M. W. Veldkamp

发表

A single Na(+) channel mutation causing both long-QT and Brugada syndromes.

A. Wilde, M. P. van den Berg, C. Bezzina, 1999, Circulation research.

Two distinct congenital arrhythmias evoked by a multidysfunctional Na(+) channel.

P. C. Viswanathan, A. Wilde, J. Balser, 2000, Circulation research.

Norepinephrine induces action potential prolongation and early afterdepolarizations in ventricular myocytes isolated from human end-stage failing hearts.

A. V. van Ginneken, T. Opthof, J. D. de Bakker, 2001, European heart journal.

Busting the myth of the ‘Chinese restaurant syndrome’

M. W. Veldkamp, S. Baalman, J. R. de Groot, 2017 .

A mutation in the human cardiac sodium channel (E161K) contributes to sick sinus syndrome, conduction disease and Brugada syndrome in two families.

A. Wilde, H. Tan, T. Opthof, 2005, Journal of molecular and cellular cardiology.

UvA-DARE ( Digital Academic Repository ) HERG channel ( dys ) function revealed by dynamic action potential clamp technique

Ronald Wilders, B. Jonge, A. V. van Ginneken, 2004 .

Biphasic response of action potential duration to metabolic inhibition in rabbit and human ventricular myocytes: role of transient outward current and ATP-regulated potassium current.

A. V. van Ginneken, A. Verkerk, M. W. Veldkamp, 1996, Journal of molecular and cellular cardiology.

Ion Channels, the QT Interval, and Arrhythmias

A. Wilde, A. Wilde, M. W. Veldkamp, 1997, Pacing and clinical electrophysiology : PACE.

Treatment of Atrial and Ventricular Arrhythmias Through Autonomic Modulation.

A. Wilde, R. Coronel, J. D. de Bakker, 2015, JACC. Clinical electrophysiology.

Implications of inhomogeneous distribution of I and I channels inKS Kr ventricle with respect to effects of class III agents and beta-agonists

M. W. Veldkamp, A. V. Ginneken, 1999 .

Is IGF‐1 a useful inhibitor of Na+/H+‐exchanger activity?

M. W. Veldkamp, A. Baartscheer, M. Veldkamp, 2018, Acta physiologica.