C. Lauderback

发表

Glutathione elevation and its protective role in acrolein-induced protein damage in synaptosomal membranes: relevance to brain lipid peroxidation in neurodegenerative disease

D. Butterfield, C. Pocernich, A. L. Cardin, 2001, Neurochemistry International.

Amyloid beta-peptide effects on synaptosomes from apolipoprotein E-deficient mice.

D. Butterfield, J. Keller, M. Kindy, 2000, Journal of neurochemistry.

Derivatives of Xanthic Acid are Novel Antioxidants: Application to Synaptosomes

A. Castegna, C. Lauderback, S. Varadarajan, 2003, Free radical research.

Amyloid b-Peptide Effects on Synaptosomes from Apolipoprotein E-Deficient Mice *

D. Butterfield, W. Markesbery, J. Keller, 2000 .

Amyloid β‐Peptide Effects on Synaptosomes from Apolipoprotein E‐Deficient Mice

D. Butterfield, W. Markesbery, J. Keller, 2000 .

5-Aminosalicylic Acid Protection against Oxidative Damage to Synaptosomal Membranes by Alkoxyl Radicals In Vitro

D. Butterfield, C. Lauderback, J. Kański, 2004, Neurochemical Research.

Different mechanisms of oxidative stress and neurotoxicity for Alzheimer's A beta(1--42) and A beta(25--35).

D. Butterfield, C. Lauderback, M. Aksenova, 2001, Journal of the American Chemical Society.

D609 inhibits ionizing radiation-induced oxidative damage by acting as a potent antioxidant.

D. Butterfield, C. Lauderback, T. Yu, 2001, The Journal of pharmacology and experimental therapeutics.

Modulation of phospholipid asymmetry in synaptosomal membranes by the lipid peroxidation products, 4-hydroxynonenal and acrolein: implications for Alzheimer's disease

D. Butterfield, A. Castegna, C. Lauderback, 2004, Brain Research.

Lipid peroxidation and protein oxidation in Alzheimer's disease brain: Potential causes and consequences involving amyloid β-peptide-associated free radical oxidative stress

D. Butterfield, C. Lauderback, D. Butterfield, 2002 .

Evidence that amyloid beta-peptide-induced lipid peroxidation and its sequelae in Alzheimer’s disease brain contribute to neuronal death

D. Butterfield, A. Castegna, C. Lauderback, 2002, Neurobiology of Aging.

Apolipoprotein E modulates Alzheimer’s Aβ(1–42)-induced oxidative damage to synaptosomes in an allele-specific manner

D. Butterfield, M. Kindy, C. Lauderback, 2002, Brain Research.

The glial glutamate transporter, GLT‐1, is oxidatively modified by 4‐hydroxy‐2‐nonenal in the Alzheimer's disease brain: the role of Aβ1–42

D. Butterfield, W. Markesbery, J. Keller, 2001, Journal of neurochemistry.

The pyrrolopyrimidine U101033E is a potent free radical scavenger and prevents Fe(II)-induced lipid peroxidation in synaptosomal membranes.

D. Butterfield, A. Breier, Adam M. Breier, 2000, Biochimica et biophysica acta.

Amyloid β-peptide inhibits Na+-dependent glutamate uptake

D. Butterfield, N. W. Pedigo, J. Carney, 1999 .

Vulnerability of synaptosomes from apoE knock-out mice to structural and oxidative modifications induced by A beta(1-40): implications for Alzheimer's disease.

D. Butterfield, J. Keller, M. Kindy, 2001, Biochemistry.

Different Mechanisms of Oxidative Stress and Neurotoxicity for AlzheimersA ‚(1-42) and A‚(25-35)

D. Butterfield, C. Lauderback, M. Aksenova, 2001 .

Serial Review : Causes and Consequences of Oxidative Stress in Alzheimer ’ s Disease

D. Butterfield, Mark A. Smith, C. Lauderback, 2002 .

Lipid peroxidation and protein oxidation in Alzheimer's disease brain: Potential causes and consequences involving amyloid β-peptide-associated free radical oxidative stress

C. Lauderback, D. Butterfield, 2002 .

Amyloid beta-peptide inhibits Na+-dependent glutamate uptake.

D. Butterfield, N. W. Pedigo, J. Carney, 1999, Life sciences.

Lipid peroxidation and protein oxidation in Alzheimer's disease brain: potential causes and consequences involving amyloid beta-peptide-associated free radical oxidative stress.

C. Lauderback, D. Butterfield, 2002, Free radical biology & medicine.