D. Hajjar

发表

Anti‐adipogenic action of pitavastatin occurs through the coordinate regulation of PPARγ and Pref‐1 expression

A. Gotto, A. Gotto, Jihong Han, 2007 .

Statins synergize dexamethasone-induced adipocyte fatty acid binding protein expression in macrophages.

Yan Zhu, Jihong Han, Yuanli Chen, 2012, Atherosclerosis.

Adipogenic differentiating agents regulate expression of fatty acid binding protein and CD36 in the J744 macrophage cell line Published, JLR Papers in Press, July 16, 2003. DOI 10.1194/jlr.M300084-JLR200

A. Gotto, Jihong Han, A. C. Nicholson, 2003, Journal of Lipid Research.

Functional Interplay Between the Macrophage Scavenger Receptor Class B Type I and Pitavastatin (NK-104)

A. Gotto, Jihong Han, A. C. Nicholson, 2004, Circulation.

Triglyceride, high density lipoprotein, and coronary heart disease.

W. A. Bradley, D. Gordon, G. Hutchins, 1993, Consensus statement. National Institutes of Health Consensus Development Conference.

Role of Injury in Arterial Lipid Metabolism

D. Hajjar, C. Minick, D. Falcone, 1982 .

Lipoprotein and albumin accumulation in reendothelialized and deendothelialized aorta.

D. Hajjar, C. Minick, D. Falcone, 1984, The American journal of pathology.

Enhancement of cholesterol and cholesteryl ester accumulation in re-endothelialized aorta.

D. Hajjar, C. Minick, D. Falcone, 1980, The American journal of pathology.

Recovery of prostacyclin production by de-endothelialized rabbit aorta. Critical role of neointimal smooth muscle cells.

B. Weksler, D. Hajjar, C. Minick, 1981, The Journal of clinical investigation.

Diet-induced hypercholesterolemia inhibits the recovery of prostacyclin production by injured rabbit aorta.

B. Weksler, D. Hajjar, C. Minick, 1982, The American journal of pathology.

Repair Mechanisms in Oxidant-Driven Chronic Inflammatory Disease.

D. Hajjar, R. Deeb, 2016, The American journal of pathology.

Herpesvirus in atherosclerosis and thrombosis: etiologic agents or ubiquitous bystanders?

A. C. Nicholson, D. Hajjar, 1998, Arteriosclerosis, thrombosis, and vascular biology.

Inhibition of ERK1/2 and Activation of Liver X Receptor Synergistically Induce Macrophage ABCA1 Expression and Cholesterol Efflux*

Jihong Han, D. Hajjar, Xiaoye Zhou, 2009, The Journal of Biological Chemistry.

Signal transduction in atherosclerosis: second messengers and regulation of cellular cholesterol trafficking.

A. C. Nicholson, D. Hajjar, K. Pomerantz, 1995, Advances in experimental medicine and biology.

Fibroblast growth factor receptor is a portal of cellular entry for herpes simplex virus type 1.

R. Kaner, D. Hajjar, R. Florkiewicz, 1990, Science.

NITRIC OXIDE ENHANCES PROSTAGLANDIN-H SYNTHASE-1 ACTIVITY BY A HEME-INDEPENDENT MECHANISM : EVIDENCE IMPLICATING NITROSOTHIOLS

S. F. Pearce, D. Hajjar, H. Lander, 1995 .

Rosiglitazone alleviates intrahepatic cholestasis induced by α‐naphthylisothiocyanate in mice: The role of circulating 15‐deoxy‐Δ12,14‐PGJ2 and Nogo

Yan Zhu, Jihong Han, Xiaoxiao Yang, 2020, British Journal of Pharmacology.

Evidence for cytokine regulation of cholesterol metabolism in herpesvirus-infected arterial cells by the lipoxygenase pathway.

D. Hajjar, O. Etingin, 1990, Journal of lipid research.

25-Hydroxycholesterol activates the expression of cholesterol 25-hydroxylase in an LXR-dependent mechanism[S]

Y. Liu, Jihong Han, Xiaoxiao Yang, 2018, Journal of Lipid Research.

Genetic Deletion of Low Density Lipoprotein Receptor Impairs Sterol-induced Mouse Macrophage ABCA1 Expression

A. Gotto, Jihong Han, D. Hajjar, 2008, Journal of Biological Chemistry.

High-density-lipoprotein-induced cholesterol efflux from arterial smooth muscle cell derived foam cells: functional relationship of the cholesteryl ester cycle and eicosanoid biosynthesis.

D. Hajjar, K. Pomerantz, 1990, Biochemistry.

Erratum: Macrophage scavenger receptor CD36 is the major receptor for LDL modified by monocyte-generated reactive nitrogen species (Journal of Clinical Investigation (2000) 105 (1095-1108))

S. Hazen, E. Podrez, N. Sheibani, 2000 .

Viral activation of the coagulation cascade: Molecular interactions at the surface of infected endothelial cells

H. Friedman, R. Silverstein, D. Hajjar, 1990, Cell.

Physical evidence for substrate binding in preventing cyclooxygenase inactivation under nitrative stress.

C. Cheung, D. Hajjar, S. Gross, 2010, Journal of the American Chemical Society.

Reprint of "oxidative alterations of cyclooxygenase during atherogenesis" [Prostag. Oth. Lipid. M. 80 (2006) 1-14].

D. Hajjar, R. Deeb, R. Upmacis, 2007, Prostaglandins & other lipid mediators.

Oxidative alterations of cyclooxygenase during atherogenesis.

D. Hajjar, R. Deeb, R. Upmacis, 2006, Prostaglandins & other lipid mediators.

Identification of interferon-γ as a new molecular target of liver X receptor.

Jihong Han, Yuanli Chen, Yajun Duan, 2014, The Biochemical journal.

Activation of Liver X Receptor Induces Macrophage Interleukin-5 Expression*

Jihong Han, Xiaoxiao Yang, Yuanli Chen, 2012, The Journal of Biological Chemistry.

Infection and atherosclerosis: potential roles of pathogen burden and molecular mimicry.

M. Burnett, S. Epstein, G. Vercellotti, 2000, Arteriosclerosis, thrombosis, and vascular biology.

Interactions of arterial cells: III. Stathmokinetic analyses of smooth muscle cells cocultured with endothelial cells

M. Kimmel, K. Hajjar, L. Staiano‐Coico, 1988, Journal of cellular physiology.

Transforming Growth Factor-β1 (TGF-β1) and TGF-β2 Decrease Expression of CD36, the Type B Scavenger Receptor, through Mitogen-activated Protein Kinase Phosphorylation of Peroxisome Proliferator-activated Receptor-γ*

A. Gotto, Jihong Han, A. C. Nicholson, 2000, The Journal of Biological Chemistry.

Eicosanoids in regulation of arterial smooth muscle cell phenotype, proliferative capacity, and cholesterol metabolism.

D. Hajjar, K. Pomerantz, 1989, Arteriosclerosis.

Pitavastatin Differentially Modulates MicroRNA-Associated Cholesterol Transport Proteins in Macrophages

T. Sun, A. Gotto, Haijun Zhang, 2016, PloS one.

Transforming growth factor-beta1 (TGF-beta1) and TGF-beta2 decrease expression of CD36, the type B scavenger receptor, through mitogen-activated protein kinase phosphorylation of peroxisome proliferator-activated receptor-gamma.

A. Gotto, J. Han, A. C. Nicholson, 2000, The Journal of biological chemistry.

Inducible nitric oxide synthase provides protection against injury-induced thrombosis in female mice.

K. Hajjar, D. Hajjar, R. Deeb, 2011, American journal of physiology. Heart and circulatory physiology.

CD36, oxidized LDL and PPARγ: pathological interactions in macrophages and atherosclerosis

A. C. Nicholson, D. Hajjar, 2004 .

Oxidized Low Density Lipoprotein Decreases Macrophage Expression of Scavenger Receptor B-I*

A. Gotto, Jihong Han, A. C. Nicholson, 2001, The Journal of Biological Chemistry.

CD36, the Macrophage Class B Scavenger Receptor: Regulation and Role in Atherosclerosis

A. Gotto, Jihong Han, M. Febbraio, 2001 .

Role of CD36, the Macrophage Class B Scavenger Receptor, in Atherosclerosis

Jihong Han, M. Febbraio, A. C. Nicholson, 2001, Annals of the New York Academy of Sciences.

CD36 in Atherosclerosis: The Role of a Class B Macrophage Scavenger Receptor

Jihong Han, R. Silverstein, M. Febbraio, 2000 .

Cellular cholesterol regulates expression of the macrophage type B scavenger receptor, CD36.

Jihong Han, J. Han, A. C. Nicholson, 1999, Journal of lipid research.

Recombinant Glutathione S-Transferase/CD36 Fusion Proteins Define an Oxidized Low Density Lipoprotein-binding Domain*

R. Silverstein, M. Febbraio, A. C. Nicholson, 1998, The Journal of Biological Chemistry.

Native and Modified Low Density Lipoproteins Increase the Functional Expression of the Macrophage Class B Scavenger Receptor, CD36*

Jihong Han, M. Febbraio, A. C. Nicholson, 1997, The Journal of Biological Chemistry.

Cytokine regulation of low density lipoprotein receptor gene transcription in HepG2 cells.

A. Stopeck, A. C. Nicholson, D. Hajjar, 1993, Journal of Biological Chemistry.

Offensives Against Vascular Injury Maintaining Equilibrium by Selective Targeting of Cyclooxygenase Pathways : Promising

D. Hajjar, S. Gross, R. Deeb, 2013 .

Inducible nitric oxide synthase mediates prostaglandin h2 synthase nitration and suppresses eicosanoid production.

N. Maeda, D. Hajjar, S. Gross, 2006, The American journal of pathology.

Cholesterol enrichment enhances expression of sterol-carrier protein-2: implications for its function in intracellular cholesterol trafficking.

D. Hajjar, S. Kesav, R. Kraemer, 1995, Journal of lipid research.

Viral Activation of Coagulation: Implications for Thrombosis and Atherosclerosis

A. C. Nicholson, D. Hajjar, 1997, Annals of the New York Academy of Sciences.

Tamoxifen inhibits macrophage FABP4 expression through the combined effects of the GR and PPARγ pathways.

Yan Zhu, Jihong Han, Yuanli Chen, 2013, The Biochemical journal.

Peroxisome Proliferator-activated Receptor γ Activation by Ligands and Dephosphorylation Induces Proprotein Convertase Subtilisin Kexin Type 9 and Low Density Lipoprotein Receptor Expression*

Jihong Han, Xiaoxiao Yang, Yuanli Chen, 2012, The Journal of Biological Chemistry.

Regulation of CD36 by PPARγ: Pro- or Antiatherogenic?

S. Hazen, E. Podrez, A. Gotto, 2002 .

Oxygen tension within the arterial wall: relationship to altered bioenergetic metabolism and lipid accumulation.

D. Hajjar, I. Farber, S. C. Smith, 1988, Archives of biochemistry and biophysics.

Role of eicosanoids and the cytokine network in transmembrane signaling in vascular cells.

D. Hajjar, K. Pomerantz, 1991, Advances in experimental medicine and biology.

Cholesterol metabolism is altered by hydrolytic metabolites of prostacyclin in arterial smooth muscle cells.

B. Weksler, D. Hajjar, O. Etingin, 1986, Journal of lipid research.

Prostacyclin modulates cholesteryl ester hydrolytic activity by its effect on cyclic adenosine monophosphate in rabbit aortic smooth muscle cells.

B. Weksler, D. Hajjar, C. Minick, 1982, The Journal of clinical investigation.

Endothelium modifies the altered metabolism of the injured aortic wall.

S. Fowler, D. Hajjar, C. Minick, 1981, The American journal of pathology.

Atherosclerosis in LDLR-knockout mice is inhibited, but not reversed, by the PPARgamma ligand pioglitazone.

H. Nakaya, A. Gotto, Jihong Han, 2009, The American journal of pathology.

Correlation of Morphological and Biochemical Components of Atherosclerotic Plaques

W. Insull, D. Hajjar, C. Minick, 1983 .

Activation of peroxisome proliferator-activated receptor-α in mice induces expression of the hepatic low-density lipoprotein receptor

Z. Huang, A. Gotto, Jihong Han, 2008, British journal of pharmacology.

Dihydropyridine calcium antagonist modulates cholesterol metabolism and eicosanoid biosynthesis in vascular cells

A. C. Nicholson, D. Hajjar, A. Wolff, 1992, Journal of cellular biochemistry.

Herpes simplex virus infection in human arterial cells. Implications in arteriosclerosis.

B. Weksler, D. Hajjar, D. Falcone, 1987, The Journal of clinical investigation.

Virus-induced atherosclerosis. Herpesvirus infection alters aortic cholesterol metabolism and accumulation.

D. Hajjar, C. Minick, J. Fabricant, 1986, The American journal of pathology.

von Willebrand factor mediates platelet adhesion to virally infected endothelial cells.

R. Silverstein, D. Hajjar, O. Etingin, 1993, Proceedings of the National Academy of Sciences of the United States of America.

Inhibition of ERK1/2 and Activation of LXR Synergistically Reduce Atherosclerotic Lesions in ApoE-Deficient Mice

Jihong Han, Xiaoxiao Yang, Yuanli Chen, 2015, Arteriosclerosis, thrombosis, and vascular biology.

Tumor necrosis factor-mediated release of platelet-derived growth factor from cultured endothelial cells

K. Hajjar, R. Silverstein, D. Hajjar, 1987, The Journal of experimental medicine.

Arterial cell interactions: mechanistic studies related to eicosanoid and growth factor-induced alterations in cholesterol metabolism.

A. Marcus, K. Hajjar, D. Hajjar, 1988, Advances in experimental medicine and biology.

Cytokine Regulation o Arterial Cholesterol Trafficking

D. Hajjar, K. Pomerantz, 1996 .

Inhibition of cholesterol esterification in macrophages and vascular smooth muscle foam cells: Evaluation of E5324, an Acyl-CoA cholesterol acyltransferase inhibitor

A. C. Nicholson, D. Hajjar, T. Fujimori, 1995, Lipids.

Eicosanoid metabolism in cholesterol-enriched arterial smooth muscle cells: reduced arachidonate release with concomitant decrease in cyclooxygenase products.

D. Hajjar, K. Pomerantz, 1989, Journal of lipid research.

Protein 3-nitrotyrosine in complex biological samples: quantification by high-pressure liquid chromatography/electrochemical detection and emergence of proteomic approaches for unbiased identification of modification sites.

D. Hajjar, S. Gross, R. Deeb, 2008, Methods in enzymology.

Viral activation of the coagulation cascade.

A. C. Nicholson, D. Hajjar, 1999, American heart journal.

Pitavastatin suppresses mitogen activated protein kinase-mediated Erg-1 induction in human vascular smooth muscle cells.

A. Gotto, D. Hajjar, B. Summers, 2009, European journal of pharmacology.

Lipid accumulation and ultrastructural change within the aortic wall during early spontaneous atherogenesis.

D. Hajjar, T. Wight, S. C. Smith, 1980, The American journal of pathology.

Interaction of arterial cells. I. Endothelial cells alter cholesterol metabolism in co-cultured smooth muscle cells.

D. Hajjar, D. Falcone, J. Amberson, 1985, Journal of lipid research.

Silent Partner in Blood Vessel Homeostasis? Pervasive Role of Nitric Oxide in Vascular Disease.

D. Hajjar, R. Deeb, B. Lamon, 2009, Current hypertension reviews.

BIOENERGETIC METABOLISM AND LIPID ACCUMULATION IN PIGEON ATHEROGENESIS

D. Hajjar, 1978 .

Regulation of cholesteryl ester hydrolases.

D. Hajjar, 1994, Advances in enzymology and related areas of molecular biology.

Molecular motions and thermotropic phase behavior of triacylglycerols and cholesteryl esters in herpesvirus-infected arterial smooth muscle cells: a deuterium nuclear magnetic resonance study.

F. Picart, D. Hajjar, K. Pomerantz, 1992, Biophysical chemistry.

Prostaglandins and cyclic nucleotides. Modulators of arterial cholesterol metabolism.

D. Hajjar, 1985, Biochemical pharmacology.

Metabolic activity of cholesteryl esters in aortic smooth muscle cells is altered by prostaglandins I2 and E2.

B. Weksler, D. Hajjar, 1983, Journal of lipid research.

Oxidized lipoproteins and infectious agents: are they in collusion to accelerate atherogenesis?

D. Hajjar, 2000, Arteriosclerosis, thrombosis, and vascular biology.

Recruitment and activation of Raf-1 kinase by nitric oxide-activated Ras.

D. Hajjar, H. Lander, A. Deora, 2000, Biochemistry.

Regulation of neutral cholesteryl esterase in arterial smooth muscle cells: stimulation by agonists of adenylate cyclase and cyclic AMP-dependent protein kinase.

D. Hajjar, 1986, Archives of biochemistry and biophysics.

Biochemical Mechanisms Associated with the Lipolytic Effects of Calcium Channel Blockers

A. C. Nicholson, D. Hajjar, B. Summers, 1993 .

Calcium channel blockers enhance cholesteryl ester hydrolysis and decrease total cholesterol accumulation in human aortic tissue.

D. Hajjar, O. Etingin, 1990, Circulation research.

Nifedipine increases cholesteryl ester hydrolytic activity in lipid-laden rabbit arterial smooth muscle cells. A possible mechanism for its antiatherogenic effect.

D. Hajjar, O. Etingin, 1985, The Journal of clinical investigation.

Herpesvirus infection enhances cholesterol and cholesteryl ester accumulation in cultured arterial smooth muscle cells.

D. Hajjar, C. Minick, J. Fabricant, 1981, The American journal of pathology.

Lipoprotein (a) regulates plasminogen activator inhibitor-1 expression in endothelial cells. A potential mechanism in thrombogenesis.

K. Hajjar, D. Hajjar, R. Nachman, 1991, The Journal of biological chemistry.

Regulation of prostaglandin H2 synthase activity by nitrogen oxides.

D. Hajjar, R. Deeb, R. Upmacis, 1999, Biochemistry.

Mediation of virion penetration into vascular cells by association of basic fibroblast growth factor with herpes simplex virus type 1

P. Maher, R. Kaner, D. Hajjar, 1990, Nature.

CD36, oxidized LDL and PPAR gamma: pathological interactions in macrophages and atherosclerosis.

A. C. Nicholson, D. Hajjar, 2004, Vascular pharmacology.

Arterial Endothelial Cell‐derived Eicosanoids Alter Cholesterol Metabolism in Arterial Smooth Muscle Cells

A. Marcus, K. Hajjar, D. Hajjar, 1988 .

Diet-Induced Hyperc holesterolemia Inhibits the Recovery of Prostacyclin Production by Injured Ra bbit Aorta

B. Weksler, D. Hajjar, C. Minick, 2007 .

Endothelium in Experimental Atherosclerosis

D. Hajjar, C. Minick, D. Falcone, 1980 .

Isolation of Total RNA , Purification of Poly ( A ) RNA , and Northern Blotting

A. Gotto, Jihong Han, A. C. Nicholson, 2004 .

Beta-VLDL increases endothelial cell plasma membrane cholesterol.

J. Berliner, D. Hajjar, J. Kim, 1991, Journal of lipid research.

Prostaglandins modulate arterial cholesteryl ester metabolism.

D. Hajjar, 1984, Enzyme.

Identification of a monocyte receptor on herpesvirus-infected endothelial cells.

R. Silverstein, D. Hajjar, O. Etingin, 1991, Proceedings of the National Academy of Sciences of the United States of America.

Leukotriene production by human glia.

J. Ghajar, D. Hajjar, K. Pomerantz, 1990, Acta neurochirurgica. Supplementum.

Focal differences in bioenergetic metabolism of atherosclerosis-susceptible and -resistant pigeon aortas.

D. Hajjar, S. C. Smith, 1980, Atherosclerosis.

Transforming growth factor-beta up-regulates low density lipoprotein receptor-mediated cholesterol metabolism in vascular smooth muscle cells.

A. C. Nicholson, D. Hajjar, 1992, The Journal of biological chemistry.

Cholesterol enrichment of arterial smooth muscle cells upregulates cytokine-induced nitric oxide synthesis.

D. Hajjar, S. Gross, R. Levi, 1993, Biochemical and biophysical research communications.

Inducible nitric oxide synthase gene deletion exaggerates MAPK-mediated cyclooxygenase-2 induction by inflammatory stimuli.

D. Hajjar, R. Deeb, R. Upmacis, 2010, American journal of physiology. Heart and circulatory physiology.

Developmentally regulated herpesvirus plaque formation in arterial smooth muscle cells.

S. Schwartz, R. Kaner, A. C. Nicholson, 1993, Circulation research.

Signal transduction in atherosclerosis: integration of cytokines and the eicosanoid network

D. Hajjar, K. Pomerantz, 1992, FASEB journal : official publication of the Federation of American Societies for Experimental Biology.

Human glial cell production of lipoxygenase-generated eicosanoids: a potential role in the pathophysiology of vascular changes following traumatic brain injury

J. Ghajar, D. Hajjar, R. Patterson, 1988 .

G-protein-mediated signaling in cholesterol-enriched arterial smooth muscle cells. 1. Reduced membrane-associated G-protein content due to diminished isoprenylation of G-gamma subunits and p21ras.

D. Hajjar, H. Lander, B. Summers, 1997, Biochemistry.

Induction of basic fibroblast growth factor mRNA and protein synthesis in smooth muscle cells by cholesteryl ester enrichment and 25-hydroxycholesterol.

D. Hajjar, R. Kraemer, K. Pomerantz, 1993, The Journal of biological chemistry.

Platelet-neutrophil-smooth muscle cell interactions: lipoxygenase-derived mono- and dihydroxy acids activate cholesteryl ester hydrolysis by the cyclic AMP dependent protein kinase cascade.

A. Marcus, D. Hajjar, O. Etingin, 1989, Biochemistry.

Interactions between prostacyclin metabolism and cholesteryl ester metabolism in the vascular wall.

B. Weksler, D. Hajjar, C. Minick, 1983, Advances in prostaglandin, thromboxane, and leukotriene research.

Histidine-rich glycoprotein inhibits the antiproliferative effect of heparin on smooth muscle cells

D. Hajjar, R. Nachman, P. Harpel, 1987, The Journal of experimental medicine.

A Molecular Redox Switch on p21ras

B. Chait, B. Hempstead, L. Quilliam, 1997, The Journal of Biological Chemistry.

Redox regulation of cell signalling

B. Chait, B. Hempstead, L. Quilliam, 1996, Nature.

Involvement of the mitogen-activated protein kinase cascade in peroxynitrite-mediated arachidonic acid release in vascular smooth muscle cells.

D. Hajjar, M. Resnick, D. Mittar, 2004, American journal of physiology. Cell physiology.

CD36: a class B scavenger receptor involved in angiogenesis, atherosclerosis, inflammation, and lipid metabolism.

R. Silverstein, M. Febbraio, D. Hajjar, 2001, The Journal of clinical investigation.

Transplantation of cryopreserved cultured epidermal allografts.

L. Staiano‐Coico, D. Hajjar, M. Madden, 1996, The Journal of trauma.

Targeted disruption of the class B scavenger receptor CD36 protects against atherosclerotic lesion development in mice.

S. Hazen, E. Podrez, R. Silverstein, 2000, The Journal of clinical investigation.

A Null Mutation in Murine CD36 Reveals an Important Role in Fatty Acid and Lipoprotein Metabolism*

R. Silverstein, M. Febbraio, N. Abumrad, 1999, The Journal of Biological Chemistry.

Inhibition of Macrophage Scavenger Receptor Activity by Tumor Necrosis Factor- Is Transcriptionally and Post-transcriptionally Regulated (*)

A. C. Nicholson, D. Hajjar, H Y Hsu, 1996, The Journal of Biological Chemistry.

Inflammation in Atherosclerosis and Implications for Therapy

A. Gotto, R. Paoletti, D. Hajjar, 2004, Circulation.

Characterization of a cellular denitrase activity that reverses nitration of cyclooxygenase.

D. Hajjar, S. Gross, B. Summers, 2013, American journal of physiology. Heart and circulatory physiology.

Warner-Lambert/Parke-Davis Award Lecture. Viral pathogenesis of atherosclerosis. Impact of molecular mimicry and viral genes.

D. Hajjar, 1991, The American journal of pathology.

The Role of Phosphatidylinositol 3-Kinase in Vascular Endothelial Growth Factor Signaling*

G. Thakker, D. Hajjar, T. Rosengart, 1999, The Journal of Biological Chemistry.

Aging and arteriosclerosis. Cell cycle kinetics of young and old arterial smooth muscle cells.

D. Hajjar, D. Alonso, Daniel R. Alonso, 1988, The American journal of pathology.

Inflammatory Networks Linking Cardiovascular Disease, Diabetes, and Cancer

A. Gotto, K. Hajjar, D. Hajjar, 2017 .

Macrophage scavenger receptor CD36 is the major receptor for LDL modified by monocyte-generated reactive nitrogen species.

S. Hazen, E. Podrez, N. Sheibani, 2000, The Journal of clinical investigation.

CD 36 : a class B scavenger receptor involved in angiogenesis , atherosclerosis , inflammation , and lipid metabolism

R. Silverstein, M. Febbraio, D. Hajjar, 2001 .

Mammalian Target of Rapamycin: A Metabolic Rheostat for Regulating Adipose Tissue Function and Cardiovascular Health.

A. Gotto, M. Wipperman, D. Hajjar, 2019, The American journal of pathology.

Effector Cell Protease Receptor-1 Is a Vascular Receptor for Coagulation Factor Xa*

T. Edgington, W. Ruf, A. C. Nicholson, 1996, The Journal of Biological Chemistry.

Herpesviruses and thrombosis: activation of coagulation on the endothelium.

A. C. Nicholson, D. Hajjar, 1999, Clinica chimica acta; international journal of clinical chemistry.

Tobacco constituents are mitogenic for arterial smooth-muscle cells.

D. Hajjar, C. G. Becker, J. Hefton, 1985, The American journal of pathology.

Biological relevance of inflammation and oxidative stress in the pathogenesis of arterial diseases.

A. Gotto, D. Hajjar, 2013, The American journal of pathology.

Altered glycosaminoglycan metabolism in injured arterial wall.

D. Hajjar, C. Minick, B. Salisbury, 1985, Experimental and molecular pathology.

Induction of CD 36 expression by oxidized LDL and IL-4 by a common signaling pathway dependent on protein kinase C and PPAR-g

A. Gotto, Jihong Han, R. Silverstein, 2000 .

Maintaining equilibrium by selective targeting of cyclooxygenase pathways: promising offensives against vascular injury.

D. Hajjar, S. Gross, R. Deeb, 2008, Hypertension.

Lipoprotein Trafficking in Vascular Cells

D. Hajjar, M. Haberland, D P Hajjar, 1997, The Journal of Biological Chemistry.

Heme catalyzes tyrosine 385 nitration and inactivation of prostaglandin H2 synthase-1 by peroxynitrite Published, JLR Papers in Press, February 9, 2006.

D. Hajjar, S. Gross, M. Lainé, 2006, Journal of Lipid Research.

Ligand Binding to Macrophage Scavenger Receptor-A Induces Urokinase-type Plasminogen Activator Expression by a Protein Kinase-dependent Signaling Pathway*

D. Hajjar, D. Falcone, K. Khan, 1998, The Journal of Biological Chemistry.

Pitavastatin Downregulates Expression of the Macrophage Type B Scavenger Receptor, CD36

A. Gotto, Jihong Han, A. C. Nicholson, 2004, Circulation.

MEK1/2 inhibitors activate macrophage ABCG1 expression and reverse cholesterol transport-An anti-atherogenic function of ERK1/2 inhibition.

Jihong Han, Xiaoxiao Yang, Yuanli Chen, 2016, Biochimica et biophysica acta.

A Novel Family of Atherogenic Oxidized Phospholipids Promotes Macrophage Foam Cell Formation via the Scavenger Receptor CD36 and Is Enriched in Atherosclerotic Lesions*

S. Hazen, E. Podrez, Renliang Zhang, 2002, Journal of Biological Chemistry.

Alterations of Cholesterol Metabolism in Inflammation-Induced Atherogenesis.

K. Hajjar, D. Hajjar, 2016, Journal of enzymology and metabolism.

Regulation of peroxisome proliferator-activated receptor-gamma-mediated gene expression. A new mechanism of action for high density lipoprotein.

A. Gotto, Jihong Han, A. C. Nicholson, 2002, The Journal of biological chemistry.

A molecular redox switch on p21(ras). Structural basis for the nitric oxide-p21(ras) interaction.

B. Chait, B. Hempstead, L. Quilliam, 1997, The Journal of biological chemistry.

Inflammation at the molecular interface of atherogenesis: an anthropological journey.

D. Hajjar, Brian D Lamon, David P Hajjar, 2008, The American journal of pathology.

Arterial neutral cholesteryl esterase. A hormone-sensitive enzyme distinct from lysosomal cholesteryl esterase.

S. Fowler, D. Hajjar, C. Minick, 1983, The Journal of biological chemistry.

Vascular transcellular signaling.

A. Marcus, D. Hajjar, A. Marcus, 1993, Journal of lipid research.

GeneticDeletionofLowDensityLipoproteinReceptorImpairs Sterol-inducedMouseMacrophageABCA1Expression

Jihong Han, D. Hajjar, Xiaoye Zhou, 2008 .

Induction of CD36 expression by oxidized LDL and IL-4 by a common signaling pathway dependent on protein kinase C and PPAR-gamma.

A. Gotto, Jihong Han, R. Silverstein, 2000, Journal of lipid research.

Aging and arteriosclerosis. I. Development of myointimal hyperplasia after endothelial injury

D. Hajjar, D. Alonso, M. Weksler, 1986, The Journal of experimental medicine.

Tyrosine nitration in prostaglandin H(2) synthase.

D. Hajjar, M. Resnick, D. Mittar, 2002, Journal of lipid research.

Cicletanine stimulates cholesteryl ester hydrolase activities and prostacyclin production in arterial smooth muscle cells.

D. Hajjar, K. Pomerantz, 1989, Archives des maladies du coeur et des vaisseaux.

Direct Observation of Nitrosylated Heme in Myoglobin and Hemoglobin by Electrospray Ionization Mass Spectrometry

B. Chait, D. Hajjar, U. Mirza, 1997 .

Altered Cholesterol Trafficking in Herpesvirus-infected Arterial Cells

R. Kaner, A. C. Nicholson, D. Hajjar, 1995, The Journal of Biological Chemistry.

Human glial cell production of lipoxygenase-generated eicosanoids: a potential role in the pathophysiology of vascular changes following traumatic brain injury.

J. Ghajar, D. Hajjar, R. Patterson, 1989, The Journal of trauma.

Structurally homologous ligand binding of integrin Mac-1 and viral glycoprotein C receptors.

D. Altieri, T. Edgington, D. Hajjar, 1991, Science.

Pitavastatin alters the expression of thrombotic and fibrinolytic proteins in human vascular cells

A. Gotto, Jihong Han, K. Hajjar, 2003, Journal of cellular biochemistry.

Altered cholesteryl ester cycle is associated with lipid accumulation in herpesvirus-infected arterial smooth muscle cells.

D. Hajjar, D. Falcone, J. Fabricant, 1985, The Journal of biological chemistry.

Measuring Teaching: A Relative Value Scale in Teaching

D. Hajjar, D. Falcone, C. Bardes, 1998 .

De-endothelialized Rabbit Aorta CRITICAL ROLE OF NEOINTIMAL SMOOTH MUSCLE CELLS

B. Weksler, D. Hajjar, C. Minick, 1981 .

Interactions of arterial cells. Studies on the mechanisms of endothelial cell modulation of cholesterol metabolism in co-cultured smooth muscle cells.

A. Marcus, K. Hajjar, D. Hajjar, 1987, The Journal of biological chemistry.

REVIEW Repair Mechanisms in Oxidant-Driven Chronic In ﬂ ammatory Disease

D. Hajjar, R. Deeb, 2016 .

Profound biopterin oxidation and protein tyrosine nitration in tissues of ApoE-null mice on an atherogenic diet: contribution of inducible nitric oxide synthase.

M. Crabtree, N. Maeda, D. Hajjar, 2007, American journal of physiology. Heart and circulatory physiology.

Launching of an American Medical College in the Middle East: Educational Challenges in a Multicultural Environment

A. Gotto, D. Hajjar, 2013 .

Prospects for Policy Advances in Science and Technology in the Gulf Arab States: "The Role for International Partnerships".

J. Richardson, V. Narayanamurti, A. Siddiqi, 2014 .

Rosiglitazone alleviates intrahepatic cholestasis induced by α‐naphthylisothiocyanate in mice: The role of circulating 15‐deoxy‐Δ12,14‐PGJ2 and Nogo

Yan Zhu, Jihong Han, Xiaoxiao Yang, 2020, British journal of pharmacology.

A Molecular Redox Switch on p21ras

B. Chait, B. Hempstead, L. Quilliam, 1997, The Journal of Biological Chemistry.

Repair Mechanisms in Oxidant-Driven Chronic Inflammatory Disease.

D. Hajjar, R. Deeb, 2016, The American journal of pathology.

Inflammation at the molecular interface of atherogenesis: an anthropological journey.

D. Hajjar, B. Lamon, 2008, The American journal of pathology.

Inhibition of ERK1/2 and Activation of LXR Synergistically Reduce Atherosclerotic Lesions in ApoE-Deficient Mice

Jihong Han, Xiaoxiao Yang, Yuanli Chen, 2015, Arteriosclerosis, thrombosis, and vascular biology.

Eicosanoid metabolism in cholesterol-enriched arterial smooth muscle cells. Evidence for reduced posttranscriptional processing of cyclooxygenase I and reduced cyclooxygenase II gene expression.

D. Hajjar, B. Summers, K. Pomerantz, 1993, Biochemistry.

Inducible nitric oxide synthase provides protection against injury-induced thrombosis in female mice.

K. Hajjar, D. Hajjar, R. Deeb, 2011, American journal of physiology. Heart and circulatory physiology.

Rosiglitazone alleviates intrahepatic cholestasis induced by α -naphthylisothiocyanate in mice: The role of circulating 15-deoxy- Δ 12,14 -PGJ 2 and Nogo

Jihong Han, Xiaoxiao Yang, Yuanli Chen, 2019 .

Infection and atherosclerosis: potential roles of pathogen burden and molecular mimicry.

M. Burnett, G. Vercellotti, D. Hajjar, 2000, Arteriosclerosis, thrombosis, and vascular biology.

REVIEW Mammalian Target of Rapamycin A Metabolic Rheostat for Regulating Adipose Tissue Function and Cardiovascular Health

M. Wipperman, D. Hajjar, David C. Montrose, 2022 .

Identification of interferon-γ as a new molecular target of liver X receptor.

Jihong Han, Yuanli Chen, Yajun Duan, 2014, The Biochemical journal.