Hailong Wang

发表

CtIP Protein Dimerization Is Critical for Its Recruitment to Chromosomal DNA Double-stranded Breaks*

Linda Z. Shi, David J. Chen, M. Berns, 2012, The Journal of Biological Chemistry.

The Interaction of CtIP and Nbs1 Connects CDK and ATM to Regulate HR–Mediated Double-Strand Break Repair

Linda Z. Shi, David J. Chen, J. Yates, 2013, PLoS genetics.

CtIP maintains stability at common fragile sites and inverted repeats by end resection-independent endonuclease activity.

Jing He, Linda Z Shi, Michael W Berns, 2014, Molecular cell.

The concerted roles of FANCM and Rad52 in the protection of common fragile sites

Lei Li, Shibo Li, Lei Li, 2018, Nature Communications.

Microhomology-mediated End Joining and Homologous Recombination share the initial end resection step to repair DNA double-strand breaks in mammalian cells

Linda Z. Shi, Michael W. Berns, Hailong Wang, 2013, Proceedings of the National Academy of Sciences.

PIF1 helicase promotes break‐induced replication in mammalian cells

L. Truong, T. Chiba, Shibo Li, 2021, The EMBO journal.

PLK1-mediated phosphorylation of PPIL2 regulates HR via CtIP

Z. Jia, Hailong Wang, Jimin Zheng, 2022, Frontiers in Cell and Developmental Biology.

Catalytic and noncatalytic roles of the CtIP endonuclease in double-strand break end resection.

Hailong Wang, Ji-Hoon Lee, Yi Zhou, 2014, Molecular cell.

The concerted roles of FANCM and Rad52 in the protection of common fragile sites

Lei Li, Shibo Li, Hailong Wang, 2018, Nature Communications.

CtIP maintains stability at common fragile sites and inverted repeats by end resection-independent endonuclease activity.

J. Shiloach, M. Berns, L. Truong, 2014, Molecular cell.

MutSβ protects common fragile sites by facilitating homology-directed repair at DNA double-strand breaks with secondary structures

Hailong Wang, Xiaohua Wu, Youhang Li, 2023, Nucleic acids research.