E. Schafer

发表

Analysis of the interaction between host factor Sam68 and viral elements during foot-and-mouth disease virus infections

D. Rai, P. Lawrence, A. Kloc, 2015, Virology Journal.

Beyond the biology: Evaluating the role of political, economic, and social factors associated with the incidence and mortality of SARS-CoV-2 during the first seven months of the COVID-19 pandemic (preprint)

E. Schafer, S. Eberl, L. Velazquez-Salinas, 2022, 2204.13995.

Structure-functional analysis of human immunodeficiency virus type 1 (HIV-1) Vpr: role of leucine residues on Vpr-mediated transactivation and virus replication.

Simon C Watkins, B. Majumder, A. Srinivasan, 2004, Virology.

Dendritic Cells Infected with vpr-Positive Human Immunodeficiency Virus Type 1 Induce CD8+ T-Cell Apoptosis via Upregulation of Tumor Necrosis Factor Alpha

Narasimhan J. Venkatachari, B. Majumder, E. Schafer, 2007, Journal of Virology.

Human Immunodeficiency Virus Type 1 Vpr Impairs Dendritic Cell Maturation and T-Cell Activation: Implications for Viral Immune Escape

C. Rinaldo, Xiao-li Huang, B. Majumder, 2005, Journal of Virology.

Novel 6xHis tagged foot-and-mouth disease virus vaccine bound to nanolipoprotein adjuvant via metal ions provides antigenic distinction and effective protective immunity.

P. Hoeprich, T. Burrage, D. Rai, 2016, Virology.

The nuclear protein Sam68 is cleaved by the FMDV 3C protease redistributing Sam68 to the cytoplasm during FMDV infection of host cells.

P. Lawrence, E. Schafer, E. Rieder, 2012, Virology.

Differential regulation of host cellular genes by HIV-1 viral protein R (Vpr): cDNA microarray analysis using isogenic virus.

B. Majumder, A. Srinivasan, R. Collman, 2004, Biochemical and biophysical research communications.

Molecular and functional characterization of a novel splice variant of ANKHD1 that lacks the KH domain and its role in cell survival and apoptosisc

Ansuman Chattopadhyay, Velpandi Ayyavoo, B. Majumder, 2005 .

Antiviral effects of mifepristone on human immunodeficiency virus type-1 (HIV-1): targeting Vpr and its cellular partner, the glucocorticoid receptor (GR).

Narasimhan J. Venkatachari, Velpandi Ayyavoo, E. Schafer, 2006, Antiviral research.

Foot-and-mouth disease virus 5'-terminal S fragment is required for replication and modulation of the innate immune response in host cells.

T. de los Santos, D. Rai, A. Kloc, 2017, Virology.

Novel Antiviral Strategies Targeting the Human Immunodeficiency Virus Type 1 (HIV-1) Viral Protein R and Its Cellular Partner, the Glucocorticoid Receptor

E. Schafer, 2005 .

Attenuation of Foot-and-Mouth Disease Virus by Engineered Viral Polymerase Fidelity

Grace Campagnola, O. Peersen, T. de los Santos, 2017, Journal of Virology.

Corrigendum to “Structure-functional analysis of human immunodeficiency virus type 1 (HIV-1) Vpr: role of leucine residues in Vpr-mediated transactivation and virus replication” [Virology 328 (2004) 89–100]

Simon C Watkins, B. Majumder, A. Srinivasan, 2005 .

Residues within the Foot-and-Mouth Disease Virus 3Dpol Nuclear Localization Signal Affect Polymerase Fidelity

D. Rai, A. Kloc, E. Schafer, 2020, Journal of Virology.

Dendritic Cells Infected with vpr -Positive Human Immunodeﬁciency Virus Type 1 Induce CD8 (cid:1) T-Cell Apoptosis via Upregulation of Tumor Necrosis Factor Alpha (cid:1)

Narasimhan J. Venkatachari, B. Majumder, E. Schafer, 2007 .

Analysis of the interaction between host factor Sam68 and viral elements during foot-and-mouth disease virus infections

D. Rai, P. Lawrence, A. Kloc, 2015, Virology Journal.

Inhibitors of Foot and Mouth Disease Virus Targeting a Novel Pocket of the RNA-Dependent RNA Polymerase

S. Sarafianos, J. Moran, M. Mcintosh, 2010, PloS one.

Repeated exposure to 5D9, an inhibitor of 3D polymerase, effectively limits the replication of foot-and-mouth disease virus in host cells.

S. Sarafianos, M. Mcintosh, D. Rai, 2013, Antiviral research.

Repeated exposure to 5 D 9 , an inhibitor of 3 D polymerase , effectively limits the replication

S. Sarafianos, M. Mcintosh, D. Rai, 2013 .