D. Surman

发表

CTLA-4 dysregulation of self/tumor-reactive CD8+ T-cell function is CD4+ T-cell dependent.

S. Rosenberg, N. Restifo, L. Gattinoni, 2006, Blood.

This information is current as Occurring T Regulatory Cells T Helper Cells and Hindered by Naturally + Tumor/Self-Antigen Is Augmented by CD4 T Cell Immunity Against a + CD8

S. Rosenberg, C. Piccirillo, N. Restifo, 2013 .

Enhancing efficacy of recombinant anticancer vaccines with prime/boost regimens that use two different vectors.

S. Rosenberg, K. Irvine, E. P. Shulman, 1997, Journal of the National Cancer Institute.

Vaccination with a recombinant vaccinia virus encoding a "self" antigen induces autoimmune vitiligo and tumor cell destruction in mice: requirement for CD4(+) T lymphocytes.

B. Moss, S. Rosenberg, K. Irvine, 1999, Proceedings of the National Academy of Sciences of the United States of America.

Apoptotic death of CD8+ T lymphocytes after immunization: induction of a suppressive population of Mac-1+/Gr-1+ cells.

S. Rosenberg, F. Pericle, V. Bronte, 1998, Journal of immunology.

Suppressive Population of Mac-1 After Immunization : Induction of a T Lymphocytes + Apoptotic Death of CD 8 and

S. Rosenberg, F. Pericle, V. Bronte, 1998 .

Transfectant influenza A viruses are effective recombinant immunogens in the treatment of experimental cancer.

S. Rosenberg, A. García-Sastre, P. Palese, 1998, Virology.

Construction and characterization of a triple-recombinant vaccinia virus encoding B7-1, interleukin 12, and a model tumor antigen.

B. Moss, M. Carroll, N. Restifo, 1998, Journal of the National Cancer Institute.

Generation of polyclonal rabbit antisera to mouse melanoma associated antigens using gene gun immunization.

S. Rosenberg, K. Irvine, E. P. Shulman, 1998, Journal of immunological methods.

Genetic vaccination with "self" tyrosinase-related protein 2 causes melanoma eradication but not vitiligo.

R. Ronca, V. Bronte, N. Restifo, 2000, Cancer research.

Causes Melanoma Eradication but not Vitiligo Genetic Vaccination with ''Self'' Tyrosinase-related Protein 2

R. Ronca, V. Bronte, N. Restifo, 2000 .

Tumor Regression and Autoimmunity after Reversal of a Functionally Tolerant State of Self-reactive CD8+ T Cells

S. Rosenberg, C. Klebanoff, N. Restifo, 2003, The Journal of experimental medicine.

IL-15 enhances the in vivo antitumor activity of tumor-reactive CD8+ T Cells

T. Waldmann, S. Rosenberg, C. Klebanoff, 2004, Proceedings of the National Academy of Sciences of the United States of America.

Therapeutic Synergy in Esophageal Cancer and Mesothelioma Is Predicted by Dynamic BH3 Profiling

J. Trepel, H. C. Hodges, Hyun-Sung Lee, 2021, Molecular Cancer Therapeutics.

Bile acid-induced “Minority MOMP” promotes esophageal carcinogenesis while maintaining apoptotic resistance via Mcl-1

Jonathan M. Hernandez, Chuong D. Hoang, C. Hoang, 2019, Oncogene.

Deletion of Mthfd1l causes embryonic lethality and neural tube and craniofacial defects in mice

S. Vokes, D. Surman, D. Appling, 2012, Proceedings of the National Academy of Sciences.

Targeting mutant p53-R248W reactivates WT p53 function and alters the onco-metabolic profile

W. Linehan, E. Appella, D. Schrump, 2023, Frontiers in Oncology.

Bile acid and cigarette smoke enhance the aggressive phenotype of esophageal adenocarcinoma cells by downregulation of the mitochondrial uncoupling protein-2

Jonathan M. Hernandez, S. Xi, D. Schrump, 2017, Oncotarget.

Bedside to bench and back again: how animal models are guiding the development of new immunotherapies for cancer

S. Rosenberg, C. Klebanoff, N. Restifo, 2004, Journal of leukocyte biology.

Cyclin-dependent kinase activation and S-phase induction of the cyclin B1 gene are linked through the CCAAT elements.

J. Yates, J. Yates, K. Wright, 1997, Cell growth & differentiation : the molecular biology journal of the American Association for Cancer Research.

Cutting Edge: CD4+ T Cell Control of CD8+ T Cell Reactivity to a Model Tumor Antigen

N. Restifo, M. Dudley, D. Surman, 2000, The Journal of Immunology.

CD8+ T Cell Immunity Against a Tumor/Self-Antigen Is Augmented by CD4+ T Helper Cells and Hindered by Naturally Occurring T Regulatory Cells

S. Rosenberg, C. Klebanoff, C. Piccirillo, 2005, The Journal of Immunology.

Poor immunogenicity of a self/tumor antigen derives from peptide-MHC-I instability and is independent of tolerance.

B. Baker, N. Restifo, D. Surman, 2004, The Journal of clinical investigation.

Identification of a Kb-restricted CTL epitope of beta-galactosidase: potential use in development of immunization protocols for "self" antigens.

N. Restifo, D. Surman, W. Overwijk, 1997, Methods.

Induction of Thioredoxin-Interacting Protein by a Histone Deacetylase Inhibitor, Entinostat, Is Associated with DNA Damage and Apoptosis in Esophageal Adenocarcinoma

Jonathan M. Hernandez, Chuong D. Hoang, D. Beer, 2018, Molecular Cancer Therapeutics.

Metabolomic and BH3 profiling of esophageal cancers: novel assessment methods for precision therapy

Jonathan M. Hernandez, S. Steinberg, A. Letai, 2018, BMC Gastroenterology.

Poor immunogenicity of a self/tumor antigen derives from peptide-MHC-I instability and is independent of tolerance.

B. Baker, N. Restifo, D. Surman, 2004, The Journal of clinical investigation.