl ether.

Salomon Sand, Anders Fredriksson, Katarina Victorin, 2004, Toxicological sciences : an official journal of the Society of Toxicology.

Neonatal exposure to a moderate dose of ionizing radiation causes behavioural defects and altered levels of tau protein in mice.

B. Stenerlöw, H. Viberg, P. Eriksson, 2014, Neurotoxicology.

Developmental neurotoxic effects of two pesticides: Behavior and biomolecular studies on chlorpyrifos and carbaryl.

H. Viberg, P. Eriksson, A. Fredriksson, 2015, Toxicology and applied pharmacology.

Developmental exposure to the polybrominated diphenyl ether PBDE 209: Neurobehavioural and neuroprotein analysis in adult male and female mice.

H. Viberg, P. Eriksson, A. Fredriksson, 2014, Environmental toxicology and pharmacology.

Neonatal exposure to higher brominated diphenyl ethers, hepta-, octa-, or nonabromodiphenyl ether, impairs spontaneous behavior and learning and memory functions of adult mice.

Anders Fredriksson, G. Marsh, H. Viberg, 2006, Toxicological sciences : an official journal of the Society of Toxicology.

A Cannabinoid Receptor Type 1 (CB1R) Agonist Enhances the Developmental Neurotoxicity of Acetaminophen (Paracetamol)

T. Gordh, R. Fredriksson, H. Viberg, 2018, Toxicological sciences : an official journal of the Society of Toxicology.

More signs of neurotoxicity of surfactants and flame retardants - Neonatal PFOS and PBDE 99 cause transcriptional alterations in cholinergic genes in the mouse CNS.

H. Viberg, A. Fredriksson, S. Hallgren, 2015, Environmental toxicology and pharmacology.

Dose-dependent behavioral disturbances after a single neonatal Bisphenol A dose.

H. Viberg, P. Eriksson, A. Fredriksson, 2011, Toxicology.

Effects on adult cognitive function after neonatal exposure to clinically relevant doses of ionising radiation and ketamine in mice

B. Stenerlöw, T. Gordh, H. Viberg, 2018, British journal of anaesthesia.

Neonatal exposure to the brominated flame retardant 2,2',4,4',5-pentabromodiphenyl ether causes altered susceptibility in the cholinergic transmitter system in the adult mouse.

Anders Fredriksson, H. Viberg, P. Eriksson, 2002, Toxicological sciences : an official journal of the Society of Toxicology.

A single exposure to bisphenol A alters the levels of important neuroproteins in adult male and female mice.

H. Viberg, Iwa Lee, 2012, Neurotoxicology.

Deranged spontaneous behaviour and decrease in cholinergic muscarinic receptors in hippocampus in the adult rat, after neonatal exposure to the brominated flame-retardant, 2,2',4,4',5-pentabromodiphenyl ether (PBDE 99).

H. Viberg, P. Eriksson, A. Fredriksson, 2005, Environmental toxicology and pharmacology.

PBDE, 2,2',4,4'5-pentabromodiphenyl ether causes permanent neurotoxic effects during a defined period of neonatal brain development

Eva Jakobsson, Anders Fredriksson, E. Jakobsson, 1999 .

Neonatal exposure to sucralose does not alter biochemical markers of neuronal development or adult behavior.

H. Viberg, A. Fredriksson, 2011, Nutrition.

Exposure to polybrominated diphenyl ethers 203 and 206 during the neonatal brain growth spurt affects proteins important for normal neurodevelopment in mice.

H. Viberg, 2009, Toxicological sciences : an official journal of the Society of Toxicology.

Response to the comment on Viberg et al. (2008) “Neonatal ketamine exposure results in changes in biochemical substrates of neuronal growth and synaptogenesis, and alters adult behavior irreversibly” by Ching-Hung Hsu

H. Viberg, 2008 .

Neonatal ontogeny and neurotoxic effect of decabrominated diphenyl ether (PBDE 209) on levels of synaptophysin and tau

H. Viberg, 2009, International Journal of Developmental Neuroscience.

Evaluation of the dentate gyrus in adult mice exposed to acetaminophen (paracetamol) on postnatal day 10

R. Fredriksson, S. Hellsten, H. Viberg, 2020, International journal of developmental neuroscience : the official journal of the International Society for Developmental Neuroscience.

Clonidine abolishes the adverse effects on apoptosis and behaviour after neonatal ketamine exposure in mice

H. Viberg, A. Fredriksson, E. Pontén, 2012, Acta anaesthesiologica Scandinavica.

Neonatal ketamine exposure results in changes in biochemical substrates of neuronal growth and synaptogenesis, and alters adult behavior irreversibly.

T. Gordh, H. Viberg, P. Eriksson, 2008, Toxicology.