S. Campbell

发表

Effect of transmurally heterogeneous myocyte excitation–contraction coupling on canine left ventricular electromechanics

Roy C. P. Kerckhoffs, Elliot J. Howard, A. McCulloch, 2009, Experimental physiology.

Mechanisms of transmurally varying myocyte electromechanics in an integrated computational model

Andrew D. McCulloch, Sarah N. Flaim, Stuart G. Campbell, 2008, Philosophical Transactions of the Royal Society A: Mathematical, Physical and Engineering Sciences.

Modular Design of a Tissue Engineered Pulsatile Conduit using Human Induced Pluripotent Stem Cell-derived Cardiomyocytes.

L. Niklason, M. Kural, Yan Huang, 2020, Acta biomaterialia.

Skeletal myosin binding protein-C isoforms regulate thin filament activity in a Ca2+-dependent manner

C. D. dos Remedios, S. Sadayappan, D. Warshaw, 2018, Scientific Reports.

Altered ventricular torsion and transmural patterns of myocyte relaxation precede heart failure in aging F344 rats.

K. Campbell, S. Campbell, Premi Haynes, 2013, American journal of physiology. Heart and circulatory physiology.

Predicting Effects of Tropomyosin Mutations on Cardiac Muscle Contraction through Myofilament Modeling

Jeffrey R. Moore, W. Lehman, S. Campbell, 2016, Front. Physiol..

Prospects for In Vitro Myofilament Maturation in Stem Cell-Derived Cardiac Myocytes

S. Campbell, Jonas Schwan, 2015, Biomarker insights.

Myofilament glycation in diabetes reduces contractility by inhibiting tropomyosin movement, is rescued by cMyBPC domains.

Samantha K. Barrick, M. Papadaki, P. Rainer, 2021, Journal of molecular and cellular cardiology.

Myofilament Glycation in Diabetes Reduces Contractility by Inhibiting Tropomyosin Movement, is Rescued by cMyBPC Domains

Samantha K. Barrick, M. Papadaki, P. Rainer, 2021, bioRxiv.

Potential impacts of the cardiac troponin I mobile domain on myofilament activation and relaxation.

S. Campbell, Jenette G Creso, 2021, Journal of molecular and cellular cardiology.

Contractile work directly modulates mitochondrial protein levels in human engineered heart tissues.

B. Ehrlich, S. Campbell, Y. Qyang, 2020, American journal of physiology. Heart and circulatory physiology.

Shortening Velocity Causes Myosin Isoform Shift in Human Engineered Heart Tissues.

S. Campbell, Y. Qyang, L. Sewanan, 2020, Circulation research.

Tissue-Engineered Vascular Grafts with Advanced Mechanical Strength from Human iPSCs.

L. Niklason, M. Kural, Yifan Yuan, 2020, Cell stem cell.

A Protocol for Collecting Human Cardiac Tissue for Research

K. Campbell, M. Mitov, M. Bonnell, 2016, The VAD journal : the journal of mechanical assisted circulation and heart failure.

Transmural heterogeneity of cellular level power output is reduced in human heart failure.

Mihail I Mitov, A. Stromberg, K. Campbell, 2014, Journal of molecular and cellular cardiology.

Tissue-Engineered Stem Cell Models of Cardiovascular Diseases

L. Niklason, M. Kural, Jiesi Luo, 2019, Cardiovascular Regenerative Medicine.

Extracellular Matrix From Hypertrophic Myocardium Provokes Impaired Twitch Dynamics in Healthy Cardiomyocytes

Jinkyu Park, D. Jacoby, S. Campbell, 2019, JACC. Basic to translational science.

MicroRNA function can be reversed by altering target gene expression levels

D. Engelman, S. Campbell, Alexander A. Svoronos, 2021, iScience.

Signaling network model of cardiomyocyte morphological changes in familial cardiomyopathy

A. McCulloch, J. Omens, J. Saucerman, 2021, bioRxiv.

Low-dose dasatinib rescues cardiac function in Noonan syndrome.

Ivana Y. Kuo, B. Ehrlich, A. Bennett, 2016, JCI insight.

pressure-volume relationships ventricular integrating cardiac muscle contraction into Dynamic left ventricular elastance: a model for

B. Slinker, A. McCulloch, J. Omens, 2015 .

Dynamic left ventricular elastance: a model for integrating cardiac muscle contraction into ventricular pressure-volume relationships.

B. Slinker, H. Granzier, S. Campbell, 2008, Journal of applied physiology.

Humanized Dsp ACM Mouse Model Displays Stress-Induced Cardiac Electrical and Structural Phenotypes

Maegen A. Borzok, P. Mohler, F. Akar, 2022, Cells.

A microwell cell capture device reveals variable response to dobutamine in isolated cardiomyocytes

S. Campbell, J. A. Clark, J. Weiss, 2019, bioRxiv.

Modelling sarcomeric cardiomyopathies with human cardiomyocytes derived from induced pluripotent stem cells

S. Campbell, L. Sewanan, 2020, The Journal of physiology.

Tissue-Engineered Vascular Rings from Human iPSC-Derived Smooth Muscle Cells

Hongwei Wu, Jiesi Luo, Biraja C. Dash, 2016, Stem cell reports.

Polycystin 2 is increased in disease to protect against stress-induced cell death

J. Nerbonne, L. Cantley, H. J. Chung, 2020, Scientific Reports.

A short history of the development of mathematical models of cardiac mechanics

Kenneth S Campbell, Steven A Niederer, Stuart G Campbell, 2019, Journal of molecular and cellular cardiology.

Engineered Lung Tissues Prepared from Decellularized Lung Slices.

L. Niklason, S. Campbell, K. Leiby, 2022, Journal of visualized experiments : JoVE.

Cardiomyocyte d-dopachrome tautomerase protects against heart failure.

L. Leng, R. Bucala, K. Margulies, 2019, JCI insight.

Muscle LIM Protein Force-Sensing Mediates Sarcomeric Biomechanical Signaling in Human Familial Hypertrophic Cardiomyopathy

C. Marboe, I. Chen, Jiesi Luo, 2022, Circulation.

Loss of crossbridge inhibition drives pathological cardiac hypertrophy in patients harboring the TPM1 E192K mutation

M. Rynkiewicz, Jeffrey R. Moore, W. Lehman, 2021, The Journal of general physiology.

Force-Dependent Recruitment from the Myosin Off State Contributes to Length-Dependent Activation.

Kenneth S Campbell, Stuart G Campbell, K. Campbell, 2018, Biophysical journal.

chains in single muscle fibers Quantitative electrophoretic analysis of myosin heavy

L. Rome, H. Sweeney, M. Geeves, 2015 .

Diverse relaxation rates exist among rat cardiomyocytes isolated from a single myocardial region

S. Campbell, J. A. Clark, J. A. Clark, 2018, The Journal of physiology.

Mouse and computational models link Mlc2v dephosphorylation to altered myosin kinetics in early cardiac disease.

Andrew D McCulloch, Richard L Moss, Ju Chen, 2012, The Journal of clinical investigation.

Fast-relaxing cardiomyocytes exert a dominant role in the relaxation behavior of heterogeneous myocardium.

K. Campbell, S. Campbell, Jonas Schwan, 2020, Archives of biochemistry and biophysics.

Functional and structural differences between skinned and intact muscle preparations

K. Campbell, S. Niederer, S. Campbell, 2022, The Journal of general physiology.

Precise Binding of Tropomyosin on Actin Involves Sequence-Dependent Variance in Coiled-Coil Twisting.

S. Fischer, M. Rynkiewicz, Jeffrey R. Moore, 2018, Biophysical journal.

ratsmyosin heavy chain isoform expression in Age-dependent changes in contraction and regional

C. Carnes, G. Diffee, S. Campbell, 2015 .

Anisotropic engineered heart tissue made from laser-cut decellularized myocardium

Thomas J. Ryan, D. Jacoby, S. Campbell, 2016, Scientific Reports.

Multi-scale computational models of familial hypertrophic cardiomyopathy: genotype to phenotype

Andrew D McCulloch, Stuart G Campbell, A. McCulloch, 2011, Journal of The Royal Society Interface.

Stem Cell Biology

M. Nikkhah, D. Brafman, S. Campbell, 2015, Biomarker insights.

Vascular smooth muscle cells derived from inbred swine induced pluripotent stem cells for vascular tissue engineering.

Xia Li, L. Niklason, M. Kural, 2017, Biomaterials.

Decreased Polycystin 2 Expression Alters Calcium-Contraction Coupling and Changes Beta-Adrenergic Signaling Pathways

Ivana Y. Kuo, B. Ehrlich, S. Campbell, 2015 .

Decreased polycystin 2 expression alters calcium-contraction coupling and changes β-adrenergic signaling pathways

Ivana Y. Kuo, B. Ehrlich, S. Campbell, 2014, Proceedings of the National Academy of Sciences.

Skeletal myosin binding protein-C isoforms regulate thin filament activity in a Ca2+-dependent manner

C. D. dos Remedios, S. Sadayappan, D. Warshaw, 2018, Scientific Reports.

Danicamtiv Enhances Systolic Function and Frank‐Starling Behavior at Minimal Diastolic Cost in Engineered Human Myocardium

D. Jacoby, S. Campbell, L. Sewanan, 2021, Journal of the American Heart Association.

Use of Human Cells and Heart Muscle Tissue Patches as Therapeutics for Heart Diseases

Y. Huang, Jiesi Luo, Jinkyu Park, 2019, Reference Module in Biomedical Sciences.

Mavacamten preserved length-dependent contractility and improved diastolic function in human engineered heart tissue.

S. Campbell, L. Sewanan, Shi Shen, 2021, American journal of physiology. Heart and circulatory physiology.

Rational engineering of lung alveolar epithelium

L. Niklason, Pavlina Baevova, Yifan Yuan, 2023, NPJ Regenerative medicine.

In vitro engineering of the lung alveolus

L. Niklason, Pavlina Baevova, Yifan Yuan, 2022, bioRxiv.

Patient mutations linked to arrhythmogenic cardiomyopathy enhance calpain-mediated desmoplakin degradation.

T. Bunch, Jinkyu Park, D. Jacoby, 2019, JCI insight.

Sarcomere-Directed Calcium Reporters in Cardiomyocytes.

S. Campbell, Y. Qyang, J. T. Hinson, 2019, Circulation research.

Slowing of contractile kinetics by myosin-binding protein C can be explained by its cooperative binding to the thin filament.

S. Campbell, Jonas Schwan, Clinton J. Wang, 2016, Journal of molecular and cellular cardiology.

Coupling of adjacent tropomyosins enhances cross-bridge-mediated cooperative activation in a markov model of the cardiac thin filament.

Andrew D McCulloch, Kenneth S Campbell, Stuart G Campbell, 2010, Biophysical journal.

Mechanisms of pathogenicity in the hypertrophic cardiomyopathy-associated TPM1 variant S215L

M. Rynkiewicz, Jeffrey R. Moore, W. Lehman, 2023, PNAS nexus.

KBTBD13 and the ever-expanding sarcomeric universe.

S. Niederer, S. Campbell, 2020, The Journal of clinical investigation.

The Effect of Tropomyosin Mutations on Actin-Tropomyosin Binding: In Search of Lost Time.

M. Rynkiewicz, Jeffrey R. Moore, W. Lehman, 2019, Biophysical journal.

Mechanisms of residual force enhancement in skeletal muscle: insights from experiments and mathematical models

Kenneth S. Campbell, Stuart G. Campbell, K. Campbell, 2011, Biophysical Reviews.

Modeling predicts a connection between sinus vortex effects and aortic compliance.

Yasser Aboelkassem, Dragana Savic, Stuart G Campbell, 2016, Journal of theoretical biology.

Mathematical modeling of aortic valve dynamics during systole.

S. Campbell, Y. Aboelkassem, D. Savić, 2015, Journal of theoretical biology.

A Mathematical Model of Muscle Containing Heterogeneous Half-Sarcomeres Exhibits Residual Force Enhancement

Kenneth S. Campbell, Stuart G. Campbell, P. Chris Hatfield, 2011, PLoS Comput. Biol..

Polycystin 2 is increased in disease to protect against stress-induced cell death

J. Nerbonne, L. Cantley, H. J. Chung, 2020, Scientific Reports.

A model with Heterogeneous Half-Sarcomeres Exhibits Residual Force Enhancement After Active Stretch

K. Campbell, S. Campbell, C. Hatfield, 2011 .

Structural determinants of muscle thin filament cooperativity.

Stuart G Campbell, Jeffrey R. Moore, W. Lehman, 2016, Archives of biochemistry and biophysics.

Multi-scale modeling of excitation-contraction coupling in the normal and failing heart

Andrew D. McCulloch, Roy C. P. Kerckhoffs, Stuart G. Campbell, 2009, 2009 Annual International Conference of the IEEE Engineering in Medicine and Biology Society.

Multiscale Modeling of Cardiovascular Function Predicts That the End-Systolic Pressure Volume Relationship Can Be Targeted via Multiple Therapeutic Strategies

K. Campbell, S. Campbell, B. Chrisman, 2020, Frontiers in Physiology.

Evidence for synergy between sarcomeres and fibroblasts in an in vitro model of myocardial reverse remodeling.

S. Campbell, L. Sewanan, Shi Shen, 2021, Journal of Molecular and Cellular Cardiology.

Timing and magnitude of systolic stretch affect myofilament activation and mechanical work.

Andrew D McCulloch, Stuart G Campbell, A. McCulloch, 2014, American journal of physiology. Heart and circulatory physiology.

Mouse and computational models link Mlc2v dephosphorylation to altered myosin kinetics in early cardiac disease.

A. McCulloch, Yusu Gu, N. Dalton, 2012, The Journal of clinical investigation.

Skeletal myosin binding protein-C isoforms regulate thin filament Skeletal myosin binding protein-C isoforms regulate thin filament activity in a Ca(2+)-dependent manner activity in a Ca(2+)-dependent manner us know how access to this document benefits you.

S. Sadayappan, D. Warshaw, Amy Li, 2022 .