J. Pu

Alterations of Na+ currents in myocytes from epicardial border zone of the infarcted heart. A possible ionic mechanism for reduced excitability and postrepolarization refractoriness.

J. Pu, P. Boyden, 1997, Circulation research.

Density and function of inward currents in right atrial cells from chronically fibrillating canine atria.

M. Rosen, P. Danilo, T. Yagi, 2002, Cardiovascular research.

Effects of Bay Y5959 on Ca2+ currents and intracellular Ca2+ in cells that have survived in the epicardial border of the infarcted canine heart.

J. Pu, P. Boyden, F. Ruffy, 1999, Journal of cardiovascular pharmacology.

Lidocaine action on Na+ currents in ventricular myocytes from the epicardial border zone of the infarcted heart.

J. Balser, J. Pu, P. Boyden, 1998, Circulation research.

Ca(2+) transients and Ca(2+) waves in purkinje cells : role in action potential initiation.

H. E. Keurs, J. Pu, P. Boyden, 2000, Circulation research.

Ca(2+)-dependent outward currents in myocytes from epicardial border zone of 5-day infarcted canine heart.

J. Pu, P. Boyden, R. Aggarwal, 1997, The American journal of physiology.

Abnormalities in Ca(i)handling in myocytes that survive in the infarcted heart are not just due to alterations in repolarization.

R. Robinson, J. Pu, P. Boyden, 2000, Journal of molecular and cellular cardiology.