D. Hartley

发表

Minocycline inhibits cytochrome c release and delays progression of amyotrophic lateral sclerosis in mice

Betty Y. S. Kim, S. Gullans, R. Ferrante, 2002, Nature.

Appropriate Use Criteria for Amyloid PET: A Report of the Amyloid Imaging Task Force, the Society of Nuclear Medicine and Molecular Imaging, and the Alzheimer’s Association

Keith A. Johnson, M. Carrillo, J. Karlawish, 2013, The Journal of Nuclear Medicine.

Oxygen or glucose deprivation-induced neuronal injury in cortical cell cultures is reduced by tetanus toxin

H. Monyer, D. Choi, R. Giffard, 1992, Neuron.

Abeta protofibrils possess a stable core structure resistant to hydrogen exchange.

P. Lansbury, T. Walz, H. Lashuel, 2003, Biochemistry.

Small non-fibrillar assemblies of amyloid β-protein bearing the Arctic mutation induce rapid neuritic degeneration

D. Selkoe, D. Hartley, B. Whalen, 2005, Neurobiology of Disease.

Preparation and characterization of toxic Aβ aggregates for structural and functional studies in Alzheimer's disease research

Asad Jan, H. Lashuel, D. Hartley, 2010, Nature Protocols.

Use of the freshwater clamCorbicula manilensis as a monitor for organochlorine pesticides

D. Hartley, J. Johnston, 1983, Bulletin of environmental contamination and toxicology.

Gemfibrozil Ameliorates Relapsing-Remitting Experimental Autoimmune Encephalomyelitis Independent of Peroxisome Proliferator-Activated Receptor-α

D. Hartley, K. Pahan, M. Jana, 2007, Molecular Pharmacology.

Delayed rescue of N-methyl-D-aspartate receptor-mediated neuronal injury in cortical culture.

D. Choi, D. Hartley, Dean M. Hartley, 1989, The Journal of pharmacology and experimental therapeutics.

Attenuation of cortical neuronal apoptosis by gangliosides.

E. Costa, D. Choi, I. Jou, 1999, The Journal of pharmacology and experimental therapeutics.

Attenuation of Cortical Neuronal Apoptosis by Gangliosides 1

E. Costa, D. Choi, I. Jou, 1999 .

7‐Chlorokynurenate Blocks NMDA Receptor‐Mediated Neurotoxicity in Murine Cortical Culture

H. Monyer, D. Choi, D. Hartley, 1990, The European journal of neuroscience.

Endogenous galanin protects mouse hippocampal neurons against amyloid toxicity in vitro via activation of galanin receptor-2.

E. Mufson, S. Perez, D. Hartley, 2011, Journal of Alzheimer's disease : JAD.

In vitro studies of amyloid beta-protein fibril assembly and toxicity provide clues to the aetiology of Flemish variant (Ala692-->Gly) Alzheimer's disease.

D. Selkoe, D. Walsh, D. Teplow, 2001, The Biochemical journal.

An improved method of preparing the amyloid β-protein for fibrillogenesis and neurotoxicity experiments

D. Walsh, D. Teplow, A. Fink, 2000, Neurobiology of Aging.

Automated determination of excitatory amino acid neurotoxicity in cortical culture

D. Choi, D. Hartley, J. Klingman, 1990, Journal of Neuroscience Methods.

New Class of Inhibitors of Amyloid-β Fibril Formation

H. Lashuel, A. Aggarwal, D. Hartley, 2002, The Journal of Biological Chemistry.

Amyloid β-Protein Fibrillogenesis

D. Selkoe, D. Walsh, D. Teplow, 1999, The Journal of Biological Chemistry.

Calcium and glutamate-induced cortical neuronal death.

D. Choi, D. Hartley, 1993, Research publications - Association for Research in Nervous and Mental Disease.

Amyloid beta-protein fibrillogenesis. Structure and biological activity of protofibrillar intermediates.

D. Selkoe, D. Walsh, D. Teplow, 1999, The Journal of biological chemistry.

Folate interactions with cerebral G proteins

S. R. Snodgrass, D. Hartley, 1990, Neurochemical Research.

Protofibrils of amyloid β-protein inhibit specific K+ currents in neocortical cultures

D. Selkoe, D. Hartley, C. Ye, 2003, Neurobiology of Disease.

Distinct Roles for Ras-Guanine Nucleotide-Releasing Factor 1 (Ras-GRF1) and Ras-GRF2 in the Induction of Long-Term Potentiation and Long-Term Depression

Shaomin Li, Shaomin Li, L. Feig, 2006, The Journal of Neuroscience.

AMPA receptor activation potentiates zinc neurotoxicity

D. Choi, J. Koh, D. Hartley, 1993, Neuron.

A Preliminary Study of Cu Exposure Effects upon Alzheimer’s Amyloid Pathology

Xudong Huang, A. Dedeoglu, Kerry A. Cormier, 2020, Biomolecules.

Interleukin‐1α stimulates non‐amyloidogenic pathway by α‐secretase (ADAM‐10 and ADAM‐17) cleavage of APP in human astrocytic cells involving p38 MAP kinase

S. Bandyopadhyay, C. Cahill, N. Chattopadhyay, 2006 .

Interleukin-1alpha stimulates non-amyloidogenic pathway by alpha-secretase (ADAM-10 and ADAM-17) cleavage of APP in human astrocytic cells involving p38 MAP kinase.

S. Bandyopadhyay, C. Cahill, N. Chattopadhyay, 2006, Journal of neuroscience research.

Selective inhibition of NF-κB activation prevents dopaminergic neuronal loss in a mouse model of Parkinson's disease

Howard E. Gendelman, Sankar Ghosh, Jeffrey H. Kordower, 2007, Proceedings of the National Academy of Sciences.

Down syndrome and Alzheimer's disease: Common pathways, common goals

M. Carrillo, A. Granholm, M. Krams, 2015, Alzheimer's & Dementia.

GA4GH: International policies and standards for data sharing across genomic research and healthcare

Grant M. Wood, James A. Eddy, Julius O. B. Jacobsen, 2021, Cell genomics.

Neurons Regulate Extracellular Levels of Amyloid β-Protein via Proteolysis by Insulin-Degrading Enzyme

D. Selkoe, D. Walsh, M. Rosner, 2000, The Journal of Neuroscience.

The measurement of gamma-glutamyl hydrolase (conjugase) activity in rat brain

S. R. Snodgrass, D. Hartley, P. Bradshaw, 1988, Neurochemical Research.

Considerations for the assessment of suicidal ideation and behavior in older adults with cognitive decline and dementia

David S. Miller, E. Siemers, K. Yaffe, 2016, Alzheimer's & dementia.

Can we prevent Alzheimer's disease? Secondary “prevention” trials in Alzheimer's disease

R. Petersen, M. Carrillo, E. Siemers, 2013, Alzheimer's & Dementia.

Transgenerational Rescue of a Genetic Defect in Long-Term Potentiation and Memory Formation by Juvenile Enrichment

Shaomin Li, L. Feig, D. Hartley, 2009, The Journal of Neuroscience.

Neurodegenerative disease: Amyloid pores from pathogenic mutations

P. Lansbury, T. Walz, H. Lashuel, 2002, Nature.

Appropriate use criteria for amyloid PET: A report of the Amyloid Imaging Task Force, the Society of Nuclear Medicine and Molecular Imaging, and the Alzheimer's Association

Peter Herscovitch, Nicolaas I. Bohnen, Norman L. Foster, 2013, Alzheimer's & Dementia.

A de novo designed helix-turn-helix peptide forms nontoxic amyloid fibrils

D. Selkoe, D. Walsh, D. Teplow, 2000, Nature Structural Biology.

Intraneuronal Aβ detection in 5xFAD mice by a new Aβ-specific antibody

Sean M. Riordan, G. Bu, Chunjiang Yu, 2012, Molecular Neurodegeneration.

Mixtures of wild-type and a pathogenic (E22G) form of Abeta40 in vitro accumulate protofibrils, including amyloid pores.

P. Lansbury, T. Walz, H. Lashuel, 2003, Journal of molecular biology.

Protofibrillar Intermediates of Amyloid β-Protein Induce Acute Electrophysiological Changes and Progressive Neurotoxicity in Cortical Neurons

Dominic M. Walsh, D. Selkoe, D. Walsh, 1999, The Journal of Neuroscience.

Transglutaminase Induces Protofibril-like Amyloid β-Protein Assemblies That Are Protease-resistant and Inhibit Long-term Potentiation*

Shaomin Li, T. Walz, Zongli Li, 2008, Journal of Biological Chemistry.

The Environment versus Genetics in Controlling the Contribution of MAP Kinases to Synaptic Plasticity

Shaomin Li, L. Feig, D. Hartley, 2006, Current Biology.

Certain Inhibitors of Synthetic Amyloid β-Peptide (Aβ) Fibrillogenesis Block Oligomerization of Natural Aβ and Thereby Rescue Long-Term Potentiation

Dominic M. Walsh, D. Selkoe, D. Walsh, 2005, The Journal of Neuroscience.

Certain inhibitors of synthetic amyloid beta-peptide (Abeta) fibrillogenesis block oligomerization of natural Abeta and thereby rescue long-term potentiation.

D. Selkoe, D. Walsh, J. Fadeeva, 2005, The Journal of neuroscience : the official journal of the Society for Neuroscience.

21-Aminosteroids attenuate excitotoxic neuronal injury in cortical cell cultures

D. Choi, D. Hartley, H. Monyer, 1990, Neuron.

A brief, but repeated, swimming protocol is sufficient to overcome amyloid β‐protein inhibition of hippocampal long‐term potentiation

Shaomin Li, L. Feig, D. Hartley, 2007, The European journal of neuroscience.

Update on appropriate use criteria for amyloid PET imaging: Dementia experts, mild cognitive impairment, and education

Keith A. Johnson, M. Carrillo, J. Karlawish, 2013, Alzheimer's & Dementia.

Update on Appropriate Use Criteria for Amyloid PET Imaging: Dementia Experts, Mild Cognitive Impairment, and Education

Keith A. Johnson, M. Carrillo, J. Karlawish, 2013, The Journal of Nuclear Medicine.

Neuronal exosomes reveal Alzheimer's disease biomarkers in Down syndrome

A. Granholm, E. Mufson, E. Goetzl, 2017, Alzheimer's & Dementia.

The calcium channel blocker nifedipine attenuates slow excitatory amino acid neurotoxicity.

D. Choi, J. Koh, D. Hartley, 1990, Science.

Communicating mild cognitive impairment diagnoses with and without amyloid imaging

E. Teng, J. Karlawish, S. Kremen, 2017, Alzheimer's Research & Therapy.

New class of inhibitors of amyloid-beta fibril formation. Implications for the mechanism of pathogenesis in Alzheimer's disease.

H. Lashuel, A. Aggarwal, D. Hartley, 2002, The Journal of biological chemistry.

Title Amyloid β-protein fibrillogenesis . Structure and biological activity of protofibrillar intermediates

D. Selkoe, D. Walsh, D. Teplow, 1999 .

Suicidal ideation and behavior assessment in dementia studies: An Internet survey

David S. Miller, E. Siemers, K. Yaffe, 2016, Alzheimer's & dementia.

Is Secondary Prevention of Alzheimer's Disease Possible? A Discussion of Studies in the Alzheimer's Disease Field

M. Carrillo, H. Snyder, K. Fargo, 2014 .

The Many Faces of A: Structures and Activity

D. Selkoe, D. Walsh, D. Hartley, 2003 .

Amyloid β-protein induced electrophysiological changes are dependent on aggregation state: N-methyl-d-aspartate (NMDA) versus non-NMDA receptor/channel activation

D. Selkoe, D. Walsh, D. Hartley, 2004, Neuroscience Letters.

Amyloid beta-protein induced electrophysiological changes are dependent on aggregation state: N-methyl-D-aspartate (NMDA) versus non-NMDA receptor/channel activation.

D. Selkoe, D. Walsh, D. Hartley, 2004, Neuroscience letters.