Mingxin Tang

发表

Doxycycline attenuates protein aggregation in cardiomyocytes and improves survival of a mouse model of cardiac proteinopathy.

Xuejun Wang, Hanqiao Zheng, Mingxin Tang, 2010, Journal of the American College of Cardiology.

Cardiotoxic and Cardioprotective Features of Chronic &bgr;-Adrenergic Signaling

F. Wang, Hui Gao, E. Gao, 2013, Circulation research.

&agr;B-Crystallin Suppresses Pressure Overload Cardiac Hypertrophy

Wei Huang, Xuejun Wang, Hanqiao Zheng, 2008, Circulation research.

(cid:1) B-Crystallin Suppresses Pressure Overload Cardiac Hypertrophy

Xuejun Wang, Hanqiao Zheng, Mingxin Tang, 2008 .

Cardiac GRK2 Ablation Induces a Novel Ca Handling Phenotype Resistant to Adverse Alterations and Remodeling After Myocardial Infarction

S. Houser, G. Dorn, W. Koch, 2012 .

Heart Failure Cardiac G-Protein–Coupled Receptor Kinase 2 Ablation Induces a Novel Ca Handling Phenotype Resistant to Adverse Alterations and Remodeling After Myocardial Infarction

S. Houser, G. Dorn, W. Koch, 2012 .

Increasing Cardiac Contractility After Myocardial Infarction Exacerbates Cardiac Injury and Pump Dysfunction

David M. Harris, S. Houser, W. Koch, 2009, Circulation research.

Proteasome functional insufficiency activates the calcineurin-NFAT pathway in cardiomyocytes and promotes maladaptive remodelling of stressed mouse hearts.

J. Li, J. Molkentin, Wei Huang, 2010, Cardiovascular research.

Increasing T‐type calcium channel activity by β‐adrenergic stimulation contributes to β‐adrenergic regulation of heart rates

S. Houser, J. Molkentin, M. Xie, 2017, The Journal of physiology.

Cardiomyocyte PKA Ablation Enhances Basal Contractility While Eliminates Cardiac β-Adrenergic Response Without Adverse Effects on the Heart.

S. Houser, Daohai Yu, Ying Tian, 2019, Circulation research.

Genomic and “Polyomic” Studies of Cardiovascular and Inﬂammatory Diseases HIF1 mediates a switch in pyruvate kinase isoforms after myocardial infarction

R. Shohet, Mingxin Tang, Mark Menor, 2018 .

A therapeutic dose of doxorubicin activates ubiquitin-proteasome system-mediated proteolysis by acting on both the ubiquitination apparatus and proteasome.

Jinbao Liu, Xuejun Wang, Y. Ryu, 2008, American journal of physiology. Heart and circulatory physiology.

Enhanced basal contractility but reduced excitation-contraction coupling efficiency and beta-adrenergic reserve of hearts with increased Cav1.2 activity.

S. Houser, H. Nakayama, J. Molkentin, 2010, American journal of physiology. Heart and circulatory physiology.

PKI-GFP Prevents Cardiac Dysfunction and Myocyte Death Induced by ISO

Naresh, F. Wang, Hui Gao, 2013 .

Aberrant protein aggregation is essential for a mutant desmin to impair the proteolytic function of the ubiquitin-proteasome system in cardiomyocytes.

Jinbao Liu, R. Mestril, Xuejun Wang, 2006, Journal of molecular and cellular cardiology.

β-Adrenergic Stimulation Increases Cav3.1 Activity in Cardiac Myocytes through Protein Kinase A

Y. Li, Mingxin Tang, Xiongwen Chen, 2012, PloS one.

The TGF-β pathway mediates doxorubicin effects on cardiac endothelial cells.

R. Shohet, Mingxin Tang, E. Konorev, 2016, Journal of molecular and cellular cardiology.

Cardiac G-Protein–Coupled Receptor Kinase 2 Ablation Induces a Novel Ca2+ Handling Phenotype Resistant to Adverse Alterations and Remodeling After Myocardial Infarction

S. Houser, G. Dorn, W. Koch, 2012, Circulation.

The activin receptor ligand trap ActRIIB:ALK4‐Fc ameliorates cardiomyopathy induced by neuromuscular disease and diabetes

S. Joshi, R. Suragani, Ravindra Kumar, 2022, FEBS letters.

Calcium influx through Cav1.2 is a proximal signal for pathological cardiomyocyte hypertrophy.

David M. Harris, S. Houser, W. Koch, 2011, Journal of molecular and cellular cardiology.

Persistent increases in Ca2+ influx through Cav1.2 shortens action potential and causes Ca2+ overload-induced afterdepolarizations and arrhythmias

David M. Harris, W. Koch, H. Nakayama, 2015, Basic Research in Cardiology.

Anomaly detection of industrial state quantity time-Series data based on correlation and long short-term memory

Wei Chen, Wenwen Yang, Mingxin Tang, 2022, Connect. Sci..

HIF1 mediates a switch in pyruvate kinase isoforms after myocardial infarction.

R. Shohet, Mingxin Tang, Mark Menor, 2018, Physiological genomics.

Increasing Cardiac Contractility After Myocardial Infarction Exacerbates Cardiac Injury and Pump Dysfunction

David M. Harris, S. Houser, W. Koch, 2010, Circulation research.

Cav3.1/α1G T-Type Ca2+ Channels are Involved in the Heart Rate Regulation

J. Molkentin, F. Wang, Mingxin Tang, 2011 .

Proteasome functional insufficiency activates the calcineurin-NFAT pathway in cardiomyocytes and promotes maladaptive remodelling of stressed mouse hearts.

J. Li, J. Molkentin, Wei Huang, 2010, Cardiovascular research.

Cardiotoxic and Cardioprotective Features of Chronic β -Adrenergic Signaling

Naresh, F. Wang, Hui Gao, 2013 .

Cardiotoxic and Cardioprotective Features of Chronic β -Adrenergic Signaling

S. Kunapuli, Mingxin Tang, Xiongwen Chen, 2013 .

A therapeutic dose of doxorubicin activates ubiquitin-proteasome system-mediated proteolysis by acting on both the ubiquitination apparatus and proteasome.

Jinbao Liu, Xuejun Wang, Y. Ryu, 2008, American journal of physiology. Heart and circulatory physiology.

Doxycycline attenuates protein aggregation in cardiomyocytes and improves survival of a mouse model of cardiac proteinopathy.

Xuejun Wang, Hanqiao Zheng, Mingxin Tang, 2010, Journal of the American College of Cardiology.

Protein Kinase A Is a Master Regulator of Physiological and Pathological Cardiac Hypertrophy

S. Houser, Mingxin Tang, Xiongwen Chen, 2024, Circulation research.